Integrating GRK2 and NFkappaB in the Pathophysiology of Cardiac Hypertrophy

Journal of Cardiovascular Translational Research

Volumn 8, Issue 8, 2015, Pages 493-502

  Sorriento, Daniela ^a   Santulli, Gaetano ^b   Franco, Antonietta ^c   Cipolletta, Ersilia ^d   Napolitano, Luigi ^c   Gambardella, Jessica ^d   Gomez Monterrey, Isabel ^c   Campiglia, Pietro ^e   Trimarco, Bruno ^c   Iaccarino, Guido ^d   Ciccarelli, Michele ^d  

^a CNR   (Italy)

^b Columbia University Medical Center ^*  (United States)

^c UNIVERSITY OF NAPLES FEDERICO II   (Italy)

^d UNIVERSITY OF SALERNO   (Italy)

^e UNIVERSITY OF SALERNO   (Italy)

Author keywords

ANF; GRK2; Left ventricular hypertrophy; NF B

Indexed keywords

ATRIAL NATRIURETIC FACTOR; G PROTEIN COUPLED RECEPTOR KINASE 2; I KAPPA B ALPHA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; PHENYLEPHRINE; ADRBK1 PROTEIN, MOUSE; ADRBK1 PROTEIN, RAT; PROTEIN KINASE INHIBITOR;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME INHIBITION; GENE OVEREXPRESSION; GENETIC TRANSCRIPTION; GRK2 GENE; HEART LEFT VENTRICLE HYPERTROPHY; HEART MUSCLE CELL; HEART VENTRICLE HYPERTROPHY; IN VIVO STUDY; MALE; MYOBLAST; NFKAPPAB GENE; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; RAT; UPREGULATION; ANIMAL; ANTAGONISTS AND INHIBITORS; CARDIAC MUSCLE CELL; CELL LINE; CHEMICALLY INDUCED; DEFICIENCY; DISEASE MODEL; DRUG EFFECTS; ENZYMOLOGY; GENE SILENCING; GENETIC TRANSDUCTION; GENETIC TRANSFECTION; GENETICS; HYPERTROPHY, LEFT VENTRICULAR; KNOCKOUT MOUSE; METABOLISM; MUTATION; PATHOLOGY; PHOSPHORYLATION; SIGNAL TRANSDUCTION; SPONTANEOUSLY HYPERTENSIVE RAT; WISTAR KYOTO RAT;

ANIMALS; ATRIAL NATRIURETIC FACTOR; CELL LINE; DISEASE MODELS, ANIMAL; G-PROTEIN-COUPLED RECEPTOR KINASE 2; GENE KNOCKDOWN TECHNIQUES; HYPERTROPHY, LEFT VENTRICULAR; MALE; MICE, KNOCKOUT; MUTATION; MYOCYTES, CARDIAC; NF-KAPPA B; PHENYLEPHRINE; PHOSPHORYLATION; PROTEIN KINASE INHIBITORS; RATS; RATS, INBRED SHR; RATS, INBRED WKY; SIGNAL TRANSDUCTION; TRANSDUCTION, GENETIC; TRANSFECTION;

EID: 84947493668     PISSN: 19375387     EISSN: 19375395     Source Type: Journal    
DOI: 10.1007/s12265-015-9646-0     Document Type: Article

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