-
2
-
-
70349705441
-
Prions: Protein aggregation and infectious diseases
-
Aguzzi A, Calella AM. Prions: protein aggregation and infectious diseases. Physiol Rev 2009;89:1105-1152
-
(2009)
Physiol Rev
, vol.89
, pp. 1105-1152
-
-
Aguzzi, A.1
Calella, A.M.2
-
4
-
-
33750224729
-
Prion protein in cardiac muscle of elk (cervus elaphus nelsoni) and white-Tailed deer (odocoileus virginianus) infected with chronic wasting disease
-
Jewell JE, Brown J, Kreeger T, Williams ES. Prion protein in cardiac muscle of elk (Cervus elaphus nelsoni) and white-Tailed deer (Odocoileus virginianus) infected with chronic wasting disease. J Gen Virol 2006;87:3443-3450
-
(2006)
J Gen Virol
, vol.87
, pp. 3443-3450
-
-
Jewell, J.E.1
Brown, J.2
Kreeger, T.3
Williams, E.S.4
-
5
-
-
0035033712
-
Primary myopathy and accumulation of PrPSc-like like molecules in peripheral tissues of transgenic mice expressing a prion protein insertional mutation
-
Chiesa R, Pestronk A, Schmidt RE, Tourtellotte WG, Ghetti B, Piccardo P, Harris DA. Primary myopathy and accumulation of PrPSc-like like molecules in peripheral tissues of transgenic mice expressing a prion protein insertional mutation. Neurobiol Dis 2001; 8:279-288
-
(2001)
Neurobiol Dis
, vol.8
, pp. 279-288
-
-
Chiesa, R.1
Pestronk, A.2
Schmidt, R.E.3
Tourtellotte, W.G.4
Ghetti, B.5
Piccardo, P.6
Harris, D.A.7
-
6
-
-
0026600865
-
Normal development and behaviour of mice lacking the neuronal cell-surface PrP protein
-
Budddeler H, Fischer M, Lang Y, Bluethmann H, Lipp HP, DeArmond SJ, Prusiner SB, Aguet M, Weissmann C. Normal development and behaviour of mice lacking the neuronal cell-surface PrP protein. Nature 1992;356:577-582
-
(1992)
Nature
, vol.356
, pp. 577-582
-
-
Budddeler, H.1
Fischer, M.2
Lang, Y.3
Bluethmann, H.4
Lipp, H.P.5
Dearmond, S.J.6
Prusiner, S.B.7
Aguet, M.8
Weissmann, C.9
-
7
-
-
0028420937
-
129/Ola mice carrying a null mutation in PrP that abolishes mRNA production are developmentally normal
-
Manson JC, Clarke AR, Hooper ML, Aitchison L, McConnell I, Hope J. 129/Ola mice carrying a null mutation in PrP that abolishes mRNA production are developmentally normal. Mol Neurobiol 1994;8:121-127
-
(1994)
Mol Neurobiol
, vol.8
, pp. 121-127
-
-
Manson, J.C.1
Clarke, A.R.2
Hooper, M.L.3
Aitchison, L.4
McConnell, I.5
Hope, J.6
-
8
-
-
0036470471
-
Post-natal knockout of prion protein alters hippocampal CA1 properties, but does not result in neurodegeneration
-
Mallucci GR, Ratteaaa S, Asante EA, Linehan J, Gowland I, Jefferys JG, Collinge J. Post-natal knockout of prion protein alters hippocampal CA1 properties, but does not result in neurodegeneration. EMBO J 2002;21:202-210
-
(2002)
EMBO J
, vol.21
, pp. 202-210
-
-
Mallucci, G.R.1
Ratteaaa, S.2
Asante, E.A.3
Linehan, J.4
Gowland, I.5
Jefferys, J.G.6
Collinge, J.7
-
9
-
-
43149121009
-
The prion protein knockout mouse: A phenotype under challenge
-
Steele AD, Lindquist S, Aguzzi A. The prion protein knockout mouse: a phenotype under challenge. Prion 2007;1:83-93
-
(2007)
Prion
, vol.1
, pp. 83-93
-
-
Steele, A.D.1
Lindquist, S.2
Aguzzi, A.3
-
11
-
-
42049099020
-
Physiology of the prion protein
-
Linden R, Martins VR, Prado MA, Cammarota M, Izquierdo I, Brentani RR. Physiology of the prion protein. Physiol Rev 2008;88:673-728
-
(2008)
Physiol Rev
, vol.88
, pp. 673-728
-
-
Linden, R.1
Martins, V.R.2
Prado, M.A.3
Cammarota, M.4
Izquierdo, I.5
Brentani, R.R.6
-
12
-
-
0036124813
-
Oxidative stress and the prion protein in transmissible spongiform encephalopathies
-
Milhavet O, Lehmann S. Oxidative stress and the prion protein in transmissible spongiform encephalopathies. Brain Res Brain Res Rev 2002;38:328-339
-
(2002)
Brain Res Brain Res Rev
, vol.38
, pp. 328-339
-
-
Milhavet, O.1
Lehmann, S.2
-
14
-
-
3042594982
-
Prion protein accumulation and neuroprotection in hypoxic brain Damage
-
McLennan NF, Brennan PM, McNeill A, Davies I, Fotheringham A, Rennison KA, Ritchie D, Brannan F, Head MW, Ironside JW, Williams A, Bell JE. Prion protein accumulation and neuroprotection in hypoxic brain Damage. Am J Pathol 2004;165:227-235
-
(2004)
Am J Pathol
, vol.165
, pp. 227-235
-
-
McLennan, N.F.1
Brennan, P.M.2
McNeill, A.3
Davies, I.4
Fotheringham, A.5
Rennison, K.A.6
Ritchie, D.7
Brannan, F.8
Head, M.W.9
Ironside, J.W.10
Williams, A.11
Bell, J.E.12
-
15
-
-
8644289373
-
Upregulation of cellular prion protein (PrPC) after focal cerebral ischemia and influence of lesion severity
-
Weise J, Crome O, Sandau R, Schulz-Schaeffer W, Badddhr M, Zerr I. Upregulation of cellular prion protein (PrPC) after focal cerebral ischemia and influence of lesion severity. Neurosci Lett 2004;372:146-150
-
(2004)
Neurosci Lett
, vol.372
, pp. 146-150
-
-
Weise, J.1
Crome, O.2
Sandau, R.3
Schulz-Schaeffer, W.4
Badddhr, M.5
Zerr, I.6
-
16
-
-
33847289684
-
Cellular prion protein is increased in the plasma and periinfarcted brain tissue after acute stroke
-
Mitsios N, Saka M, Krupinski J, Pennucci R, Sanfeliu C, Miguel Turu M, Gaffney J, Kumar P, Kumar S, Sullivan M, Slevin M. Cellular prion protein is increased in the plasma and periinfarcted brain tissue after acute stroke. J Neurosci Res 2007;85:602-611
-
(2007)
J Neurosci Res
, vol.85
, pp. 602-611
-
-
Mitsios, N.1
Saka, M.2
Krupinski, J.3
Pennucci, R.4
Sanfeliu, C.5
Miguel Turu, M.6
Gaffney, J.7
Kumar, P.8
Kumar, S.9
Sullivan, M.10
Slevin, M.11
-
17
-
-
25644433562
-
Overexpression of PrPC by adenovirus-mediated gene targeting reduces ischemic injury in a stroke rat model
-
Shyu WC, Lin SZ, Chiang MF, Ding DC, Li KW, Chen SF, Yang HI, Li H. Overexpression of PrPC by adenovirus-mediated gene targeting reduces ischemic injury in a stroke rat model. J Neurosci 2005;25:8967-8977
-
(2005)
J Neurosci
, vol.25
, pp. 8967-8977
-
-
Shyu, W.C.1
Lin, S.Z.2
Chiang, M.F.3
Ding, D.C.4
Li, K.W.5
Chen, S.F.6
Yang, H.I.7
Li, H.8
-
18
-
-
43049116184
-
Overexpression of cellular prion protein alters postischemic Erk1/2 phosphorylation but not Akt phosphorylation and protects against focal cerebral ischemia
-
Weise J, Doeppner TR, Mudddller T, Wrede A, Schulz-Schaeffer W, Zerr I, Witte OW, BadddhrM. Overexpression of cellular prion protein alters postischemic Erk1/2 phosphorylation but not Akt phosphorylation and protects against focal cerebral ischemia. Restor Neurol Neurosci 2008;26:57-64
-
(2008)
Restor Neurol Neurosci
, vol.26
, pp. 57-64
-
-
Weise, J.1
Doeppner, T.R.2
Mudddller, T.3
Wrede, A.4
Schulz-Schaeffer, W.5
Zerr, I.6
Witte, O.W.7
Badddhr, M.8
-
19
-
-
26944462341
-
Aggravation of ischemic brain injury by prion protein Deficiency: Role of ERK-1/-2 and STAT-1
-
Spudich A, Frigg R, Kilic E, Kilic U, Oesch B, Raeber A, Bassetti CL, Hermann DM. Aggravation of ischemic brain injury by prion protein Deficiency: role of ERK-1/-2 and STAT-1. Neurobiol Dis 2005;20:442-449
-
(2005)
Neurobiol Dis
, vol.20
, pp. 442-449
-
-
Spudich, A.1
Frigg, R.2
Kilic, E.3
Kilic, U.4
Oesch, B.5
Raeber, A.6
Bassetti, C.L.7
Hermann, D.M.8
-
20
-
-
33646695909
-
Deletion of cellular prion protein results in reduced Akt activation, enhanced postischemic caspase-3 activation, and exacerbation of ischemic brain injury
-
Weise J, Sandau R, Schwarting S, Crome O, Wrede A, Schulz-Schaeffer W, Zerr I, BadddhrM. Deletion of cellular prion protein results in reduced Akt activation, enhanced postischemic caspase-3 activation, and exacerbation of ischemic brain injury. Stroke 2006;37: 1296-1300
-
(2006)
Stroke
, vol.37
, pp. 1296-1300
-
-
Weise, J.1
Sandau, R.2
Schwarting, S.3
Crome, O.4
Wrede, A.5
Schulz-Schaeffer, W.6
Zerr, I.7
Badddhr, M.8
-
21
-
-
0035873869
-
Imbalance of antioxidant defense in mice lacking cellular prion protein
-
Klamt F, Dal-Pizzol F, Conte da Frota ML Jr, Walz R, Andrades ME, da Silva EG, Brentani RR, Izquierdo I, Fonseca Moreira JC. Imbalance of antioxidant defense in mice lacking cellular prion protein. Free Radic Biol Med 2001;30:1137-1144
-
(2001)
Free Radic Biol Med
, vol.30
, pp. 1137-1144
-
-
Klamt, F.1
Dal-Pizzol, F.2
Conte Da Frota, M.L.3
Walz, R.4
Andrades, M.E.5
Da Silva, E.G.6
Brentani, R.R.7
Izquierdo, I.8
Fonseca Moreira, J.C.9
-
22
-
-
79952438784
-
Gene therapy with hypoxia-inducible factor 1 alpha in skeletal muscle is cardioprotective in vivo
-
Czibik G, Gravning J, Martinov V, Ishaq B, Knudsen E, Attramadal H, Valen G. Gene therapy with hypoxia-inducible factor 1 alpha in skeletal muscle is cardioprotective in vivo. Life Sci 2011;88:543-550
-
(2011)
Life Sci
, vol.88
, pp. 543-550
-
-
Czibik, G.1
Gravning, J.2
Martinov, V.3
Ishaq, B.4
Knudsen, E.5
Attramadal, H.6
Valen, G.7
-
23
-
-
34447581554
-
Untersuchungen am udddberlebenden Sadddugethierherzen
-
Langendorff O. Untersuchungen am udddberlebenden Sadddugethierherzen. Pfludddegers Archiv 1895;61:291-322
-
(1895)
Pfludddegers Archiv
, vol.61
, pp. 291-322
-
-
Langendorff, O.1
-
24
-
-
67649243697
-
The cardioprotective effects elicited by p66(Shc) ablation Demonstrate the crucial role of mitochondrial ROSformation in ischemia/reperfusion injury
-
Carpi A, Menabò R, Kaludercic N, Pelicci P, Di Lisa F, Giorgio M. The cardioprotective effects elicited by p66(Shc) ablation Demonstrate the crucial role of mitochondrial ROSformation in ischemia/reperfusion injury. Biochim Biophys Acta 2009;1787:774-780
-
(2009)
Biochim Biophys Acta
, vol.1787
, pp. 774-780
-
-
Carpi, A.1
Menabò, R.2
Kaludercic, N.3
Pelicci, P.4
Di Lisa, F.5
Giorgio, M.6
-
25
-
-
0014691273
-
The utility of superoxide dismutase in studying free radical reactions i Radicals generated by the interaction of sulfite, dimethyl sulfoxide, and oxygen
-
McCord JM, Fridovich I. The utility of superoxide dismutase in studying free radical reactions. I. Radicals generated by the interaction of sulfite, dimethyl sulfoxide, and oxygen. J Biol Chem 1969;244:6056-6063
-
(1969)
J Biol Chem
, vol.244
, pp. 6056-6063
-
-
McCord, J.M.1
Fridovich, I.2
-
26
-
-
0021318441
-
Catalase in vitro
-
Aebi H. Catalase in vitro. Methods Enzymol 1984;105:121-126
-
(1984)
Methods Enzymol
, vol.105
, pp. 121-126
-
-
Aebi, H.1
-
27
-
-
33745234267
-
Nadph oxidases are in part responsible for increased cardiovascular superoxide production during aging
-
Oudot A, Martin C, Busseuil D, Vergely C, Demaison L, Rochette L. NADPH oxidases are in part responsible for increased cardiovascular superoxide production During aging. Free Radic Biol Med 2006;40:2214-2222
-
(2006)
Free Radic Biol Med
, vol.40
, pp. 2214-2222
-
-
Oudot, A.1
Martin, C.2
Busseuil, D.3
Vergely, C.4
Demaison, L.5
Rochette, L.6
-
28
-
-
14644442283
-
Oxygen, oxidative stress, hypoxia, and heart failure
-
Giordano FJ. Oxygen, oxidative stress, hypoxia, and heart failure. J Clin Invest 2005;115: 500-508
-
(2005)
J Clin Invest
, vol.115
, pp. 500-508
-
-
Giordano, F.J.1
-
29
-
-
79551675865
-
Mitochondrial injury and protection in ischemic pre- and postconditioning
-
Di Lisa F, Canton M, Carpi A, Kaludercic N, Menabò R, Menazza S, Semenzato M. Mitochondrial injury and protection in ischemic pre- And postconditioning. Antioxid Redox Signal 2011;14:881-891
-
(2011)
Antioxid Redox Signal
, vol.14
, pp. 881-891
-
-
Di Lisa, F.1
Canton, M.2
Carpi, A.3
Kaludercic, N.4
Menabò, R.5
Menazza, S.6
Semenzato, M.7
-
30
-
-
77954080841
-
The contribution of reactive oxygen species and p38 mitogen-Activated protein kinase to myofilament oxidation and progression of heart failure in rabbits
-
Heusch P, Canton M, Aker S, van De Sand A, Konietzka I, Rassaf T, Menazza S, Brodde OE, Di Lisa F, Heusch G, Schulz R. The contribution of reactive oxygen species and p38 mitogen-Activated protein kinase to myofilament oxidation and progression of heart failure in rabbits. Br J Pharmacol 2010;160:1408-1416
-
(2010)
Br J Pharmacol
, vol.160
, pp. 1408-1416
-
-
Heusch, P.1
Canton, M.2
Aker, S.3
Van De Sand, A.4
Konietzka, I.5
Rassaf, T.6
Menazza, S.7
Brodde, O.E.8
Di Lisa, F.9
Heusch, G.10
Schulz, R.11
-
31
-
-
33645023189
-
Oxidative modification of tropomyosin and myocardial dysfunction following coronary microembolization
-
Canton M, Skyschally A, Menabò R, Boengler K, Gres P, Schulz R, Haude M, Erbel R, Di Lisa F, Heusch G. Oxidative modification of tropomyosin and myocardial dysfunction following coronary microembolization. Eur Heart J 2006;27:875-881
-
(2006)
Eur Heart J
, vol.27
, pp. 875-881
-
-
Canton, M.1
Skyschally, A.2
Menabò, R.3
Boengler, K.4
Gres, P.5
Schulz, R.6
Haude, M.7
Erbel, R.8
Di Lisa, F.9
Heusch, G.10
-
33
-
-
0034665065
-
Opening of mitochondrial k(atp) channels triggers the preconditioned state by generating free radicals
-
Pain T, Yan XM, Critz SD, Yue Y, Nakano A, Liu GS, Heusch G, Cohen MV, Downey JM. Opening of mitochondrial K(ATP) channels triggers the preconditioned state by generating free radicals. Circ Res 2000;87:460-466
-
(2000)
Circ Res
, vol.87
, pp. 460-466
-
-
Pain, T.1
Yan, X.M.2
Critz, S.D.3
Yue, Y.4
Nakano, A.5
Liu, G.S.6
Heusch, G.7
Cohen, M.V.8
Downey, J.M.9
-
34
-
-
0001534793
-
-
Cohen GM, ed 1st ed. Boca Raton, Florida, USA: CRC Press Inc
-
Ishikawa T, Akerboom TPM, Sies H. In: Cohen GM, ed. Target Organ Toxicity. 1st ed. Boca Raton, Florida, USA: CRC Press Inc.; 1986. p. 129-143
-
(1986)
Target Organ Toxicity
, pp. 129-143
-
-
Ishikawa, T.1
Akerboom, T.P.M.2
Sies, H.3
-
35
-
-
0034764599
-
Oxidative impairment in scrapie-infected mice is associated with brain metals perturbations and altered antioxidant activities
-
Wong BS, Brown DR, Pan T, Whiteman M, Liu T, Bu X, Li R, Gambetti P, Olesik J, Rubenstein R, Sy MS. Oxidative impairment in scrapie-infected mice is associated with brain metals perturbations and altered antioxidant activities. J Neurochem 2001;79: 689-698
-
(2001)
J Neurochem
, vol.79
, pp. 689-698
-
-
Wong, B.S.1
Brown, D.R.2
Pan, T.3
Whiteman, M.4
Liu, T.5
Bu, X.6
Li, R.7
Gambetti, P.8
Olesik, J.9
Rubenstein, R.10
Sy, M.S.11
-
36
-
-
0141717118
-
No superoxide dismutase activity of cellular prion protein in vivo
-
Hutter G, Heppner FL, Aguzzi A. No superoxide dismutase activity of cellular prion protein in vivo. Biol Chem 2003;384:1279-1285
-
(2003)
Biol Chem
, vol.384
, pp. 1279-1285
-
-
Hutter, G.1
Heppner, F.L.2
Aguzzi, A.3
-
37
-
-
0034678023
-
Brain copper content and cuproenzyme activity do not vary with prion protein expression level
-
Waggoner DJ, Drisaldi B, Bartnikas TB, Casareno RL, Prohaska JR, Gitlin JD, Harris DA. Brain copper content and cuproenzyme activity do not vary with prion protein expression level. J Biol Chem 2000;275:7455-7458
-
(2000)
J Biol Chem
, vol.275
, pp. 7455-7458
-
-
Waggoner, D.J.1
Drisaldi, B.2
Bartnikas, T.B.3
Casareno, R.L.4
Prohaska, J.R.5
Gitlin, J.D.6
Harris, D.A.7
-
38
-
-
42249097446
-
Final common molecular pathways of aging and cardiovascular disease: Role of the p66Shc protein
-
Cosentino F, Francia P, Camici GG, Pelicci PG, Ludddscher TF, Volpe M. Final common molecular pathways of aging and cardiovascular disease: role of the p66Shc protein. Arterioscler Thromb Vasc Biol 2008;28:622-628
-
(2008)
Arterioscler Thromb Vasc Biol
, vol.28
, pp. 622-628
-
-
Cosentino, F.1
Francia, P.2
Camici, G.G.3
Pelicci, P.G.4
Ludddscher, T.F.5
Volpe, M.6
-
39
-
-
0033581704
-
The p66Shc adaptor protein controls oxidative stress response and life span in mammals
-
Migliaccio E, Giorgio M, Mele S, Pelicci G, Reboldi P, Pandolfi PP, Lanfrancone L, Pelicci PG. The p66Shc adaptor protein controls oxidative stress response and life span in mammals. Nature 1999;402:309-313
-
(1999)
Nature
, vol.402
, pp. 309-313
-
-
Migliaccio, E.1
Giorgio, M.2
Mele, S.3
Pelicci, G.4
Reboldi, P.5
Pandolfi, P.P.6
Lanfrancone, L.7
Pelicci, P.G.8
-
40
-
-
0032510660
-
Medical and cellular implications of stunning, hibernation, and preconditioning: An nhlbi workshop
-
Kloner RA, Bolli R, Marban E, Reinlib L, Braunwald E. Medical and cellular implications of stunning, hibernation, and preconditioning: an NHLBI workshop. Circulation 1998;97: 1848-1867
-
(1998)
Circulation
, vol.97
, pp. 1848-1867
-
-
Kloner, R.A.1
Bolli, R.2
Marban, E.3
Reinlib, L.4
Braunwald, E.5
-
41
-
-
22744447211
-
Electron transfer between cytochrome c and p66Shc generates reactive oxygen species that trigger mitochondrial apoptosis
-
Giorgio M, Migliaccio E, Orsini F, Paolucci D, Moroni M, Contursi C, Pelliccia G, Luzi L, Minucci S, Marcaccio M, Pinton P, Rizzuto R, Bernardi P, Paolucci F, Pelicci PG. Electron transfer between cytochrome c and p66Shc generates reactive oxygen species that trigger mitochondrial apoptosis. Cell 2005;122:221-233
-
(2005)
Cell
, vol.122
, pp. 221-233
-
-
Giorgio, M.1
Migliaccio, E.2
Orsini, F.3
Paolucci, D.4
Moroni, M.5
Contursi, C.6
Pelliccia, G.7
Luzi, L.8
Minucci, S.9
Marcaccio, M.10
Pinton, P.11
Rizzuto, R.12
Bernardi, P.13
Paolucci, F.14
Pelicci, P.G.15
-
42
-
-
74249118091
-
Is indeed the prion protein a Harlequin servant of many masters?
-
Sorgato MC, Peggion C, Bertoli A. Is, indeed, the prion protein a Harlequin servant of many masters?. Prion 2009;3:202-205
-
(2009)
Prion
, vol.3
, pp. 202-205
-
-
Sorgato, M.C.1
Peggion, C.2
Bertoli, A.3
-
43
-
-
79959519400
-
Possible role for Ca2+ in the pathophysiology of the prion protein?
-
Peggion C, Bertoli A, Sorgato MC. Possible role for Ca2+ in the pathophysiology of the prion protein?. Biofactors 2011;37:241-249
-
(2011)
Biofactors
, vol.37
, pp. 241-249
-
-
Peggion, C.1
Bertoli, A.2
Sorgato, M.C.3
-
44
-
-
43149109488
-
Prion protein attenuates excitotoxicity by inhibiting NMDA receptors
-
Khosravani H, Zhang Y, Tsutsui S, Hameed S, Altier C, Hamid J, Chen L, Villemaire M, Ali Z, Jirik FR, Zamponi GW. Prion protein attenuates excitotoxicity by inhibiting NMDA receptors. J Cell Biol 2008;181:551-565
-
(2008)
J Cell Biol
, vol.181
, pp. 551-565
-
-
Khosravani, H.1
Zhang, Y.2
Tsutsui, S.3
Hameed, S.4
Altier, C.5
Hamid, J.6
Chen, L.7
Villemaire, M.8
Ali, Z.9
Jirik, F.R.10
Zamponi, G.W.11
-
45
-
-
78049403334
-
Cellular prion protein promotes regeneration of adult muscle tissue
-
Stella R, Massimino ML, Sandri M, Sorgato MC, Bertoli A. Cellular prion protein promotes regeneration of adult muscle tissue. Mol Cell Biol 2010;30:4864-4876
-
(2010)
Mol Cell Biol
, vol.30
, pp. 4864-4876
-
-
Stella, R.1
Massimino, M.L.2
Sandri, M.3
Sorgato, M.C.4
Bertoli, A.5
-
46
-
-
79851481136
-
Cellular prion protein is implicated in the regulation of local Ca2+ movements in cerebellar granule neurons
-
Lazzari C, Peggion C, Stella R, Massimino ML, Lim D, Bertoli A, Sorgato MC. Cellular prion protein is implicated in the regulation of local Ca2+ movements in cerebellar granule neurons. J Neurochem 2011;116:881-890
-
(2011)
J Neurochem
, vol.116
, pp. 881-890
-
-
Lazzari, C.1
Peggion, C.2
Stella, R.3
Massimino, M.L.4
Lim, D.5
Bertoli, A.6
Sorgato, M.C.7
-
47
-
-
51649087972
-
Physiological role of the cellular prion protein (PrPC): Protein profiling study in two cell culture systems
-
Ramljak S, Asif AR, Armstrong VW, Wrede A, Groschup MH, Buschmann A, Schulz-Schaeffer W, Bodemer W, Zerr I. Physiological role of the cellular prion protein (PrPC): protein profiling study in two cell culture systems. J Proteome Res 2008; 7:2681-2695
-
(2008)
J Proteome Res
, vol.7
, pp. 2681-2695
-
-
Ramljak, S.1
Asif, A.R.2
Armstrong, V.W.3
Wrede, A.4
Groschup, M.H.5
Buschmann, A.6
Schulz-Schaeffer, W.7
Bodemer, W.8
Zerr, I.9
-
48
-
-
84856641537
-
Relative quantification of membrane proteins in wild-Type and prion protein (PrP)-knockout cerebellar granule neurons
-
Stella R, Cifani P, Peggion C, Hansson K, Lazzari C, Bendz M, Levander F, Sorgato MC, Bertoli A, James P. Relative quantification of membrane proteins in wild-Type and prion protein (PrP)-knockout cerebellar granule neurons. J Proteome Res 2012;11: 523-536
-
(2012)
J Proteome Res
, vol.11
, pp. 523-536
-
-
Stella, R.1
Cifani, P.2
Peggion, C.3
Hansson, K.4
Lazzari, C.5
Bendz, M.6
Levander, F.7
Sorgato, M.C.8
Bertoli, A.9
James, P.10
-
49
-
-
84874678796
-
A-lipoic acid reduces infarct size and preserves cardiac function in rat myocardial ischemia/ reperfusion injury through activation of pi3k/akt/nrf2 pathway
-
Deng C, Sun Z, Tong G, Yi W, Ma L, Zhao B, Cheng L, Zhang J, Cao F, Yi D. a-Lipoic acid reduces infarct size and preserves cardiac function in rat myocardial ischemia/ reperfusion injury through activation of PI3K/Akt/Nrf2 pathway. PLoS ONE 2013;8: e58371
-
(2013)
PLoS ONE
, vol.8
, pp. e58371
-
-
Deng, C.1
Sun, Z.2
Tong, G.3
Yi, W.4
Ma, L.5
Zhao, B.6
Cheng, L.7
Zhang, J.8
Cao, F.9
Yi, D.10
-
50
-
-
13044304201
-
Nrf2 is essential for protection against acute pulmonary injury in mice
-
Chan K, Kan YW. Nrf2 is essential for protection against acute pulmonary injury in mice. Proc Natl Acad Sci USA 1999;96:12731-12736
-
(1999)
Proc Natl Acad Sci USA
, vol.96
, pp. 12731-12736
-
-
Chan, K.1
Kan, Y.W.2
-
51
-
-
67650229881
-
Nrf2-dependent upregulation of antioxidative enzymes: A novel pathway for proteasome inhibitor-mediated cardioprotection
-
Dreger H, Westphal K, Weller A, Baumann G, Stangl V, Meiners S, Stangl K. Nrf2-dependent upregulation of antioxidative enzymes: a novel pathway for proteasome inhibitor-mediated cardioprotection. Cardiovasc Res 2009;83:354-361
-
(2009)
Cardiovasc Res
, vol.83
, pp. 354-361
-
-
Dreger, H.1
Westphal, K.2
Weller, A.3
Baumann, G.4
Stangl, V.5
Meiners, S.6
Stangl, K.7
-
52
-
-
84880832061
-
Mechanisms of nrf2 protection in astrocytes as identified by quantitative proteomics and sirna screening
-
Dowell JA, Johnson JA. Mechanisms of Nrf2 protection in astrocytes as identified by quantitative proteomics and siRNA screening. PLoS ONE 2013;8:e70163
-
(2013)
PLoS ONE
, vol.8
, pp. e70163
-
-
Dowell, J.A.1
Johnson, J.A.2
|