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Oncogene

Volumn 21, Issue 20, 2002, Pages 3207-3212

Human p14arf-mediated cell cycle arrest strictly depends on intact p53 signaling pathways

(4) Weber, H Oliver a,b Samuel, Temesgen a,c Rauch, Pia a Funk, Jens Oliver a

a UNIVERSITY OF ERLANGEN NUREMBERG (Germany)

b NATIONAL CANCER INSTITUTE (United States)

c BURNHAM INSTITUTE (United States)

Author keywords

14 3 3 ; DNA tumor virus; INK4a ARF; p21; p53

Indexed keywords

CYCLIN DEPENDENT KINASE INHIBITOR; PROTEIN P14; PROTEIN P21; PROTEIN P53;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL CYCLE; CELL CYCLE G1 PHASE; CELL CYCLE G2 PHASE; CELL TYPE; COLON CARCINOMA; CONTROLLED STUDY; GENE ISOLATION; GENE LOCUS; GENETIC TRANSCRIPTION; HUMAN; HUMAN CELL; INK4A ARF GENE; MITOSIS INHIBITION; NONHUMAN; OPEN READING FRAME; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN DEGRADATION; SIGNAL TRANSDUCTION; TUMOR SUPPRESSOR GENE; WILD TYPE;

CELL CYCLE; CELL CYCLE PROTEINS; CELL LINE; CYCLIN-DEPENDENT KINASE INHIBITOR P27; ECDYSONE; EXONUCLEASES; G1 PHASE; G2 PHASE; GENE EXPRESSION REGULATION; GENE TARGETING; GENES, P53; HUMANS; KIDNEY; NEOPLASM PROTEINS; PROTEINS; RECEPTORS, STEROID; RECOMBINANT FUSION PROTEINS; SIGNAL TRANSDUCTION; SPECIES SPECIFICITY; TRANSFECTION; TUMOR MARKERS, BIOLOGICAL; TUMOR SUPPRESSOR PROTEIN P14ARF; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

ADENOVIRIDAE; ANIMALIA;

EID: 85047700405 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205429 Document Type: Article

Times cited : (61)

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