De novo cerebrovascular malformation in the adult mouse after endothelial alkl deletion and angiogenic stimulation

Stroke

Volumn 45, Issue 3, 2014, Pages 900-902

  Chen, Wanqiu ^a   Sun, Zhengda ^b   Han, Zhenying ^a   Jun, Kristine ^a   Camus, Marine ^a   Wankhede, Mamta ^a   Mao, Lei ^a   Arnold, Tom ^a   Young, William L ^a,b   Su, Hua ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Animal; AVM (arteriovenous malformation) intracranial; Hereditary hemorrhagic; Models; Telangiectasia; Type 2

Indexed keywords

ACTIVIN RECEPTOR LIKE KINASE 1; ADENOVIRUS VECTOR; TAMOXIFEN; VASCULOTROPIN; ACTIVIN RECEPTOR 1; ACVRL1 PROTEIN, MOUSE; ANGIOGENIC FACTOR; ANTIMETABOLITE; ANTINEOPLASTIC HORMONE AGONISTS AND ANTAGONISTS; BROXURIDINE; VASCULOTROPIN A;

ADULT; ANGIOGENESIS; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTERIOVENOUS MALFORMATION; ARTICLE; BRAIN REGION; CELL PROLIFERATION; CEREBROVASCULAR MALFORMATION; CONTROLLED STUDY; DYSPLASIA; ENDOTHELIUM CELL; GENE DELETION; MOUSE; NONHUMAN; PRIORITY JOURNAL; ADENOVIRUS; ANIMAL; AVM (ARTERIOVENOUS MALFORMATION) INTRACRANIAL; CENTRAL NERVOUS SYSTEM MALFORMATION; EXON; GENETICS; METABOLISM; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; RENDU OSLER WEBER DISEASE; TELANGIECTASIA, HEREDITARY HEMORRHAGIC, TYPE 2; TRANSGENIC ORGANISM;

AVM (ARTERIOVENOUS MALFORMATION) INTRACRANIAL; MODELS, ANIMAL; TELANGIECTASIA, HEREDITARY HEMORRHAGIC, TYPE 2;

ACTIVIN RECEPTORS, TYPE I; ADENOVIRIDAE; ANGIOGENESIS INDUCING AGENTS; ANIMALS; ANTIMETABOLITES; ANTINEOPLASTIC AGENTS, HORMONAL; BROMODEOXYURIDINE; CELL PROLIFERATION; CENTRAL NERVOUS SYSTEM VASCULAR MALFORMATIONS; ENDOTHELIAL CELLS; EXONS; GENE DELETION; MICE; ORGANISMS, GENETICALLY MODIFIED; TAMOXIFEN; TELANGIECTASIA, HEREDITARY HEMORRHAGIC; VASCULAR ENDOTHELIAL GROWTH FACTOR A;

EID: 84899155539     PISSN: 00392499     EISSN: 15244628     Source Type: Journal    
DOI: 10.1161/STROKEAHA.113.003655     Document Type: Article

References (8)

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