-
1
-
-
63349088070
-
Endoplasmic reticulum stress in the kidney as a novel mediator of kidney injury
-
Inagi R. Endoplasmic reticulum stress in the kidney as a novel mediator of kidney injury. Nephron Exp. Nephrol. 2009, 112:e1-e9.
-
(2009)
Nephron Exp. Nephrol.
, vol.112
, pp. e1-e9
-
-
Inagi, R.1
-
2
-
-
1842843855
-
Roles of CHOP/GADD153 in endoplasmic reticulum stress
-
Oyadomari S., Mori M. Roles of CHOP/GADD153 in endoplasmic reticulum stress. Cell Death Differ. 2004, 11:381-389.
-
(2004)
Cell Death Differ.
, vol.11
, pp. 381-389
-
-
Oyadomari, S.1
Mori, M.2
-
3
-
-
84916602072
-
Endoplasmic reticulum stress and cancer
-
Yadav R.K., Chae S.W., Kim H.R., Chae H.J. Endoplasmic reticulum stress and cancer. J. Cancer Prev. 2014, 19:75-88.
-
(2014)
J. Cancer Prev.
, vol.19
, pp. 75-88
-
-
Yadav, R.K.1
Chae, S.W.2
Kim, H.R.3
Chae, H.J.4
-
4
-
-
78650511500
-
Ischemic postconditioning protects brain from ischemia/reperfusion injury by attenuating endoplasmic reticulum stress-induced apoptosis through PI3K-Akt pathway
-
Yuan Y., Guo Q., Ye Z., Pingping X., Wang N., Song Z. Ischemic postconditioning protects brain from ischemia/reperfusion injury by attenuating endoplasmic reticulum stress-induced apoptosis through PI3K-Akt pathway. Brain Res. 2011, 1367:85-93.
-
(2011)
Brain Res.
, vol.1367
, pp. 85-93
-
-
Yuan, Y.1
Guo, Q.2
Ye, Z.3
Pingping, X.4
Wang, N.5
Song, Z.6
-
5
-
-
84882807441
-
Activation of ERK accelerates repair of renal tubular epithelial cells, whereas it inhibits progression of fibrosis following ischemia/reperfusion injury
-
Jang H.S., Han S.J., Kim J.I., Lee S., Lipschutz J.H., Park K.M. Activation of ERK accelerates repair of renal tubular epithelial cells, whereas it inhibits progression of fibrosis following ischemia/reperfusion injury. Biochim. Biophys. Acta 2013, 1832:1998-2008.
-
(2013)
Biochim. Biophys. Acta
, vol.1832
, pp. 1998-2008
-
-
Jang, H.S.1
Han, S.J.2
Kim, J.I.3
Lee, S.4
Lipschutz, J.H.5
Park, K.M.6
-
6
-
-
0035853676
-
Prevention of kidney ischemia/reperfusion-induced functional injury and JNK, p38, and MAPK kinase activation by remote ischemic pretreatment
-
Park K.M., Chen A., Bonventre J.V. Prevention of kidney ischemia/reperfusion-induced functional injury and JNK, p38, and MAPK kinase activation by remote ischemic pretreatment. J. Biol. Chem. 2001, 276:11870-11876.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 11870-11876
-
-
Park, K.M.1
Chen, A.2
Bonventre, J.V.3
-
8
-
-
33644515957
-
Cytoprotective gene bi-1 is required for intrinsic protection from endoplasmic reticulum stress and ischemia-reperfusion injury
-
Bailly-Maitre B., Fondevila C., Kaldas F., Droin N., Luciano F., Ricci J.E., Croxton R., Krajewska M., Zapata J.M., Kupiec-Weglinski J.W., Farmer D., Reed J.C. Cytoprotective gene bi-1 is required for intrinsic protection from endoplasmic reticulum stress and ischemia-reperfusion injury. Proc. Natl. Acad. Sci. U. S. A. 2006, 103:2809-2814.
-
(2006)
Proc. Natl. Acad. Sci. U. S. A.
, vol.103
, pp. 2809-2814
-
-
Bailly-Maitre, B.1
Fondevila, C.2
Kaldas, F.3
Droin, N.4
Luciano, F.5
Ricci, J.E.6
Croxton, R.7
Krajewska, M.8
Zapata, J.M.9
Kupiec-Weglinski, J.W.10
Farmer, D.11
Reed, J.C.12
-
9
-
-
84891505671
-
Ischemia-reperfusion induces renal tubule pyroptosis via the CHOP-caspase-11 pathway
-
Yang J.R., Yao F.H., Zhang J.G., Ji Z.Y., Li K.L., Zhan J., Tong Y.N., Lin L.R., He Y.N. Ischemia-reperfusion induces renal tubule pyroptosis via the CHOP-caspase-11 pathway. Am. J. Physiol. Ren. Physiol. 2014, 306:F75-F84.
-
(2014)
Am. J. Physiol. Ren. Physiol.
, vol.306
, pp. F75-F84
-
-
Yang, J.R.1
Yao, F.H.2
Zhang, J.G.3
Ji, Z.Y.4
Li, K.L.5
Zhan, J.6
Tong, Y.N.7
Lin, L.R.8
He, Y.N.9
-
10
-
-
84930325694
-
Angiotensin II removes kidney resistance conferred by ischemic preconditioning
-
Jang H.S., Kim J.I., Kim J., Park J.W., Park K.M. Angiotensin II removes kidney resistance conferred by ischemic preconditioning. BioMed Res. Int. 2014, 2014:602149.
-
(2014)
BioMed Res. Int.
, vol.2014
, pp. 602149
-
-
Jang, H.S.1
Kim, J.I.2
Kim, J.3
Park, J.W.4
Park, K.M.5
-
11
-
-
84948428212
-
CCAAT-enhancer-binding protein homologous protein deficiency attenuates oxidative stress and renal ischemia-reperfusion injury
-
Chen B.L., Sheu M.L., Tsai K.S., Lan K.C., Guan S.S., Wu C.T., Chen L.P., Hung K.Y., Huang J.W., Chiang C.K., Liu S.H. CCAAT-enhancer-binding protein homologous protein deficiency attenuates oxidative stress and renal ischemia-reperfusion injury. Antioxid. Redox Signal. 2014, 10.1089/ars.2013.5768.
-
(2014)
Antioxid. Redox Signal.
-
-
Chen, B.L.1
Sheu, M.L.2
Tsai, K.S.3
Lan, K.C.4
Guan, S.S.5
Wu, C.T.6
Chen, L.P.7
Hung, K.Y.8
Huang, J.W.9
Chiang, C.K.10
Liu, S.H.11
-
12
-
-
77955173081
-
Ablation of C/EBP homologous protein attenuates endoplasmic reticulum-mediated apoptosis and cardiac dysfunction induced by pressure overload
-
Fu H.Y., Okada K., Liao Y., Tsukamoto O., Isomura T., Asai M., Sawada T., Okuda K., Asano Y., Sanada S., Asanuma H., Asakura M., Takashima S., Komuro I., Kitakaze M., Minamino T. Ablation of C/EBP homologous protein attenuates endoplasmic reticulum-mediated apoptosis and cardiac dysfunction induced by pressure overload. Circulation 2010, 122:361-369.
-
(2010)
Circulation
, vol.122
, pp. 361-369
-
-
Fu, H.Y.1
Okada, K.2
Liao, Y.3
Tsukamoto, O.4
Isomura, T.5
Asai, M.6
Sawada, T.7
Okuda, K.8
Asano, Y.9
Sanada, S.10
Asanuma, H.11
Asakura, M.12
Takashima, S.13
Komuro, I.14
Kitakaze, M.15
Minamino, T.16
-
13
-
-
84859740294
-
Emerging evidence for endoplasmic reticulum stress in the pathogenesis of idiopathic pulmonary fibrosis
-
Tanjore H., Blackwell T.S., Lawson W.E. Emerging evidence for endoplasmic reticulum stress in the pathogenesis of idiopathic pulmonary fibrosis. Am. J. Physiol. Lung Cell. Mol. Physiol. 2012, 302:L721-L729.
-
(2012)
Am. J. Physiol. Lung Cell. Mol. Physiol.
, vol.302
, pp. L721-L729
-
-
Tanjore, H.1
Blackwell, T.S.2
Lawson, W.E.3
-
14
-
-
84920263184
-
Role of C/EBP homologous protein in retinal ganglion cell death after ischemia/reperfusion injury
-
Nashine S., Liu Y., Kim B.J., Clark A.F., Pang I.H. Role of C/EBP homologous protein in retinal ganglion cell death after ischemia/reperfusion injury. Invest. Ophthalmol. Vis. Sci. 2014, 56:221-231.
-
(2014)
Invest. Ophthalmol. Vis. Sci.
, vol.56
, pp. 221-231
-
-
Nashine, S.1
Liu, Y.2
Kim, B.J.3
Clark, A.F.4
Pang, I.H.5
-
16
-
-
84872905488
-
Berberine protects human renal proximal tubular cells from hypoxia/reoxygenation injury via inhibiting endoplasmic reticulum and mitochondrial stress pathways
-
Yu W., Sheng M., Xu R., Yu J., Cui K., Tong J., Shi L., Ren H., Du H. Berberine protects human renal proximal tubular cells from hypoxia/reoxygenation injury via inhibiting endoplasmic reticulum and mitochondrial stress pathways. J. Transl. Med. 2013, 11:24.
-
(2013)
J. Transl. Med.
, vol.11
, pp. 24
-
-
Yu, W.1
Sheng, M.2
Xu, R.3
Yu, J.4
Cui, K.5
Tong, J.6
Shi, L.7
Ren, H.8
Du, H.9
-
17
-
-
84898672741
-
Proximal tubule toll-like receptor 4 expression linked to inflammation and apoptosis following hypoxia/reoxygenation injury
-
Liu X.J., Tan Y., Geng Y.Q., Wang Z., Ye J.H., Yin X.Y., Fu B. Proximal tubule toll-like receptor 4 expression linked to inflammation and apoptosis following hypoxia/reoxygenation injury. Am. J. Nephrol. 2014, 39:337-347.
-
(2014)
Am. J. Nephrol.
, vol.39
, pp. 337-347
-
-
Liu, X.J.1
Tan, Y.2
Geng, Y.Q.3
Wang, Z.4
Ye, J.H.5
Yin, X.Y.6
Fu, B.7
-
18
-
-
84904431236
-
Baicalin ameliorates H2O2 induced cytotoxicity in HK-2 cells through the inhibition of ER stress and the activation of Nrf2 signaling
-
Lin M., Li L., Zhang Y., Zheng L., Xu M., Rong R., Zhu T. Baicalin ameliorates H2O2 induced cytotoxicity in HK-2 cells through the inhibition of ER stress and the activation of Nrf2 signaling. Int. J. Mol. Sci. 2014, 15:12507-12522.
-
(2014)
Int. J. Mol. Sci.
, vol.15
, pp. 12507-12522
-
-
Lin, M.1
Li, L.2
Zhang, Y.3
Zheng, L.4
Xu, M.5
Rong, R.6
Zhu, T.7
-
19
-
-
34250308322
-
Apoptosis: a review of programmed cell death
-
Elmore S. Apoptosis: a review of programmed cell death. Toxicol. Pathol. 2007, 35:495-516.
-
(2007)
Toxicol. Pathol.
, vol.35
, pp. 495-516
-
-
Elmore, S.1
-
20
-
-
41149152733
-
How do BCL-2 proteins induce mitochondrial outer membrane permeabilization?
-
Chipuk J.E., Green D.R. How do BCL-2 proteins induce mitochondrial outer membrane permeabilization?. Trends Cell Biol. 2008, 18:157-164.
-
(2008)
Trends Cell Biol.
, vol.18
, pp. 157-164
-
-
Chipuk, J.E.1
Green, D.R.2
-
22
-
-
0034523818
-
Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c
-
Korsmeyer S.J., Wei M.C., Saito M., Weiler S., Oh K.J., Schlesinger P.H. Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c. Cell Death Differ. 2000, 7:1166-1173.
-
(2000)
Cell Death Differ.
, vol.7
, pp. 1166-1173
-
-
Korsmeyer, S.J.1
Wei, M.C.2
Saito, M.3
Weiler, S.4
Oh, K.J.5
Schlesinger, P.H.6
-
23
-
-
0035736259
-
Organelle-specific initiation of cell death pathways
-
Ferri K.F., Kroemer G. Organelle-specific initiation of cell death pathways. Nat. Cell Biol. 2001, 3:E255-E263.
-
(2001)
Nat. Cell Biol.
, vol.3
, pp. E255-E263
-
-
Ferri, K.F.1
Kroemer, G.2
-
24
-
-
33646373219
-
Endoplasmic reticulum stress-induced apoptosis: multiple pathways and activation of p53-up-regulated modulator of apoptosis (PUMA) and NOXA by p53
-
Li J., Lee B., Lee A.S. Endoplasmic reticulum stress-induced apoptosis: multiple pathways and activation of p53-up-regulated modulator of apoptosis (PUMA) and NOXA by p53. J. Biol. Chem. 2006, 281:7260-7270.
-
(2006)
J. Biol. Chem.
, vol.281
, pp. 7260-7270
-
-
Li, J.1
Lee, B.2
Lee, A.S.3
-
25
-
-
41149106798
-
Effects of targeted Bcl-2 expression in mitochondria or endoplasmic reticulum on renal tubular cell apoptosis
-
Bhatt K., Feng L., Pabla N., Liu K., Smith S., Dong Z. Effects of targeted Bcl-2 expression in mitochondria or endoplasmic reticulum on renal tubular cell apoptosis. Am. J. Physiol. Renal Physiol. 2008, 294:F499-F507.
-
(2008)
Am. J. Physiol. Renal Physiol.
, vol.294
, pp. F499-F507
-
-
Bhatt, K.1
Feng, L.2
Pabla, N.3
Liu, K.4
Smith, S.5
Dong, Z.6
-
26
-
-
84906569010
-
Bcl-xL protein protects from C/EBP homologous protein (CHOP)-dependent apoptosis during plasma cell differentiation
-
Gaudette B.T., Iwakoshi N.N., Boise L.H. Bcl-xL protein protects from C/EBP homologous protein (CHOP)-dependent apoptosis during plasma cell differentiation. J. Biol. Chem. 2014, 289:23629-23640.
-
(2014)
J. Biol. Chem.
, vol.289
, pp. 23629-23640
-
-
Gaudette, B.T.1
Iwakoshi, N.N.2
Boise, L.H.3
-
27
-
-
0032522738
-
IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspases
-
Deveraux Q.L., Roy N., Stennicke H.R., Van Arsdale T., Zhou Q., Srinivasula S.M., Alnemri E.S., Salvesen G.S., Reed J.C. IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspases. EMBO J. 1998, 17:2215-2223.
-
(1998)
EMBO J.
, vol.17
, pp. 2215-2223
-
-
Deveraux, Q.L.1
Roy, N.2
Stennicke, H.R.3
Van Arsdale, T.4
Zhou, Q.5
Srinivasula, S.M.6
Alnemri, E.S.7
Salvesen, G.S.8
Reed, J.C.9
-
28
-
-
77952706582
-
Omi/HtrA2 protease is associated with tubular cell apoptosis and fibrosis induced by unilateral ureteral obstruction
-
Kim J., Kim D.S., Park M.J., Cho H.J., Zervos A.S., Bonventre J.V., Park K.M. Omi/HtrA2 protease is associated with tubular cell apoptosis and fibrosis induced by unilateral ureteral obstruction. Am. J. Physiol. Renal Physiol. 2010, 298:F1332-F1340.
-
(2010)
Am. J. Physiol. Renal Physiol.
, vol.298
, pp. F1332-F1340
-
-
Kim, J.1
Kim, D.S.2
Park, M.J.3
Cho, H.J.4
Zervos, A.S.5
Bonventre, J.V.6
Park, K.M.7
-
29
-
-
84916594880
-
Heat shock protein 72 suppresses apoptosis by increasing the stability of X-linked inhibitor of apoptosis protein in renal ischemia/reperfusion injury
-
Zhang B., Rong R., Li H., Peng X., Xiong L., Wang Y., Yu X., Mao H. Heat shock protein 72 suppresses apoptosis by increasing the stability of X-linked inhibitor of apoptosis protein in renal ischemia/reperfusion injury. Mol. Med. Rep. 2015, 11:1793-1799.
-
(2015)
Mol. Med. Rep.
, vol.11
, pp. 1793-1799
-
-
Zhang, B.1
Rong, R.2
Li, H.3
Peng, X.4
Xiong, L.5
Wang, Y.6
Yu, X.7
Mao, H.8
-
30
-
-
84899672694
-
Translational and posttranslational regulation of XIAP by eIF2alpha and ATF4 promotes ER stress-induced cell death during the unfolded protein response
-
Hiramatsu N., Messah C., Han J., LaVail M.M., Kaufman R.J., Lin J.H. Translational and posttranslational regulation of XIAP by eIF2alpha and ATF4 promotes ER stress-induced cell death during the unfolded protein response. Mol. Biol. Cell 2014, 25:1411-1420.
-
(2014)
Mol. Biol. Cell
, vol.25
, pp. 1411-1420
-
-
Hiramatsu, N.1
Messah, C.2
Han, J.3
LaVail, M.M.4
Kaufman, R.J.5
Lin, J.H.6
-
31
-
-
2342553892
-
Mitochondrial p53 activates Bak and causes disruption of a Bak-Mcl1 complex
-
Leu J.I., Dumont P., Hafey M., Murphy M.E., George D.L. Mitochondrial p53 activates Bak and causes disruption of a Bak-Mcl1 complex. Nat. Cell Biol. 2004, 6:443-450.
-
(2004)
Nat. Cell Biol.
, vol.6
, pp. 443-450
-
-
Leu, J.I.1
Dumont, P.2
Hafey, M.3
Murphy, M.E.4
George, D.L.5
-
32
-
-
22244464818
-
Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins
-
Willis S.N., Chen L., Dewson G., Wei A., Naik E., Fletcher J.I., Adams J.M., Huang D.C. Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins. Genes Dev. 2005, 19:1294-1305.
-
(2005)
Genes Dev.
, vol.19
, pp. 1294-1305
-
-
Willis, S.N.1
Chen, L.2
Dewson, G.3
Wei, A.4
Naik, E.5
Fletcher, J.I.6
Adams, J.M.7
Huang, D.C.8
-
33
-
-
71749119515
-
Proteasome inhibitors prevent cisplatin-induced mitochondrial release of apoptosis-inducing factor and markedly ameliorate cisplatin nephrotoxicity
-
Liu L., Yang C., Herzog C., Seth R., Kaushal G.P. Proteasome inhibitors prevent cisplatin-induced mitochondrial release of apoptosis-inducing factor and markedly ameliorate cisplatin nephrotoxicity. Biochem. Pharmacol. 2010, 79:137-146.
-
(2010)
Biochem. Pharmacol.
, vol.79
, pp. 137-146
-
-
Liu, L.1
Yang, C.2
Herzog, C.3
Seth, R.4
Kaushal, G.P.5
-
34
-
-
0038661148
-
Prevention of apoptotic and necrotic cell death, caspase-3 activation, and renal dysfunction by melatonin after ischemia/reperfusion
-
Kunduzova O.R., Escourrou G., Seguelas M.H., Delagrange P., De La Farge F., Cambon C., Parini A. Prevention of apoptotic and necrotic cell death, caspase-3 activation, and renal dysfunction by melatonin after ischemia/reperfusion. FASEB J. Off. Publ. Fed. Am. Soc. Exp. Biol. 2003, 17:872-874.
-
(2003)
FASEB J. Off. Publ. Fed. Am. Soc. Exp. Biol.
, vol.17
, pp. 872-874
-
-
Kunduzova, O.R.1
Escourrou, G.2
Seguelas, M.H.3
Delagrange, P.4
De La Farge, F.5
Cambon, C.6
Parini, A.7
-
35
-
-
0035845568
-
Nitric oxide-induced apoptosis in pancreatic beta cells is mediated by the endoplasmic reticulum stress pathway
-
Oyadomari S., Takeda K., Takiguchi M., Gotoh T., Matsumoto M., Wada I., Akira S., Araki E., Mori M. Nitric oxide-induced apoptosis in pancreatic beta cells is mediated by the endoplasmic reticulum stress pathway. Proc. Natl. Acad. Sci. U. S. A. 2001, 98:10845-10850.
-
(2001)
Proc. Natl. Acad. Sci. U. S. A.
, vol.98
, pp. 10845-10850
-
-
Oyadomari, S.1
Takeda, K.2
Takiguchi, M.3
Gotoh, T.4
Matsumoto, M.5
Wada, I.6
Akira, S.7
Araki, E.8
Mori, M.9
-
36
-
-
0032054744
-
CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum
-
Zinszner H., Kuroda M., Wang X., Batchvarova N., Lightfoot R.T., Remotti H., Stevens J.L., Ron D. CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum. Genes Dev. 1998, 12:982-995.
-
(1998)
Genes Dev.
, vol.12
, pp. 982-995
-
-
Zinszner, H.1
Kuroda, M.2
Wang, X.3
Batchvarova, N.4
Lightfoot, R.T.5
Remotti, H.6
Stevens, J.L.7
Ron, D.8
-
37
-
-
84908167333
-
C/EBP homologous protein inhibits tissue repair in response to gut injury and is inversely regulated with chronic inflammation
-
Waldschmitt N., Berger E., Rath E., Sartor R.B., Weigmann B., Heikenwalder M., Gerhard M., Janssen K.P., Haller D. C/EBP homologous protein inhibits tissue repair in response to gut injury and is inversely regulated with chronic inflammation. Mucosal Immunol. 2014, 7:1452-1466.
-
(2014)
Mucosal Immunol.
, vol.7
, pp. 1452-1466
-
-
Waldschmitt, N.1
Berger, E.2
Rath, E.3
Sartor, R.B.4
Weigmann, B.5
Heikenwalder, M.6
Gerhard, M.7
Janssen, K.P.8
Haller, D.9
-
38
-
-
79955597257
-
C/EBP homologous protein deficiency attenuates myocardial reperfusion injury by inhibiting myocardial apoptosis and inflammation
-
Miyazaki Y., Kaikita K., Endo M., Horio E., Miura M., Tsujita K., Hokimoto S., Yamamuro M., Iwawaki T., Gotoh T., Ogawa H., Oike Y. C/EBP homologous protein deficiency attenuates myocardial reperfusion injury by inhibiting myocardial apoptosis and inflammation. Arterioscler. Thromb. Vasc. Biol. 2011, 31:1124-1132.
-
(2011)
Arterioscler. Thromb. Vasc. Biol.
, vol.31
, pp. 1124-1132
-
-
Miyazaki, Y.1
Kaikita, K.2
Endo, M.3
Horio, E.4
Miura, M.5
Tsujita, K.6
Hokimoto, S.7
Yamamuro, M.8
Iwawaki, T.9
Gotoh, T.10
Ogawa, H.11
Oike, Y.12
|