Chronic cerebral hypoperfusion enhances Tau hyperphosphorylation and reduces autophagy in Alzheimers disease mice

Scientific Reports

Volumn 6, Issue , 2016, Pages

  Qiu, Lifeng ^a   Ng, Gandi ^a   Tan, Eng King ^a,b   Liao, Ping ^a   Kandiah, Nagaendran ^a   Zeng, Li ^a,b  

^a NATIONAL NEUROSCIENCE INSTITUTE   (Singapore)

^b DUKE NUS MEDICAL SCHOOL   (Singapore)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID BETA-PROTEIN (1-42); MAP1LC3 PROTEIN, MOUSE; MICROTUBULE ASSOCIATED PROTEIN; PEPTIDE FRAGMENT; TAU PROTEIN;

129 MOUSE; AGE; ALZHEIMER DISEASE; ANIMAL; AUTOPHAGY; BRAIN ISCHEMIA; C57BL MOUSE; CHRONIC DISEASE; DISEASE MODEL; FEMALE; FRONTAL LOBE; GENETICS; HIPPOCAMPUS; HUMAN; IMMUNOBLOTTING; MALE; METABOLISM; PHOSPHORYLATION; TRANSGENIC MOUSE;

AGE FACTORS; ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; AUTOPHAGY; BRAIN ISCHEMIA; CHRONIC DISEASE; DISEASE MODELS, ANIMAL; FEMALE; FRONTAL LOBE; HIPPOCAMPUS; HUMANS; IMMUNOBLOTTING; MALE; MICE, 129 STRAIN; MICE, INBRED C57BL; MICE, TRANSGENIC; MICROTUBULE-ASSOCIATED PROTEINS; PEPTIDE FRAGMENTS; PHOSPHORYLATION; TAU PROTEINS;

EID: 84963638735     PISSN: None     EISSN: 20452322     Source Type: Journal    
DOI: 10.1038/srep23964     Document Type: Article

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