-
1
-
-
79952195585
-
Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47
-
COI: 1:CAS:528:DC%2BC3MXhsFyqtro%3D, PID: 21297633
-
Anderson CA et al (2011) Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47. Nat Genet 43:246–252
-
(2011)
Nat Genet
, vol.43
, pp. 246-252
-
-
Anderson, C.A.1
-
2
-
-
70649086082
-
A genome-wide association study identifies three new susceptibility loci for ulcerative colitis in the Japanese population
-
COI: 1:CAS:528:DC%2BD1MXhsVWlsLbL, PID: 19915573
-
Asano K et al (2009) A genome-wide association study identifies three new susceptibility loci for ulcerative colitis in the Japanese population. Nat Genet 41:1325–1329
-
(2009)
Nat Genet
, vol.41
, pp. 1325-1329
-
-
Asano, K.1
-
3
-
-
48349136889
-
Genome-wide association defines more than 30 distinct susceptibility loci for Crohn’s disease
-
COI: 1:CAS:528:DC%2BD1cXptFSqs7k%3D, PID: 18587394
-
Barrett JC et al (2008) Genome-wide association defines more than 30 distinct susceptibility loci for Crohn’s disease. Nat Genet 40:955–962
-
(2008)
Nat Genet
, vol.40
, pp. 955-962
-
-
Barrett, J.C.1
-
4
-
-
70649103789
-
Genome-wide association study of ulcerative colitis identifies three new susceptibility loci, including the HNF4A region
-
COI: 1:CAS:528:DC%2BD1MXhsVWlsbvM, PID: 19915572
-
Barrett JC et al (2009) Genome-wide association study of ulcerative colitis identifies three new susceptibility loci, including the HNF4A region. Nat Genet 41:1330–1334
-
(2009)
Nat Genet
, vol.41
, pp. 1330-1334
-
-
Barrett, J.C.1
-
5
-
-
70350683774
-
Expression quantitative trait loci detected in cell lines are often present in primary tissues
-
COI: 1:CAS:528:DC%2BD1MXhtlWhu7fN, PID: 19671653
-
Bullaughey K, Chavarria CI, Coop G, Gilad Y (2009) Expression quantitative trait loci detected in cell lines are often present in primary tissues. Hum Mol Genet 18:4296–4303
-
(2009)
Hum Mol Genet
, vol.18
, pp. 4296-4303
-
-
Bullaughey, K.1
Chavarria, C.I.2
Coop, G.3
Gilad, Y.4
-
6
-
-
35348905481
-
The molecular basis of lipoid proteinosis: mutations in extracellular matrix protein 1
-
COI: 1:CAS:528:DC%2BD2sXhtlOhtL%2FF, PID: 17927570
-
Chan I, Liu L, Hamada T, Sethuraman G, McGrath JA (2007) The molecular basis of lipoid proteinosis: mutations in extracellular matrix protein 1. Exp Dermatol 16:881–890
-
(2007)
Exp Dermatol
, vol.16
, pp. 881-890
-
-
Chan, I.1
Liu, L.2
Hamada, T.3
Sethuraman, G.4
McGrath, J.A.5
-
7
-
-
77649235913
-
How to do successful gene expression analysis using real-time PCR
-
COI: 1:CAS:528:DC%2BC3cXjtVCgurc%3D, PID: 19969088
-
Derveaux S, Vandesompele J, Hellemans J (2010) How to do successful gene expression analysis using real-time PCR. Methods 50:227–230. doi:10.1016/j.ymeth.2009.11.001
-
(2010)
Methods
, vol.50
, pp. 227-230
-
-
Derveaux, S.1
Vandesompele, J.2
Hellemans, J.3
-
8
-
-
28744454762
-
Gene expression variation and expression quantitative trait mapping of human chromosome 21 genes
-
COI: 1:CAS:528:DC%2BD2MXht1GgsrjM, PID: 16251198
-
Deutsch S et al (2005) Gene expression variation and expression quantitative trait mapping of human chromosome 21 genes. Hum Mol Genet 14:3741–3749
-
(2005)
Hum Mol Genet
, vol.14
, pp. 3741-3749
-
-
Deutsch, S.1
-
9
-
-
34548805282
-
A genome-wide association study of global gene expression
-
COI: 1:CAS:528:DC%2BD2sXhtV2isL%2FO, PID: 17873877
-
Dixon AL et al (2007) A genome-wide association study of global gene expression. Nat Genet 39:1202–1207
-
(2007)
Nat Genet
, vol.39
, pp. 1202-1207
-
-
Dixon, A.L.1
-
10
-
-
77950243833
-
Multiple common variants for celiac disease influencing immune gene expression
-
COI: 1:CAS:528:DC%2BC3cXisFelurY%3D, PID: 20190752
-
Dubois PC et al (2010) Multiple common variants for celiac disease influencing immune gene expression. Nat Genet 42:295–302
-
(2010)
Nat Genet
, vol.42
, pp. 295-302
-
-
Dubois, P.C.1
-
11
-
-
33845340501
-
A genome-wide association study identifies IL23R as an inflammatory bowel disease gene
-
COI: 1:CAS:528:DC%2BD28Xht1CntrjF, PID: 17068223
-
Duerr RH et al (2006) A genome-wide association study identifies IL23R as an inflammatory bowel disease gene. Science 314:1461–1463
-
(2006)
Science
, vol.314
, pp. 1461-1463
-
-
Duerr, R.H.1
-
12
-
-
77956059154
-
Genome-wide searching of rare genetic variants in WTCCC data
-
PID: 20549515
-
Feng T, Zhu X (2010) Genome-wide searching of rare genetic variants in WTCCC data. Hum Genet 128:269–280
-
(2010)
Hum Genet
, vol.128
, pp. 269-280
-
-
Feng, T.1
Zhu, X.2
-
13
-
-
76349108817
-
Genetic analysis in a Dutch study sample identifies more ulcerative colitis susceptibility loci and shows their additive role in disease risk
-
COI: 1:CAS:528:DC%2BC3cXhsFGiu7k%3D, PID: 19861958
-
Festen EA et al (2010) Genetic analysis in a Dutch study sample identifies more ulcerative colitis susceptibility loci and shows their additive role in disease risk. Am J Gastroenterol 105:395–402
-
(2010)
Am J Gastroenterol
, vol.105
, pp. 395-402
-
-
Festen, E.A.1
-
14
-
-
44349136821
-
Genetic determinants of ulcerative colitis include the ECM1 locus and five loci implicated in Crohn’s disease
-
COI: 1:CAS:528:DC%2BD1cXmsVejsb8%3D, PID: 18438406
-
Fisher SA et al (2008) Genetic determinants of ulcerative colitis include the ECM1 locus and five loci implicated in Crohn’s disease. Nat Genet 40:710–712
-
(2008)
Nat Genet
, vol.40
, pp. 710-712
-
-
Fisher, S.A.1
-
15
-
-
44349173654
-
Replication of signals from recent studies of Crohn’s disease identifies previously unknown disease loci for ulcerative colitis
-
COI: 1:CAS:528:DC%2BD1cXmsVejsbg%3D, PID: 18438405
-
Franke A et al (2008a) Replication of signals from recent studies of Crohn’s disease identifies previously unknown disease loci for ulcerative colitis. Nat Genet 40:713–715
-
(2008)
Nat Genet
, vol.40
, pp. 713-715
-
-
Franke, A.1
-
16
-
-
55049111426
-
Sequence variants in IL10, ARPC2 and multiple other loci contribute to ulcerative colitis susceptibility
-
COI: 1:CAS:528:DC%2BD1cXhtlSkurjI, PID: 18836448
-
Franke A et al (2008b) Sequence variants in IL10, ARPC2 and multiple other loci contribute to ulcerative colitis susceptibility. Nat Genet 40:1319–1323
-
(2008)
Nat Genet
, vol.40
, pp. 1319-1323
-
-
Franke, A.1
-
17
-
-
53049097281
-
Genome-wide association analysis in sarcoidosis and Crohn’s disease unravels a common susceptibility locus on 10p12.2
-
COI: 1:CAS:528:DC%2BD1cXht12lurrJ, PID: 18723019
-
Franke A et al (2008c) Genome-wide association analysis in sarcoidosis and Crohn’s disease unravels a common susceptibility locus on 10p12.2. Gastroenterology 135:1207–1215
-
(2008)
Gastroenterology
, vol.135
, pp. 1207-1215
-
-
Franke, A.1
-
18
-
-
77950296637
-
Genome-wide association study for ulcerative colitis identifies risk loci at 7q22 and 22q13 (IL17REL)
-
COI: 1:CAS:528:DC%2BC3cXjtFynu7c%3D, PID: 20228798
-
Franke A et al (2010a) Genome-wide association study for ulcerative colitis identifies risk loci at 7q22 and 22q13 (IL17REL). Nat Genet 42:292–294
-
(2010)
Nat Genet
, vol.42
, pp. 292-294
-
-
Franke, A.1
-
19
-
-
78649489009
-
Genome-wide meta-analysis increases to 71 the number of confirmed Crohn’s disease susceptibility loci
-
COI: 1:CAS:528:DC%2BC3cXhsVGhsrzO, PID: 21102463
-
Franke A et al (2010b) Genome-wide meta-analysis increases to 71 the number of confirmed Crohn’s disease susceptibility loci. Nat Genet 42:1118–1125
-
(2010)
Nat Genet
, vol.42
, pp. 1118-1125
-
-
Franke, A.1
-
20
-
-
77955362763
-
Analysis of SNPs with an effect on gene expression identifies UBE2L3 and BCL3 as potential new risk genes for Crohn’s disease
-
COI: 1:CAS:528:DC%2BC3cXpvVKjsbw%3D, PID: 20601676
-
Fransen K et al (2010) Analysis of SNPs with an effect on gene expression identifies UBE2L3 and BCL3 as potential new risk genes for Crohn’s disease. Hum Mol Genet 19:3482–3488
-
(2010)
Hum Mol Genet
, vol.19
, pp. 3482-3488
-
-
Fransen, K.1
-
21
-
-
79957604678
-
Principles for the post-GWAS functional characterization of cancer risk loci
-
COI: 1:CAS:528:DC%2BC3MXmsFyqtrk%3D, PID: 21614091
-
Freedman ML et al (2011) Principles for the post-GWAS functional characterization of cancer risk loci. Nat Genet 43:513–518
-
(2011)
Nat Genet
, vol.43
, pp. 513-518
-
-
Freedman, M.L.1
-
22
-
-
34548719076
-
Discovery of expression QTLs using large-scale transcriptional profiling in human lymphocytes
-
PID: 17873875
-
Goring HH et al (2007) Discovery of expression QTLs using large-scale transcriptional profiling in human lymphocytes. Nat Genet 39:1208–1216
-
(2007)
Nat Genet
, vol.39
, pp. 1208-1216
-
-
Goring, H.H.1
-
23
-
-
70350724550
-
Population genomics in a disease targeted primary cell model
-
COI: 1:CAS:528:DC%2BD1MXhsVSmtLjE, PID: 19654370
-
Grundberg E et al (2009) Population genomics in a disease targeted primary cell model. Genome Res 19:1942–1952
-
(2009)
Genome Res
, vol.19
, pp. 1942-1952
-
-
Grundberg, E.1
-
24
-
-
33846627302
-
A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1
-
COI: 1:CAS:528:DC%2BD2sXpvFerug%3D%3D, PID: 17200669
-
Hampe J et al (2007) A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1. Nat Genet 39:207–211
-
(2007)
Nat Genet
, vol.39
, pp. 207-211
-
-
Hampe, J.1
-
25
-
-
63449105856
-
Complex nature of SNP genotype effects on gene expression in primary human leucocytes
-
PID: 19128478
-
Heap GA et al (2009) Complex nature of SNP genotype effects on gene expression in primary human leucocytes. BMC Med Genomics 2:1
-
(2009)
BMC Med Genomics
, vol.2
, pp. 1
-
-
Heap, G.A.1
-
26
-
-
70649113728
-
Common variants at five new loci associated with early-onset inflammatory bowel disease
-
COI: 1:CAS:528:DC%2BD1MXhsVWlsLnO, PID: 19915574
-
Imielinski M et al (2009) Common variants at five new loci associated with early-onset inflammatory bowel disease. Nat Genet 41:1335–1340
-
(2009)
Nat Genet
, vol.41
, pp. 1335-1340
-
-
Imielinski, M.1
-
27
-
-
84889633349
-
Post genome-wide association studies functional characterization of prostate cancer risk loci
-
PID: 24564736
-
Jiang J, Cui W, Vongsangnak W, Hu G, Shen B (2013) Post genome-wide association studies functional characterization of prostate cancer risk loci. BMC Genomics 14(Suppl 8):S9
-
(2013)
BMC Genomics
, vol.14
, pp. 9
-
-
Jiang, J.1
Cui, W.2
Vongsangnak, W.3
Hu, G.4
Shen, B.5
-
28
-
-
84868336049
-
Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease
-
COI: 1:CAS:528:DC%2BC38Xhs1ajtbzP, PID: 23128233
-
Jostins L et al (2012) Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease. Nature 491:119–124
-
(2012)
Nature
, vol.491
, pp. 119-124
-
-
Jostins, L.1
-
29
-
-
0016669094
-
Evolution at two levels in humans and chimpanzees
-
COI: 1:CAS:528:DyaE2MXhs1Orur4%3D, PID: 1090005
-
King MC, Wilson AC (1975) Evolution at two levels in humans and chimpanzees. Science 188:107–116
-
(1975)
Science
, vol.188
, pp. 107-116
-
-
King, M.C.1
Wilson, A.C.2
-
30
-
-
52949091918
-
Loci on 20q13 and 21q22 are associated with pediatric-onset inflammatory bowel disease
-
COI: 1:CAS:528:DC%2BD1cXhtFKht7%2FL, PID: 18758464
-
Kugathasan S et al (2008) Loci on 20q13 and 21q22 are associated with pediatric-onset inflammatory bowel disease. Nat Genet 40:1211–1215
-
(2008)
Nat Genet
, vol.40
, pp. 1211-1215
-
-
Kugathasan, S.1
-
31
-
-
34247579326
-
Novel Crohn disease locus identified by genome-wide association maps to a gene desert on 5p13.1 and modulates expression of PTGER4
-
PID: 17447842
-
Libioulle C et al (2007) Novel Crohn disease locus identified by genome-wide association maps to a gene desert on 5p13.1 and modulates expression of PTGER4. PLoS Genet 3:e58
-
(2007)
PLoS Genet
, vol.3
, pp. 58
-
-
Libioulle, C.1
-
32
-
-
0035710746
-
Analysis of relative gene expression data using real-time quantitative PCR and the 2(−Delta Delta C(T)) Method
-
COI: 1:CAS:528:DC%2BD38XhtFelt7s%3D, PID: 11846609
-
Livak KJ, Schmittgen TD (2001) Analysis of relative gene expression data using real-time quantitative PCR and the 2(−Delta Delta C(T)) Method. Methods 25:402–408
-
(2001)
Methods
, vol.25
, pp. 402-408
-
-
Livak, K.J.1
Schmittgen, T.D.2
-
33
-
-
79151484424
-
The study of eQTL variations by RNA-seq: from SNPs to phenotypes
-
COI: 1:CAS:528:DC%2BC3MXhtlams7c%3D, PID: 21122937
-
Majewski J, Pastinen T (2011) The study of eQTL variations by RNA-seq: from SNPs to phenotypes. Trends Genet 27:72–79. doi:10.1016/j.tig.2010.10.006
-
(2011)
Trends Genet
, vol.27
, pp. 72-79
-
-
Majewski, J.1
Pastinen, T.2
-
34
-
-
0038724274
-
Large-scale identification and characterization of human genes that activate NF-kappaB and MAPK signaling pathways
-
COI: 1:CAS:528:DC%2BD3sXjvFaksLg%3D, PID: 12761501
-
Matsuda A et al (2003) Large-scale identification and characterization of human genes that activate NF-kappaB and MAPK signaling pathways. Oncogene 22:3307–3318
-
(2003)
Oncogene
, vol.22
, pp. 3307-3318
-
-
Matsuda, A.1
-
35
-
-
77950298959
-
Genome-wide association identifies multiple ulcerative colitis susceptibility loci
-
COI: 1:CAS:528:DC%2BC3cXjtFymsr8%3D, PID: 20228799
-
McGovern DP et al (2010a) Genome-wide association identifies multiple ulcerative colitis susceptibility loci. Nat Genet 42:332–337
-
(2010)
Nat Genet
, vol.42
, pp. 332-337
-
-
McGovern, D.P.1
-
36
-
-
77955398591
-
Fucosyltransferase 2 (FUT2) non-secretor status is associated with Crohn’s disease
-
COI: 1:CAS:528:DC%2BC3cXpvVKjsbg%3D, PID: 20570966
-
McGovern DP et al (2010b) Fucosyltransferase 2 (FUT2) non-secretor status is associated with Crohn’s disease. Hum Mol Genet 19:3468–3476
-
(2010)
Hum Mol Genet
, vol.19
, pp. 3468-3476
-
-
McGovern, D.P.1
-
37
-
-
78649395791
-
NKX2-3 and IRGM variants are associated with disease susceptibility to IBD in Eastern European patients
-
COI: 1:CAS:528:DC%2BC3cXhsVeqtLnN, PID: 21049557
-
Meggyesi N et al (2010) NKX2-3 and IRGM variants are associated with disease susceptibility to IBD in Eastern European patients. World J Gastroenterol 16:5233–5240
-
(2010)
World J Gastroenterol
, vol.16
, pp. 5233-5240
-
-
Meggyesi, N.1
-
38
-
-
79960332127
-
The use of genome-wide eQTL associations in lymphoblastoid cell lines to identify novel genetic pathways involved in complex traits
-
COI: 1:CAS:528:DC%2BC3MXhtVWqs7%2FK, PID: 21789213
-
Min JL et al (2011) The use of genome-wide eQTL associations in lymphoblastoid cell lines to identify novel genetic pathways involved in complex traits. PLoS One 6, e22070. doi:10.1371/journal.pone.0022070
-
(2011)
PLoS One
, vol.6
-
-
Min, J.L.1
-
39
-
-
4043128071
-
Genetic analysis of genome-wide variation in human gene expression
-
COI: 1:CAS:528:DC%2BD2cXmsVGls7k%3D, PID: 15269782
-
Morley M, Molony CM, Weber TM, Devlin JL, Ewens KG, Spielman RS, Cheung VG (2004) Genetic analysis of genome-wide variation in human gene expression. Nature 430:743–747
-
(2004)
Nature
, vol.430
, pp. 743-747
-
-
Morley, M.1
Molony, C.M.2
Weber, T.M.3
Devlin, J.L.4
Ewens, K.G.5
Spielman, R.S.6
Cheung, V.G.7
-
40
-
-
77951439152
-
Trait-associated SNPs are more likely to be eQTLs: annotation to enhance discovery from GWAS
-
PID: 20369019
-
Nicolae DL, Gamazon E, Zhang W, Duan S, Dolan ME, Cox NJ (2010) Trait-associated SNPs are more likely to be eQTLs: annotation to enhance discovery from GWAS. PLoS Genet 6, e1000888
-
(2010)
PLoS Genet
, vol.6
-
-
Nicolae, D.L.1
Gamazon, E.2
Zhang, W.3
Duan, S.4
Dolan, M.E.5
Cox, N.J.6
-
41
-
-
28844509940
-
The contribution of OCTN1/2 variants within the IBD5 locus to disease susceptibility and severity in Crohn's disease
-
Noble CL et al (2005) The contribution of OCTN1/2 variants within the IBD5 locus to disease susceptibility and severity in Crohn's disease. Gastroenterology 129:1854–1864
-
(2005)
Gastroenterology
, vol.129
, pp. 1854-1864
-
-
Noble, C.L.1
-
42
-
-
34347338690
-
Sequence variants in the autophagy gene IRGM and multiple other replicating loci contribute to Crohn’s disease susceptibility
-
COI: 1:CAS:528:DC%2BD2sXmvFKlsL4%3D, PID: 17554261
-
Parkes M et al (2007) Sequence variants in the autophagy gene IRGM and multiple other replicating loci contribute to Crohn’s disease susceptibility. Nat Genet 39:830–832
-
(2007)
Nat Genet
, vol.39
, pp. 830-832
-
-
Parkes, M.1
-
43
-
-
33750481513
-
Heritability and tissue specificity of expression quantitative trait loci
-
PID: 17054398
-
Petretto E et al (2006) Heritability and tissue specificity of expression quantitative trait loci. PLoS Genet 2, e172
-
(2006)
PLoS Genet
, vol.2
-
-
Petretto, E.1
-
44
-
-
35548958718
-
Genome-wide association study for Crohn’s disease in the Quebec Founder Population identifies multiple validated disease loci
-
COI: 1:CAS:528:DC%2BD2sXhtVOns7jF, PID: 17804789
-
Raelson JV et al (2007) Genome-wide association study for Crohn’s disease in the Quebec Founder Population identifies multiple validated disease loci. Proc Natl Acad Sci U S A 104:14747–14752
-
(2007)
Proc Natl Acad Sci U S A
, vol.104
, pp. 14747-14752
-
-
Raelson, J.V.1
-
45
-
-
80755189894
-
Haplotype in the IBD5 region is associated with refractory Crohn’s disease in Slovenian patients and modulates expression of the SLC22A5 gene
-
PID: 21695374
-
Repnik K, Potocnik U (2011) Haplotype in the IBD5 region is associated with refractory Crohn’s disease in Slovenian patients and modulates expression of the SLC22A5 gene. J Gastroenterol 46:1081–1091
-
(2011)
J Gastroenterol
, vol.46
, pp. 1081-1091
-
-
Repnik, K.1
Potocnik, U.2
-
46
-
-
34247554965
-
Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis
-
COI: 1:CAS:528:DC%2BD2sXksFersbk%3D, PID: 17435756
-
Rioux JD et al (2007) Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis. Nat Genet 39:596–604
-
(2007)
Nat Genet
, vol.39
, pp. 596-604
-
-
Rioux, J.D.1
-
47
-
-
0037456823
-
Genetics of gene expression surveyed in maize, mouse and man
-
COI: 1:CAS:528:DC%2BD3sXitFKmuro%3D, PID: 12646919
-
Schadt EE et al (2003) Genetics of gene expression surveyed in maize, mouse and man. Nature 422:297–302
-
(2003)
Nature
, vol.422
, pp. 297-302
-
-
Schadt, E.E.1
-
48
-
-
59149087627
-
Ulcerative colitis-risk loci on chromosomes 1p36 and 12q15 found by genome-wide association study
-
COI: 1:CAS:528:DC%2BD1MXhtVOqsQ%3D%3D, PID: 19122664
-
Silverberg MS et al (2009) Ulcerative colitis-risk loci on chromosomes 1p36 and 12q15 found by genome-wide association study. Nat Genet 41:216–220
-
(2009)
Nat Genet
, vol.41
, pp. 216-220
-
-
Silverberg, M.S.1
-
49
-
-
55449112185
-
Genome-wide associations of gene expression variation in humans
-
PID: 16362079
-
Stranger BE et al (2005) Genome-wide associations of gene expression variation in humans. PLoS Genet 1, e78
-
(2005)
PLoS Genet
, vol.1
-
-
Stranger, B.E.1
-
50
-
-
34548738566
-
Population genomics of human gene expression
-
COI: 1:CAS:528:DC%2BD2sXhtV2isL7N, PID: 17873874
-
Stranger BE et al (2007) Population genomics of human gene expression. Nat Genet 39:1217–1224
-
(2007)
Nat Genet
, vol.39
, pp. 1217-1224
-
-
Stranger, B.E.1
-
51
-
-
84969213492
-
Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls
-
The Wellcome Trust Case Control Consortium (2007) Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls. Nature 447:661–678
-
(2007)
Nature
, vol.447
, pp. 661-678
-
-
-
52
-
-
61549115034
-
Diverse genome-wide association studies associate the IL12/IL23 pathway with Crohn Disease
-
COI: 1:CAS:528:DC%2BD1MXmvFClsrk%3D, PID: 19249008
-
Wang K et al (2009) Diverse genome-wide association studies associate the IL12/IL23 pathway with Crohn Disease. Am J Hum Genet 84:399–405
-
(2009)
Am J Hum Genet
, vol.84
, pp. 399-405
-
-
Wang, K.1
-
53
-
-
84930817087
-
Cell specific eQTL analysis without sorting cells
-
PID: 25955312
-
Westra HJ et al (2015) Cell specific eQTL analysis without sorting cells. PLoS Genet 11, e1005223. doi:10.1371/journal.pgen.1005223
-
(2015)
PLoS Genet
, vol.11
-
-
Westra, H.J.1
-
54
-
-
27944464550
-
Single nucleotide polymorphisms in TNFSF15 confer susceptibility to Crohn’s disease
-
COI: 1:CAS:528:DC%2BD2MXhtFymtbvP, PID: 16221758
-
Yamazaki K et al (2005) Single nucleotide polymorphisms in TNFSF15 confer susceptibility to Crohn’s disease. Hum Mol Genet 14:3499–3506
-
(2005)
Hum Mol Genet
, vol.14
, pp. 3499-3506
-
-
Yamazaki, K.1
|