Th2 and eosinophil responses suppress inflammatory arthritis

Nature Communications

Volumn 7, Issue , 2016, Pages

  Chen, Zhu ^a,b   Andreev, Darja ^a   Oeser, Katharina ^c   Krljanac, Branislav ^c   Hueber, Axel ^a   Kleyer, Arnd ^a   Voehringer, David ^c   Schett, Georg ^a   Bozec, Aline ^a  

^a UNIVERSITY OF ERLANGEN NUREMBERG   (Germany)

^b ANHUI MEDICAL UNIVERSITY   (China)

^c UNIVERSITY HOSPITAL ERLANGEN   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

GAMMA INTERFERON; GRANULOCYTE MACROPHAGE COLONY STIMULATING FACTOR; INTERLEUKIN 10; INTERLEUKIN 13; INTERLEUKIN 17; INTERLEUKIN 2; INTERLEUKIN 4; INTERLEUKIN 5; STAT6 PROTEIN; TRANSCRIPTION FACTOR GATA 3; TUMOR NECROSIS FACTOR; BIOLOGICAL MARKER;

ABNORMALITY; ANNELID; BLOOD; CELLS AND CELL COMPONENTS; IMMUNE RESPONSE; INHIBITION; PROTEIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BONE ATROPHY; CELL PROTECTION; CONTROLLED STUDY; CYTOKINE PRODUCTION; EOSINOPHIL; HUMAN; HUMAN TISSUE; HYPEREOSINOPHILIA; IMMUNE RESPONSE; IMMUNOMODULATION; JOINT; LEUKOCYTE FUNCTION; LYMPHOCYTE FUNCTION; MACROPHAGE; MOUSE; NIPPOSTRONGYLUS BRASILIENSIS; NIPPOSTRONGYLUS BRASILIENSIS INFECTION; NONHUMAN; PROTEIN EXPRESSION; RHEUMATOID ARTHRITIS; SIGNAL TRANSDUCTION; STRONGYLE INFECTION; TH2 CELL; THERAPY WITH HELMINTHS; ANIMAL; ARTHRITIS; BAGG ALBINO MOUSE; C57BL MOUSE; CELL COUNT; COMPLICATION; EOSINOPHILIA; IMMUNOLOGY; INFLAMMATION; METABOLISM; NIPPOSTRONGYLUS; PARASITOLOGY; PATHOLOGY; PHYSIOLOGY; TRANSGENIC MOUSE;

NIPPOSTRONGYLUS BRASILIENSIS; VERMES;

ANIMALS; ARTHRITIS; BIOMARKERS; CELL COUNT; EOSINOPHILIA; EOSINOPHILS; HUMANS; INFLAMMATION; INTERLEUKIN-13; INTERLEUKIN-4; JOINTS; MACROPHAGES; MICE, INBRED BALB C; MICE, INBRED C57BL; MICE, TRANSGENIC; NIPPOSTRONGYLUS; STAT6 TRANSCRIPTION FACTOR; STRONGYLIDA INFECTIONS; TH2 CELLS; TUMOR NECROSIS FACTOR-ALPHA;

EID: 84975833997     PISSN: None     EISSN: 20411723     Source Type: Journal    
DOI: 10.1038/ncomms11596     Document Type: Article

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