Recognition of H3K9 methylation by GLP is required for efficient establishment of H3K9 methylation, rapid target gene repression, and mouse viability

Genes and Development

Volumn 29, Issue 4, 2015, Pages 379-393

  Liu, Nan ^a,b,c   Zhang, Zhuqiang ^c   Wu, Hui ^d   Jiang, Yonghua ^b   Meng, Lingjun ^b   Xiong, Jun ^b   Zhao, Zuodong ^b   Zhou, Xiaohua ^b   Li, Jia ^b   Li, Hong ^b   Zheng, Yong ^b   Chen, She ^b   Cai, Tao ^b   Gao, Shaorong ^e   Zhu, Bing ^b,c  

^a BEIJING NORMAL UNIVERSITY   (China)

^b NATIONAL INSTITUTE OF BIOLOGICAL SCIENCES   (China)

^c INSTITUTE OF BIOPHYSICS   (China)

^d JILIN UNIVERSITY   (China)

^e TONGJI UNIVERSITY   (China)

Author keywords

Cell differentiation; G9a; Gene repression; GLP; H3K9 methylation

Indexed keywords

ANKYRIN; G9A PROTEIN; GLP PROTEIN; HISTONE METHYLTRANSFERASE; OCTAMER TRANSCRIPTION FACTOR 4; TRANSCRIPTION FACTOR NANOG; UNCLASSIFIED DRUG; G9A PROTEIN, MOUSE; GLP PROTEIN, MOUSE; HISTONE; HISTONE LYSINE METHYLTRANSFERASE; HOMEODOMAIN PROTEIN; NANOG PROTEIN, MOUSE; NUCLEOSOME; POU5F1 PROTEIN, MOUSE; PROTEIN BINDING;

ANIMAL CELL; ARTICLE; BINDING AFFINITY; CELL DIFFERENTIATION; CONTROLLED STUDY; EMBRYO; EMBRYONIC STEM CELL; ENZYME ACTIVITY; FEMALE; GENE REPRESSION; GENE SILENCING; GROWTH RETARDATION; HISTONE METHYLATION; LETHALITY; MOUSE; NONHUMAN; NUCLEOSOME; OSSIFICATION; PRIORITY JOURNAL; PROTEIN DOMAIN; STARVATION; ANIMAL; C57BL MOUSE; CRANIOFACIAL MALFORMATION; CYTOLOGY; GENE EXPRESSION REGULATION; GENETICS; INTRAUTERINE GROWTH RETARDATION; MALE; METABOLISM; METHYLATION; MUTATION;

MUS;

ANIMALS; CELL DIFFERENTIATION; CRANIOFACIAL ABNORMALITIES; EMBRYONIC STEM CELLS; FEMALE; FETAL GROWTH RETARDATION; GENE EXPRESSION REGULATION, DEVELOPMENTAL; GENE SILENCING; HISTONE-LYSINE N-METHYLTRANSFERASE; HISTONES; HOMEODOMAIN PROTEINS; MALE; METHYLATION; MICE; MICE, INBRED C57BL; MUTATION; NUCLEOSOMES; OCTAMER TRANSCRIPTION FACTOR-3; PROTEIN BINDING;

EID: 85005843352     PISSN: 08909369     EISSN: 15495477     Source Type: Journal    
DOI: 10.1101/gad.254425.114     Document Type: Article

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