Resveratrol reduces amyloid-beta (Aβ1–42)-induced paralysis through targeting proteostasis in an Alzheimer model of Caenorhabditis elegans

European Journal of Nutrition

Volumn 55, Issue 2, 2016, Pages 741-747

  Regitz, Charlotte ^a   Fitzenberger, Elena ^a   Mahn, Friederike Luise ^a   Dußling, Lisa Marie ^a   Wenzel, Uwe ^a  

^a JUSTUS LIEBIG UNIVERSITY   (Germany)

Author keywords

Amyloid peptide; Autophagy; Caenorhabditis elegans; Proteasome; Resveratrol; Unfolded protein response

Indexed keywords

AMYLOID BETA PROTEIN[1-42]; CHAPERONE; PROTEASOME; PROTEIN AGGREGATE; REAGENT; RESVERATROL; X BOX BINDING PROTEIN 1; AMYLOID BETA PROTEIN; AMYLOID BETA-PROTEIN (1-42); CAENORHABDITIS ELEGANS PROTEIN; CARRIER PROTEIN; PEPTIDE FRAGMENT; STILBENE DERIVATIVE; UBIQUITIN; UBL-5 PROTEIN, C ELEGANS; XBP-1 PROTEIN, C ELEGANS;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOPHAGY; CAENORHABDITIS ELEGANS; CONTROLLED STUDY; DRUG MECHANISM; ENDOPLASMIC RETICULUM; LYSOSOME; MACROAUTOPHAGY; NONHUMAN; PARALYSIS; PROMOTER REGION; PROTEIN AGGREGATION; PROTEIN DEGRADATION; PROTEIN HOMEOSTASIS; REAL TIME POLYMERASE CHAIN REACTION; RNA INTERFERENCE; TRANSGENIC ANIMAL; UNFOLDED PROTEIN RESPONSE; ANIMAL; CHEMICALLY INDUCED; DISEASE MODEL; DRUG EFFECTS; ENDOPLASMIC RETICULUM STRESS; GENETICS; METABOLISM; MITOCHONDRION; PROTEOSTASIS DEFICIENCIES;

ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; AUTOPHAGY; CAENORHABDITIS ELEGANS; CAENORHABDITIS ELEGANS PROTEINS; CARRIER PROTEINS; DISEASE MODELS, ANIMAL; ENDOPLASMIC RETICULUM STRESS; MITOCHONDRIA; PARALYSIS; PEPTIDE FRAGMENTS; PROTEASOME ENDOPEPTIDASE COMPLEX; PROTEOSTASIS DEFICIENCIES; RNA INTERFERENCE; STILBENES; UBIQUITINS; UNFOLDED PROTEIN RESPONSE;

EID: 84959123644     PISSN: 14366207     EISSN: 14366215     Source Type: Journal    
DOI: 10.1007/s00394-015-0894-1     Document Type: Article

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