Mouse models of hereditary hemorrhagic telangiectasia: Recent advances and future challenges

Frontiers in Genetics

Volumn 5, Issue FEB, 2015, Pages

  Tual Chalot, Simon ^a   Oh, S Paul ^b   Arthur, Helen M ^a  

^a NEWCASTLE UNIVERSITY   (United Kingdom)

^b UNIVERSITY OF FLORIDA   (United States)

Author keywords

Angiogenesis; Arteriovenous malformation; Bmp Smad signaling; TGF signaling; Vascular development; Vascular disease

Indexed keywords

BONE MORPHOGENETIC PROTEIN; ENDOGLIN; SMAD1 PROTEIN; SMAD4 PROTEIN; SMAD5 PROTEIN; SMAD8 PROTEIN; TRANSFORMING GROWTH FACTOR BETA; TUMOR NECROSIS FACTOR ALPHA;

ACTIVIN RECEPTOR LIKE KINASE 1 GENE; ANGIOGENESIS; ARTERIOVENOUS MALFORMATION; CLINICAL TRIAL (TOPIC); CONGENITAL BLOOD VESSEL MALFORMATION; ENDOGLIN GENE; ENDOTHELIUM CELL; GENE MUTATION; GENETICS; HEART DEVELOPMENT; HETEROZYGOSITY LOSS; HUMAN; IMAGING AND DISPLAY; INFLAMMATION; INTRACELLULAR SIGNALING; MOUSE MODEL; NONHUMAN; PHENOTYPE; RENDU OSLER WEBER DISEASE; REVIEW; VASCULARIZATION;

ANIMALIA; MUS;

EID: 84923530182     PISSN: None     EISSN: 16648021     Source Type: Journal    
DOI: 10.3389/fgene.2015.00025     Document Type: Review

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