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Volumn 38, Issue 3, 2002, Pages 328-339

Oxidative stress and the prion protein in transmissible spongiform encephalopathies

Author keywords

Neurodegeneration; Oxidative stress; Prion disease; Signaling pathway

Indexed keywords

3 NITROTYROSINE; ADENOSINE TRIPHOSPHATASE; COPPER; COPPER ZINC SUPEROXIDE DISMUTASE; CUPRIZONE; CYTOCHROME C OXIDASE; FLUPIRTINE; GLUTATHIONE; GLUTATHIONE PEROXIDASE; GLUTATHIONE REDUCTASE; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; ISOPROTEIN; MANGANESE SUPEROXIDE DISMUTASE; MESSENGER RNA; METAL ION; MITOGEN ACTIVATED PROTEIN KINASE; NITRIC OXIDE SYNTHASE; PRION PROTEIN; PROTEIN BAX; PROTEIN BCL 2; PROTEIN KINASE C; SUPEROXIDE DISMUTASE; TUMOR NECROSIS FACTOR ALPHA;

EID: 0036124813     PISSN: 01650173     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0165-0173(01)00150-3     Document Type: Review
Times cited : (165)

References (111)
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    • Ecosystems supporting clusters of sporadic TSEs demonstrate excesses of the radical-generating divalent cation manganese and deficiencies of antioxidant co factors Cu, Se, Fe, Zn. Does a foreign cation substitution at prion protein's Cu domain initiate TSE?
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.