Resveratrol attenuates formaldehyde induced hyperphosphorylation of tau protein and cytotoxicity in N2a cells

Frontiers in Neuroscience

Volumn 10, Issue JAN, 2017, Pages

  He, Xiaping ^a,b,c   Li, Zhenhui ^a,b   Rizak, Joshua D ^a   Wu, Shihao ^a,b   Wang, Zhengbo ^a   He, Rongqiao ^d,e   Su, Min ^c   Qin, Dongdong ^a   Wang, Jingkun ^c   Hu, Xintian ^a,f  

^a KUNMING INSTITUTE OF ZOOLOGY   (China)

^b UNIVERSITY OF CHINESE ACADEMY OF SCIENCES   (China)

^c Yunnan Province Company Key Laboratory for TCM and Ethnic Drug of New Drug Creation   (China)

^d INSTITUTE OF BIOPHYSICS   (China)

^e INSTITUTE OF PSYCHOLOGY   (China)

^f Shenzhen Fundamental Research Institutions   (China)

Author keywords

Alzheimer's disease; CaMKII; Formaldehyde; GSK 3 ; PP2A; Resveratrol; Tau protein

Indexed keywords

ELONGATION FACTOR 2 KINASE; FORMALDEHYDE; GLYCOGEN SYNTHASE KINASE 3BETA; PHOSPHOPROTEIN PHOSPHATASE 2A; RESVERATROL; TAU PROTEIN;

ALZHEIMER DISEASE; ANIMAL CELL; ARTICLE; CONTROLLED STUDY; DRUG CYTOTOXICITY; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME REPRESSION; MOUSE; NEUROPROTECTION; NEUROTOXICITY; NONHUMAN; PROTEIN DEPHOSPHORYLATION; PROTEIN PHOSPHORYLATION;

EID: 85011866956     PISSN: 16624548     EISSN: 1662453X     Source Type: Journal    
DOI: 10.3389/fnins.2016.00598     Document Type: Article

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