-
1
-
-
34447559506
-
Community-based incidence of acute renal failure
-
Hsu C.Y., McCulloch C.E., Fan D., Ordonez J.D., Chertow G.M., Go A.S. Community-based incidence of acute renal failure. Kidney Int. 2007, 72:208-212.
-
(2007)
Kidney Int.
, vol.72
, pp. 208-212
-
-
Hsu, C.Y.1
McCulloch, C.E.2
Fan, D.3
Ordonez, J.D.4
Chertow, G.M.5
Go, A.S.6
-
2
-
-
34447628673
-
RIFLE criteria for acute kidney injury are associated with hospital mortality in critically ill patients: a cohort analysis
-
Hoste E.A., Clermont G., Kersten A., Venkataraman R., Angus D.C., De Bacquer D., Kellum J.A. RIFLE criteria for acute kidney injury are associated with hospital mortality in critically ill patients: a cohort analysis. Crit. Care 2006, 10:R73.
-
(2006)
Crit. Care
, vol.10
-
-
Hoste, E.A.1
Clermont, G.2
Kersten, A.3
Venkataraman, R.4
Angus, D.C.5
De Bacquer, D.6
Kellum, J.A.7
-
3
-
-
33644874086
-
Acute kidney injury, mortality, length of stay, and costs in hospitalized patients
-
Chertow G.M., Burdick E., Honour M., Bonventre J.V., Bates D.W. Acute kidney injury, mortality, length of stay, and costs in hospitalized patients. J. Am. Soc. Nephrol. 2005, 16:3365-3370.
-
(2005)
J. Am. Soc. Nephrol.
, vol.16
, pp. 3365-3370
-
-
Chertow, G.M.1
Burdick, E.2
Honour, M.3
Bonventre, J.V.4
Bates, D.W.5
-
5
-
-
80555157523
-
Cellular pathophysiology of ischemic acute kidney injury
-
Bonventre J.V., Yang L. Cellular pathophysiology of ischemic acute kidney injury. J. Clin. Invest. 2011, 121:4210-4221.
-
(2011)
J. Clin. Invest.
, vol.121
, pp. 4210-4221
-
-
Bonventre, J.V.1
Yang, L.2
-
6
-
-
77951592988
-
Reactive oxygen species differently regulate renal tubular epithelial and interstitial cell proliferation after ischemia and reperfusion injury
-
Kim J., Jung K.J., Park K.M. Reactive oxygen species differently regulate renal tubular epithelial and interstitial cell proliferation after ischemia and reperfusion injury. Am. J. Physiol. Renal Physiol. 2010, 298:F1118-F1129.
-
(2010)
Am. J. Physiol. Renal Physiol.
, vol.298
-
-
Kim, J.1
Jung, K.J.2
Park, K.M.3
-
7
-
-
76749088358
-
Pathological roles of MAPK signaling pathways in human diseases
-
Kim E.K., Choi E.J. Pathological roles of MAPK signaling pathways in human diseases. Biochim. Biophys. Acta 2010, 1802:396-405.
-
(2010)
Biochim. Biophys. Acta
, vol.1802
, pp. 396-405
-
-
Kim, E.K.1
Choi, E.J.2
-
9
-
-
0037032817
-
Mitogen-activated protein kinase pathways mediated by ERK, JNK, and p38 protein kinases
-
Johnson G.L., Lapadat R. Mitogen-activated protein kinase pathways mediated by ERK, JNK, and p38 protein kinases. Science 2002, 298:1911-1912.
-
(2002)
Science
, vol.298
, pp. 1911-1912
-
-
Johnson, G.L.1
Lapadat, R.2
-
10
-
-
0035853676
-
Prevention of kidney ischemia/reperfusion-induced functional injury and JNK, p38, and MAPK kinase activation by remote ischemic pretreatment
-
Park K.M., Chen A., Bonventre J.V. Prevention of kidney ischemia/reperfusion-induced functional injury and JNK, p38, and MAPK kinase activation by remote ischemic pretreatment. J. Biol. Chem. 2001, 276:11870-11876.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 11870-11876
-
-
Park, K.M.1
Chen, A.2
Bonventre, J.V.3
-
11
-
-
84862185871
-
Bone marrow derived cells and reactive oxygen species in hypertrophy of contralateral kidney of transient unilateral renal ischemia-induced mouse
-
Jang H.S., Kim J.I., Kim J., Na Y.K., Park J.W., Park K.M. Bone marrow derived cells and reactive oxygen species in hypertrophy of contralateral kidney of transient unilateral renal ischemia-induced mouse. Free Radic. Res. 2012, 46:903-911.
-
(2012)
Free Radic. Res.
, vol.46
, pp. 903-911
-
-
Jang, H.S.1
Kim, J.I.2
Kim, J.3
Na, Y.K.4
Park, J.W.5
Park, K.M.6
-
12
-
-
84868580063
-
Previous ischemia and reperfusion injury results in resistance of the kidney against subsequent ischemia and reperfusion insult in mice; a role for the Akt signal pathway
-
Jang H.S., Kim J., Kim K.Y., Kim J.I., Cho M.H., Park K.M. Previous ischemia and reperfusion injury results in resistance of the kidney against subsequent ischemia and reperfusion insult in mice; a role for the Akt signal pathway. Nephrol. Dial. Transplant. 2012, 27:3762-3770.
-
(2012)
Nephrol. Dial. Transplant.
, vol.27
, pp. 3762-3770
-
-
Jang, H.S.1
Kim, J.2
Kim, K.Y.3
Kim, J.I.4
Cho, M.H.5
Park, K.M.6
-
13
-
-
68049112547
-
Reactive oxygen species/oxidative stress contributes to progression of kidney fibrosis following transient ischemic injury in mice
-
Kim J., Seok Y.M., Jung K.J., Park K.M. Reactive oxygen species/oxidative stress contributes to progression of kidney fibrosis following transient ischemic injury in mice. Am. J. Physiol. Renal Physiol. 2009, 297:F461-F470.
-
(2009)
Am. J. Physiol. Renal Physiol.
, vol.297
-
-
Kim, J.1
Seok, Y.M.2
Jung, K.J.3
Park, K.M.4
-
14
-
-
40049107749
-
Infiltrated macrophages contribute to recovery after ischemic injury but not to ischemic preconditioning in kidneys
-
Jang H.S., Kim J., Park Y.K., Park K.M. Infiltrated macrophages contribute to recovery after ischemic injury but not to ischemic preconditioning in kidneys. Transplantation 2008, 85:447-455.
-
(2008)
Transplantation
, vol.85
, pp. 447-455
-
-
Jang, H.S.1
Kim, J.2
Park, Y.K.3
Park, K.M.4
-
15
-
-
0037127298
-
Prevention of kidney ischemia/reperfusion-induced functional injury, MAPK and MAPK kinase activation, and inflammation by remote transient ureteral obstruction
-
Park K.M., Kramers C., Vayssier-Taussat M., Chen A., Bonventre J.V. Prevention of kidney ischemia/reperfusion-induced functional injury, MAPK and MAPK kinase activation, and inflammation by remote transient ureteral obstruction. J. Biol. Chem. 2002, 277:2040-2049.
-
(2002)
J. Biol. Chem.
, vol.277
, pp. 2040-2049
-
-
Park, K.M.1
Kramers, C.2
Vayssier-Taussat, M.3
Chen, A.4
Bonventre, J.V.5
-
16
-
-
66249115336
-
The exocyst protein Sec10 is necessary for primary ciliogenesis and cystogenesis in vitro
-
Zuo X., Guo W., Lipschutz J.H. The exocyst protein Sec10 is necessary for primary ciliogenesis and cystogenesis in vitro. Mol. Biol. Cell 2009, 20:2522-2529.
-
(2009)
Mol. Biol. Cell
, vol.20
, pp. 2522-2529
-
-
Zuo, X.1
Guo, W.2
Lipschutz, J.H.3
-
17
-
-
84868618448
-
Intra-renal slow cell-cycle cells contribute to the restoration of kidney tubules injured by ischemia/reperfusion
-
Kim J., Kim J.I., Na Y.K., Park K.M. Intra-renal slow cell-cycle cells contribute to the restoration of kidney tubules injured by ischemia/reperfusion. Anal. Cell Biol. 2011, 44:186-193.
-
(2011)
Anal. Cell Biol.
, vol.44
, pp. 186-193
-
-
Kim, J.1
Kim, J.I.2
Na, Y.K.3
Park, K.M.4
-
18
-
-
0036839521
-
Sodium transporter abundance profiling in kidney: effect of spironolactone
-
Nielsen J., Kwon T.H., Masilamani S., Beutler K., Hager H., Nielsen S., Knepper M.A. Sodium transporter abundance profiling in kidney: effect of spironolactone. Am. J. Physiol. Renal Physiol. 2002, 283:F923-F933.
-
(2002)
Am. J. Physiol. Renal Physiol.
, vol.283
-
-
Nielsen, J.1
Kwon, T.H.2
Masilamani, S.3
Beutler, K.4
Hager, H.5
Nielsen, S.6
Knepper, M.A.7
-
19
-
-
0041352404
-
Important role for fibronectin-EIIIA during renal tubular repair and cellular recovery in uranyl acetate-induced acute renal failure of rats
-
Fujimoto T., Fujigaki Y., Sun D.F., Togawa A., Yonemura K., Hishida A. Important role for fibronectin-EIIIA during renal tubular repair and cellular recovery in uranyl acetate-induced acute renal failure of rats. Virchows Arch. 2003, 443:194-205.
-
(2003)
Virchows Arch.
, vol.443
, pp. 194-205
-
-
Fujimoto, T.1
Fujigaki, Y.2
Sun, D.F.3
Togawa, A.4
Yonemura, K.5
Hishida, A.6
-
20
-
-
1842869910
-
Fibronectin expression in proximal tubules from ischemic rat kidneys without reperfusion
-
Petrini G., Ochoa E.J., Serra E., Torres A.M., Elias M.M. Fibronectin expression in proximal tubules from ischemic rat kidneys without reperfusion. Mol. Cell. Biochem. 2002, 241:21-27.
-
(2002)
Mol. Cell. Biochem.
, vol.241
, pp. 21-27
-
-
Petrini, G.1
Ochoa, E.J.2
Serra, E.3
Torres, A.M.4
Elias, M.M.5
-
21
-
-
80053232334
-
Plasma and cellular fibronectin: distinct and independent functions during tissue repair
-
To W.S., Midwood K.S. Plasma and cellular fibronectin: distinct and independent functions during tissue repair. Fibrogenesis Tissue Repair 2011, 4:21.
-
(2011)
Fibrogenesis Tissue Repair
, vol.4
, pp. 21
-
-
To, W.S.1
Midwood, K.S.2
-
22
-
-
0027496935
-
The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases
-
Harper J.W., Adami G.R., Wei N., Keyomarsi K., Elledge S.J. The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases. Cell 1993, 75:805-816.
-
(1993)
Cell
, vol.75
, pp. 805-816
-
-
Harper, J.W.1
Adami, G.R.2
Wei, N.3
Keyomarsi, K.4
Elledge, S.J.5
-
23
-
-
84866255883
-
Epithelial-mesenchymal crosstalk alteration in kidney fibrosis
-
Prunotto M., Budd D.C., Gabbiani G., Meier M., Formentini I., Hartmann G., Pomposiello S., Moll S. Epithelial-mesenchymal crosstalk alteration in kidney fibrosis. J. Pathol. 2012, 228:131-147.
-
(2012)
J. Pathol.
, vol.228
, pp. 131-147
-
-
Prunotto, M.1
Budd, D.C.2
Gabbiani, G.3
Meier, M.4
Formentini, I.5
Hartmann, G.6
Pomposiello, S.7
Moll, S.8
-
24
-
-
0034004155
-
Cell cycle regulatory proteins in renal disease: role in hypertrophy, proliferation, and apoptosis
-
Shankland S.J., Wolf G. Cell cycle regulatory proteins in renal disease: role in hypertrophy, proliferation, and apoptosis. Am. J. Physiol. Renal Physiol. 2000, 278:F515-F529.
-
(2000)
Am. J. Physiol. Renal Physiol.
, vol.278
-
-
Shankland, S.J.1
Wolf, G.2
-
25
-
-
77952174830
-
Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury
-
531 pp. following 143
-
Yang L., Besschetnova T.Y., Brooks C.R., Shah J.V., Bonventre J.V. Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury. Nat. Med. 2010, 16:535-543. (531 pp. following 143).
-
(2010)
Nat. Med.
, vol.16
, pp. 535-543
-
-
Yang, L.1
Besschetnova, T.Y.2
Brooks, C.R.3
Shah, J.V.4
Bonventre, J.V.5
-
26
-
-
0032875388
-
The lack of a functional p21(WAF1/CIP1) gene ameliorates progression to chronic renal failure
-
Megyesi J., Price P.M., Tamayo E., Safirstein R.L. The lack of a functional p21(WAF1/CIP1) gene ameliorates progression to chronic renal failure. Proc. Natl. Acad. Sci. U. S. A. 1999, 96:10830-10835.
-
(1999)
Proc. Natl. Acad. Sci. U. S. A.
, vol.96
, pp. 10830-10835
-
-
Megyesi, J.1
Price, P.M.2
Tamayo, E.3
Safirstein, R.L.4
-
27
-
-
0035658345
-
Na,K-ATPase activity is required for formation of tight junctions, desmosomes, and induction of polarity in epithelial cells
-
Rajasekaran S.A., Palmer L.G., Moon S.Y., Peralta Soler A., Apodaca G.L., Harper J.F., Zheng Y., Rajasekaran A.K. Na,K-ATPase activity is required for formation of tight junctions, desmosomes, and induction of polarity in epithelial cells. Mol. Biol. Cell 2001, 12:3717-3732.
-
(2001)
Mol. Biol. Cell
, vol.12
, pp. 3717-3732
-
-
Rajasekaran, S.A.1
Palmer, L.G.2
Moon, S.Y.3
Peralta Soler, A.4
Apodaca, G.L.5
Harper, J.F.6
Zheng, Y.7
Rajasekaran, A.K.8
-
28
-
-
42449110394
-
Renal cilia display length alterations following tubular injury and are present early in epithelial repair
-
Verghese E., Weidenfeld R., Bertram J.F., Ricardo S.D., Deane J.A. Renal cilia display length alterations following tubular injury and are present early in epithelial repair. Nephrol. Dial. Transplant. 2008, 23:834-841.
-
(2008)
Nephrol. Dial. Transplant.
, vol.23
, pp. 834-841
-
-
Verghese, E.1
Weidenfeld, R.2
Bertram, J.F.3
Ricardo, S.D.4
Deane, J.A.5
-
29
-
-
77649129103
-
Exocyst Sec10 protects epithelial barrier integrity and enhances recovery following oxidative stress, by activation of the MAPK pathway
-
Park K.M., Fogelgren B., Zuo X., Kim J., Chung D.C., Lipschutz J.H. Exocyst Sec10 protects epithelial barrier integrity and enhances recovery following oxidative stress, by activation of the MAPK pathway. Am. J. Physiol. Renal Physiol. 2010, 298:F818-F826.
-
(2010)
Am. J. Physiol. Renal Physiol.
, vol.298
-
-
Park, K.M.1
Fogelgren, B.2
Zuo, X.3
Kim, J.4
Chung, D.C.5
Lipschutz, J.H.6
-
30
-
-
79959336759
-
Repair of injured proximal tubule does not involve specialized progenitors
-
Humphreys B.D., Czerniak S., DiRocco D.P., Hasnain W., Cheema R., Bonventre J.V. Repair of injured proximal tubule does not involve specialized progenitors. Proc. Natl. Acad. Sci. U. S. A. 2011, 108:9226-9231.
-
(2011)
Proc. Natl. Acad. Sci. U. S. A.
, vol.108
, pp. 9226-9231
-
-
Humphreys, B.D.1
Czerniak, S.2
DiRocco, D.P.3
Hasnain, W.4
Cheema, R.5
Bonventre, J.V.6
-
31
-
-
39749172401
-
Intrinsic epithelial cells repair the kidney after injury
-
Humphreys B.D., Valerius M.T., Kobayashi A., Mugford J.W., Soeung S., Duffield J.S., McMahon A.P., Bonventre J.V. Intrinsic epithelial cells repair the kidney after injury. Cell Stem Cell 2008, 2:284-291.
-
(2008)
Cell Stem Cell
, vol.2
, pp. 284-291
-
-
Humphreys, B.D.1
Valerius, M.T.2
Kobayashi, A.3
Mugford, J.W.4
Soeung, S.5
Duffield, J.S.6
McMahon, A.P.7
Bonventre, J.V.8
-
32
-
-
0037836057
-
Dedifferentiation and proliferation of surviving epithelial cells in acute renal failure
-
Bonventre J.V. Dedifferentiation and proliferation of surviving epithelial cells in acute renal failure. J. Am. Soc. Nephrol. 2003, 14(Suppl. 1):S55-S61.
-
(2003)
J. Am. Soc. Nephrol.
, vol.14
, Issue.SUPPL. 1
-
-
Bonventre, J.V.1
-
33
-
-
0032848771
-
MAPK activation determines renal epithelial cell survival during oxidative injury
-
di Mari J.F., Davis R., Safirstein R.L. MAPK activation determines renal epithelial cell survival during oxidative injury. Am. J. Physiol. 1999, 277:F195-F203.
-
(1999)
Am. J. Physiol.
, vol.277
-
-
di Mari, J.F.1
Davis, R.2
Safirstein, R.L.3
-
34
-
-
70349649783
-
Omega-3 fatty acid supplementation attenuates oxidative stress, inflammation, and tubulointerstitial fibrosis in the remnant kidney
-
An W.S., Kim H.J., Cho K.H., Vaziri N.D. Omega-3 fatty acid supplementation attenuates oxidative stress, inflammation, and tubulointerstitial fibrosis in the remnant kidney. Am. J. Physiol. Renal Physiol. 2009, 297:F895-F903.
-
(2009)
Am. J. Physiol. Renal Physiol.
, vol.297
-
-
An, W.S.1
Kim, H.J.2
Cho, K.H.3
Vaziri, N.D.4
-
35
-
-
33847069621
-
Kinin infusion prevents renal inflammation, apoptosis, and fibrosis via inhibition of oxidative stress and mitogen-activated protein kinase activity
-
Chao J., Li H.J., Yao Y.Y., Shen B., Gao L., Bledsoe G., Chao L. Kinin infusion prevents renal inflammation, apoptosis, and fibrosis via inhibition of oxidative stress and mitogen-activated protein kinase activity. Hypertension 2007, 49:490-497.
-
(2007)
Hypertension
, vol.49
, pp. 490-497
-
-
Chao, J.1
Li, H.J.2
Yao, Y.Y.3
Shen, B.4
Gao, L.5
Bledsoe, G.6
Chao, L.7
-
36
-
-
67650564908
-
Biomarkers for early and late stage chronic allograft nephropathy by proteogenomic profiling of peripheral blood
-
Kurian S.M., Heilman R., Mondala T.S., Nakorchevsky A., Hewel J.A., Campbell D., Robison E.H., Wang L., Lin W., Gaber L., Solez K., Shidban H., Mendez R., Schaffer R.L., Fisher J.S., Flechner S.M., Head S.R., Horvath S., Yates J.R., Marsh C.L., Salomon D.R. Biomarkers for early and late stage chronic allograft nephropathy by proteogenomic profiling of peripheral blood. PLoS One 2009, 4:e6212.
-
(2009)
PLoS One
, vol.4
-
-
Kurian, S.M.1
Heilman, R.2
Mondala, T.S.3
Nakorchevsky, A.4
Hewel, J.A.5
Campbell, D.6
Robison, E.H.7
Wang, L.8
Lin, W.9
Gaber, L.10
Solez, K.11
Shidban, H.12
Mendez, R.13
Schaffer, R.L.14
Fisher, J.S.15
Flechner, S.M.16
Head, S.R.17
Horvath, S.18
Yates, J.R.19
Marsh, C.L.20
Salomon, D.R.21
more..
-
37
-
-
0034071424
-
Carbon monoxide has anti-inflammatory effects involving the mitogen-activated protein kinase pathway
-
Otterbein L.E., Bach F.H., Alam J., Soares M., Tao Lu H., Wysk M., Davis R.J., Flavell R.A., Choi A.M. Carbon monoxide has anti-inflammatory effects involving the mitogen-activated protein kinase pathway. Nat. Med. 2000, 6:422-428.
-
(2000)
Nat. Med.
, vol.6
, pp. 422-428
-
-
Otterbein, L.E.1
Bach, F.H.2
Alam, J.3
Soares, M.4
Tao Lu, H.5
Wysk, M.6
Davis, R.J.7
Flavell, R.A.8
Choi, A.M.9
-
38
-
-
84862787119
-
Deletion of the epidermal growth factor receptor in renal proximal tubule epithelial cells delays recovery from acute kidney injury
-
Chen J., Chen J.K., Harris R.C. Deletion of the epidermal growth factor receptor in renal proximal tubule epithelial cells delays recovery from acute kidney injury. Kidney Int. 2012, 82:45-52.
-
(2012)
Kidney Int.
, vol.82
, pp. 45-52
-
-
Chen, J.1
Chen, J.K.2
Harris, R.C.3
-
39
-
-
48049089409
-
(-)-Epigallocatechin gallate induces Nrf2-mediated antioxidant enzyme expression via activation of PI3K and ERK in human mammary epithelial cells
-
Na H.K., Kim E.H., Jung J.H., Lee H.H., Hyun J.W., Surh Y.J. (-)-Epigallocatechin gallate induces Nrf2-mediated antioxidant enzyme expression via activation of PI3K and ERK in human mammary epithelial cells. Arch. Biochem. Biophys. 2008, 476:171-177.
-
(2008)
Arch. Biochem. Biophys.
, vol.476
, pp. 171-177
-
-
Na, H.K.1
Kim, E.H.2
Jung, J.H.3
Lee, H.H.4
Hyun, J.W.5
Surh, Y.J.6
-
40
-
-
33751082661
-
Mitochondrial aldehyde dehydrogenase attenuates hyperoxia-induced cell death through activation of ERK/MAPK and PI3K-Akt pathways in lung epithelial cells
-
Xu D., Guthrie J.R., Mabry S., Sack T.M., Truog W.E. Mitochondrial aldehyde dehydrogenase attenuates hyperoxia-induced cell death through activation of ERK/MAPK and PI3K-Akt pathways in lung epithelial cells. Am. J. Physiol. Lung Cell. Mol. Physiol. 2006, 291:L966-L975.
-
(2006)
Am. J. Physiol. Lung Cell. Mol. Physiol.
, vol.291
-
-
Xu, D.1
Guthrie, J.R.2
Mabry, S.3
Sack, T.M.4
Truog, W.E.5
-
41
-
-
33645108170
-
ERK is an anti-inflammatory signal that suppresses expression of NF-kappaB-dependent inflammatory genes by inhibiting IKK activity in endothelial cells
-
Maeng Y.S., Min J.K., Kim J.H., Yamagishi A., Mochizuki N., Kwon J.Y., Park Y.W., Kim Y.M., Kwon Y.G. ERK is an anti-inflammatory signal that suppresses expression of NF-kappaB-dependent inflammatory genes by inhibiting IKK activity in endothelial cells. Cell. Signal. 2006, 18:994-1005.
-
(2006)
Cell. Signal.
, vol.18
, pp. 994-1005
-
-
Maeng, Y.S.1
Min, J.K.2
Kim, J.H.3
Yamagishi, A.4
Mochizuki, N.5
Kwon, J.Y.6
Park, Y.W.7
Kim, Y.M.8
Kwon, Y.G.9
-
42
-
-
12344324705
-
MEK inhibitor, U0126, attenuates cisplatin-induced renal injury by decreasing inflammation and apoptosis
-
Jo S.K., Cho W.Y., Sung S.A., Kim H.K., Won N.H. MEK inhibitor, U0126, attenuates cisplatin-induced renal injury by decreasing inflammation and apoptosis. Kidney Int. 2005, 67:458-466.
-
(2005)
Kidney Int.
, vol.67
, pp. 458-466
-
-
Jo, S.K.1
Cho, W.Y.2
Sung, S.A.3
Kim, H.K.4
Won, N.H.5
-
43
-
-
0035839428
-
Ion pumps in polarized cells: sorting and regulation of the Na+, K+- and H+, K+-ATPases
-
Dunbar L.A., Caplan M.J. Ion pumps in polarized cells: sorting and regulation of the Na+, K+- and H+, K+-ATPases. J. Biol. Chem. 2001, 276:29617-29620.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 29617-29620
-
-
Dunbar, L.A.1
Caplan, M.J.2
-
44
-
-
77953382455
-
Na,K-ATPase subunits as markers for epithelial-mesenchymal transition in cancer and fibrosis
-
Rajasekaran S.A., Huynh T.P., Wolle D.G., Espineda C.E., Inge L.J., Skay A., Lassman C., Nicholas S.B., Harper J.F., Reeves A.E., Ahmed M.M., Leatherman J.M., Mullin J.M., Rajasekaran A.K. Na,K-ATPase subunits as markers for epithelial-mesenchymal transition in cancer and fibrosis. Mol. Cancer Ther. 2010, 9:1515-1524.
-
(2010)
Mol. Cancer Ther.
, vol.9
, pp. 1515-1524
-
-
Rajasekaran, S.A.1
Huynh, T.P.2
Wolle, D.G.3
Espineda, C.E.4
Inge, L.J.5
Skay, A.6
Lassman, C.7
Nicholas, S.B.8
Harper, J.F.9
Reeves, A.E.10
Ahmed, M.M.11
Leatherman, J.M.12
Mullin, J.M.13
Rajasekaran, A.K.14
-
45
-
-
70349918229
-
Renal primary cilia lengthen after acute tubular necrosis
-
Verghese E., Ricardo S.D., Weidenfeld R., Zhuang J., Hill P.A., Langham R.G., Deane J.A. Renal primary cilia lengthen after acute tubular necrosis. J. Am. Soc. Nephrol. 2009, 20:2147-2153.
-
(2009)
J. Am. Soc. Nephrol.
, vol.20
, pp. 2147-2153
-
-
Verghese, E.1
Ricardo, S.D.2
Weidenfeld, R.3
Zhuang, J.4
Hill, P.A.5
Langham, R.G.6
Deane, J.A.7
-
46
-
-
33845982872
-
Intracellular signaling via ERK/MAPK completes the pathway for tubulogenic fibronectin in MDCK cells
-
Liu Z., Greco A.J., Hellman N.E., Spector J., Robinson J., Tang O.T., Lipschutz J.H. Intracellular signaling via ERK/MAPK completes the pathway for tubulogenic fibronectin in MDCK cells. Biochem. Biophys. Res. Commun. 2007, 353:793-798.
-
(2007)
Biochem. Biophys. Res. Commun.
, vol.353
, pp. 793-798
-
-
Liu, Z.1
Greco, A.J.2
Hellman, N.E.3
Spector, J.4
Robinson, J.5
Tang, O.T.6
Lipschutz, J.H.7
-
47
-
-
24944576036
-
Activated extracellular signal-regulated kinases are necessary and sufficient to initiate tubulogenesis in renal tubular MDCK strain I cell cysts
-
Hellman N.E., Greco A.J., Rogers K.K., Kanchagar C., Balkovetz D.F., Lipschutz J.H. Activated extracellular signal-regulated kinases are necessary and sufficient to initiate tubulogenesis in renal tubular MDCK strain I cell cysts. Am. J. Physiol. Renal Physiol. 2005, 289:F777-F785.
-
(2005)
Am. J. Physiol. Renal Physiol.
, vol.289
-
-
Hellman, N.E.1
Greco, A.J.2
Rogers, K.K.3
Kanchagar, C.4
Balkovetz, D.F.5
Lipschutz, J.H.6
-
48
-
-
79959358443
-
The small GTPase Cdc42 is necessary for primary ciliogenesis in renal tubular epithelial cells
-
Zuo X., Fogelgren B., Lipschutz J.H. The small GTPase Cdc42 is necessary for primary ciliogenesis in renal tubular epithelial cells. J. Biol. Chem. 2011, 286:22469-22477.
-
(2011)
J. Biol. Chem.
, vol.286
, pp. 22469-22477
-
-
Zuo, X.1
Fogelgren, B.2
Lipschutz, J.H.3
-
49
-
-
0033638380
-
Exocyst is involved in cystogenesis and tubulogenesis and acts by modulating synthesis and delivery of basolateral plasma membrane and secretory proteins
-
Lipschutz J.H., Guo W., O'Brien L.E., Nguyen Y.H., Novick P., Mostov K.E. Exocyst is involved in cystogenesis and tubulogenesis and acts by modulating synthesis and delivery of basolateral plasma membrane and secretory proteins. Mol. Biol. Cell 2000, 11:4259-4275.
-
(2000)
Mol. Biol. Cell
, vol.11
, pp. 4259-4275
-
-
Lipschutz, J.H.1
Guo, W.2
O'Brien, L.E.3
Nguyen, Y.H.4
Novick, P.5
Mostov, K.E.6
-
50
-
-
78149298226
-
Mechanisms of tubulointerstitial fibrosis
-
Zeisberg M., Neilson E.G. Mechanisms of tubulointerstitial fibrosis. J. Am. Soc. Nephrol. 2010, 21:1819-1834.
-
(2010)
J. Am. Soc. Nephrol.
, vol.21
, pp. 1819-1834
-
-
Zeisberg, M.1
Neilson, E.G.2
-
51
-
-
80052404286
-
ERK pathway mediates P2X7 expression and cell death in renal interstitial fibroblasts exposed to necrotic renal epithelial cells
-
Ponnusamy M., Liu N., Gong R., Yan H., Zhuang S. ERK pathway mediates P2X7 expression and cell death in renal interstitial fibroblasts exposed to necrotic renal epithelial cells. Am. J. Physiol. Renal Physiol. 2011, 301:F650-F659.
-
(2011)
Am. J. Physiol. Renal Physiol.
, vol.301
-
-
Ponnusamy, M.1
Liu, N.2
Gong, R.3
Yan, H.4
Zhuang, S.5
-
53
-
-
84879080578
-
Molecular targets for treatment of kidney fibrosis
-
Chuang P.Y., Menon M.C., He J.C. Molecular targets for treatment of kidney fibrosis. J. Mol. Med. (Berl) 2013, 91:549-559.
-
(2013)
J. Mol. Med. (Berl)
, vol.91
, pp. 549-559
-
-
Chuang, P.Y.1
Menon, M.C.2
He, J.C.3
-
54
-
-
34147176657
-
Epithelial-to-mesenchymal transition of human proximal tubular epithelial cells: effects of rapamycin, mycophenolate, cyclosporin, azathioprine, and methylprednisolone
-
Copeland J.W., Beaumont B.W., Merrilees M.J., Pilmore H.L. Epithelial-to-mesenchymal transition of human proximal tubular epithelial cells: effects of rapamycin, mycophenolate, cyclosporin, azathioprine, and methylprednisolone. Transplantation 2007, 83:809-814.
-
(2007)
Transplantation
, vol.83
, pp. 809-814
-
-
Copeland, J.W.1
Beaumont, B.W.2
Merrilees, M.J.3
Pilmore, H.L.4
-
55
-
-
77955983429
-
Mycophenolic acid may delay allograft fibrosis by inhibiting transforming growth factor-beta1-induced activation of Nox-2 through the nuclear factor-kappaB pathway
-
Djamali A., Vidyasagar A., Yagci G., Huang L.J., Reese S. Mycophenolic acid may delay allograft fibrosis by inhibiting transforming growth factor-beta1-induced activation of Nox-2 through the nuclear factor-kappaB pathway. Transplantation 2010, 90:387-393.
-
(2010)
Transplantation
, vol.90
, pp. 387-393
-
-
Djamali, A.1
Vidyasagar, A.2
Yagci, G.3
Huang, L.J.4
Reese, S.5
-
56
-
-
84875615221
-
Bone marrow-derived cells play a major role in kidney fibrosis via proliferation and differentiation in the infiltrated site
-
Jang H.S., Kim J.I., Jung K.J., Kim J., Han K.H., Park K.M. Bone marrow-derived cells play a major role in kidney fibrosis via proliferation and differentiation in the infiltrated site. Biochim. Biophys. Acta 2013, 1832:817-825.
-
(2013)
Biochim. Biophys. Acta
, vol.1832
, pp. 817-825
-
-
Jang, H.S.1
Kim, J.I.2
Jung, K.J.3
Kim, J.4
Han, K.H.5
Park, K.M.6
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