Inhibition of myocardial reperfusion injury by ischemic postconditioning requires sirtuin 3-mediated deacetylation of cyclophilin D

Journal of Molecular and Cellular Cardiology

Volumn 84, Issue , 2015, Pages 61-69

  Bochaton, T ^a,b   Crola Da Silva, C ^a   Pillot, B ^a   Villedieu, C ^a   Ferreras, L ^a   Alam, M R ^a   Thibault, H ^a,b   Strina, M ^a   Gharib, A ^a   Ovize, M ^a,b   Baetz, D ^a  

^a UNIVERSITÉ LYON 1   (France)

^b LOUIS PRADEL HOSPITAL   (France)

Author keywords

Cyclophilin D; Ischemia reperfusion; Post conditioning; Sirtuin 3; Survival

Indexed keywords

ALPHA TUBULIN; CYCLOPHILIN D; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; SIRTUIN 3; CARRIER PROTEIN; CYCLOPHILIN; OXYGEN;

ACETYLATION; ANALYSIS OF VARIANCE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CATALYSIS; CELL DEATH; CONTROLLED STUDY; DEACETYLATION; DISEASE SEVERITY; ENZYME ACTIVATION; ENZYME ACTIVITY; HEART INFARCTION SIZE; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HYPOXIA; IMMUNOPRECIPITATION; IN VITRO STUDY; IN VIVO STUDY; ISCHEMIC POSTCONDITIONING; MALE; MOUSE; NITROSYLATION; NONHUMAN; PLASMID; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; RAT; REOXYGENATION; REPERFUSION INJURY; SITE DIRECTED MUTAGENESIS; ANIMAL; CELL HYPOXIA; DRUG EFFECTS; KNOCKOUT MOUSE; METABOLISM; MITOCHONDRIAL MEMBRANE POTENTIAL;

MUS;

ACETYLATION; ANIMALS; CELL DEATH; CELL HYPOXIA; CYCLOPHILINS; ISCHEMIC POSTCONDITIONING; MALE; MEMBRANE POTENTIAL, MITOCHONDRIAL; MICE, KNOCKOUT; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MYOCARDIAL REPERFUSION INJURY; OXYGEN; RATS; SIRTUIN 3;

EID: 84928121207     PISSN: 00222828     EISSN: 10958584     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2015.03.017     Document Type: Article

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