Early disruption of glial communication via connexin gap junction in multiple sclerosis, Baló's disease and neuromyelitis optica

Neuropathology

Volumn 35, Issue 5, 2015, Pages 469-480

  Masaki, Katsuhisa ^a  

^a KYUSHU UNIVERSITY   (Japan)

Author keywords

Aquaporin 4; Bal 's disease; Connexin; Multiple sclerosis; Neuromyelitis optica

Indexed keywords

CONNEXIN 30; CONNEXIN 32; CONNEXIN 43; CONNEXIN 47; GAP JUNCTION PROTEIN; MYELIN;

ARTICLE; ASTROCYTE; BRAIN DAMAGE; CELL COMMUNICATION; CELL HETEROGENEITY; DISEASE COURSE; GAP JUNCTION; GENE MUTATION; GLIA; GRAY MATTER; HEREDITARY MOTOR SENSORY NEUROPATHY; HOMEOSTASIS; HUMAN; MULTIPLE SCLEROSIS; MYELOOPTIC NEUROPATHY; NERVE DEGENERATION; NONHUMAN; OLIGODENDROGLIA; PELIZAEUS MERZBACHER DISEASE; PHENOTYPE; PRIORITY JOURNAL; PROTEIN DEPLETION; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; SYNCYTIUM; METABOLISM; PATHOLOGY; PHYSIOLOGY; SCHILDER DISEASE;

CELL COMMUNICATION; CONNEXINS; DIFFUSE CEREBRAL SCLEROSIS OF SCHILDER; GAP JUNCTIONS; HUMANS; MULTIPLE SCLEROSIS; NEUROGLIA; NEUROMYELITIS OPTICA;

EID: 84942822024     PISSN: 09196544     EISSN: 14401789     Source Type: Journal    
DOI: 10.1111/neup.12211     Document Type: Article

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