GLUT4 defects in adipose tissue are early signs of metabolic alterations in alms1GT/GT, a mouse model for obesity and insulin resistance

PLoS ONE

Volumn 9, Issue 10, 2014, Pages

  Favaretto, Francesca ^a   Milan, Gabriella ^a   Collin, Gayle B ^b   Marshall, Jan D ^b   Stasi, Fabio ^a   Maffei, Pietro ^a   Vettor, Roberto ^a   Naggert, Jürgen K ^b  

^a UNIVERSITY OF PADOVA   (Italy)

^b JACKSON LABORATORY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GLUCOSE TRANSPORTER 4; PROTEIN KINASE B; ALMS1 PROTEIN, MOUSE; BICARBONATE CHLORIDE ANTIPORTER; DNA BINDING PROTEIN; GLUCOSE; INSULIN; SLC4A2 PROTEIN, MOUSE;

ADIPOSE TISSUE; ALMS1 GENE; ALSTROM SYNDROME; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL DIFFERENTIATION; CELL EXPANSION; CELL ISOLATION; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME PHOSPHORYLATION; GENE; GENE DISRUPTION; GENE EXPRESSION; GLUCOSE HOMEOSTASIS; GLUCOSE INTOLERANCE; GLUCOSE TRANSPORT; INSULIN RESISTANCE; INSULIN RESPONSE; LIPOGENESIS; MALE; MOUSE; NONHUMAN; OBESITY; PROADIPOCYTE; PROTEIN CONTENT; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; PROTEIN TRANSPORT; SIGNAL TRANSDUCTION; WEIGHT GAIN; WILD TYPE; ADIPOCYTE; ADIPOGENESIS; ANIMAL; BODY WEIGHT; C57BL MOUSE; DEFICIENCY; DISEASE MODEL; DRUG EFFECTS; GENE EXPRESSION REGULATION; GENETICS; HUMAN; HYPERINSULINISM; METABOLISM; PATHOLOGY; PHOSPHORYLATION; PRIMARY CELL CULTURE; TRANSGENIC MOUSE; TRANSPORT AT THE CELLULAR LEVEL;

ADIPOCYTES; ADIPOGENESIS; ADIPOSE TISSUE; ALSTROM SYNDROME; ANIMALS; BIOLOGICAL TRANSPORT; BODY WEIGHT; CHLORIDE-BICARBONATE ANTIPORTERS; DISEASE MODELS, ANIMAL; DNA-BINDING PROTEINS; GENE EXPRESSION REGULATION; GLUCOSE; GLUCOSE INTOLERANCE; HUMANS; HYPERINSULINISM; INSULIN; INSULIN RESISTANCE; MALE; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; PHOSPHORYLATION; PRIMARY CELL CULTURE; SIGNAL TRANSDUCTION;

EID: 84907804616     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0109540     Document Type: Article

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