Post-natal knockout of prion protein alters hippocampal CA1 properties, but does not result in neurodegeneration

EMBO Journal

Volumn 21, Issue 3, 2002, Pages 202-210

  Mallucci, G R ^a  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

Author keywords

Afterhyperpolarization potential; Cre loxP system; Prion disease; Prion protein

Indexed keywords

GENE PRODUCT; PRION PROTEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL SURVIVAL; CONTROLLED STUDY; DISEASE MODEL; HIPPOCAMPUS; HISTOPATHOLOGY; HYPERPOLARIZATION; KNOCKOUT GENE; MOUSE; NERVE CELL; NERVE DEGENERATION; NERVE FUNCTION; NONHUMAN; PATHOGENESIS; PHENOTYPE; PRION DISEASE; PRIORITY JOURNAL; PROTEIN DEPLETION; PROTEIN FUNCTION; PROTEIN TARGETING; TRANSGENIC MOUSE;

ACTION POTENTIALS; ANIMALS; CELL SURVIVAL; GENE DELETION; HIPPOCAMPUS; MICE; MICE, TRANSGENIC; NEURONS; PRION DISEASES; PRIONS;

ANIMALIA; HIPPOCAMPUS HIPPOCAMPUS; MUS MUSCULUS;

EID: 0036470471     PISSN: 02614189     EISSN: None     Source Type: Journal    
DOI: 10.1093/emboj/21.3.202     Document Type: Article

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