Selective loss of parvalbumin-positive GABAergic interneurons in the cerebral cortex of maternally stressed Gad1-heterozygous mouse offspring

Translational Psychiatry

Volumn 4, Issue 3, 2014, Pages

  Uchida, T ^a   Furukawa, T ^a   Iwata, S ^a   Yanagawa, Y ^b   Fukuda, A ^a  

^a HAMAMATSU UNIVERSITY SCHOOL OF MEDICINE   (Japan)

^b GUNMA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

Author keywords

GAD67; Glucocorticoid receptor; Neurogenesis; Parvalbumin; Prenatal stress; Schizophrenia

Indexed keywords

4 AMINOBUTYRIC ACID RECEPTOR; BROXURIDINE; GLUCOCORTICOID RECEPTOR; GLUTAMATE DECARBOXYLASE 67; GREEN FLUORESCENT PROTEIN; PARVALBUMIN; GLUTAMATE DECARBOXYLASE; GLUTAMATE DECARBOXYLASE 1;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BRAIN CORTEX; CONTROLLED STUDY; DENSITY; FEMALE; GABAERGIC SYSTEM; GAD1 GENE; GENE; HETEROZYGOSITY; INTERNEURON; MATERNAL STRESS; MOTOR CORTEX; MOUSE; NERVOUS SYSTEM DEVELOPMENT; NONHUMAN; OUTCOME ASSESSMENT; PRENATAL STRESS; PROGENY; PROTEIN EXPRESSION; SOMATOSENSORY CORTEX; STEM CELL; WILD TYPE; ANIMAL; COMPLICATION; EMBRYOLOGY; GENETICS; GENOTYPE ENVIRONMENT INTERACTION; GROWTH, DEVELOPMENT AND AGING; MENTAL STRESS; METABOLISM; PATHOLOGY; PHYSIOLOGY; PREGNANCY; PRENATAL EXPOSURE; RISK FACTOR; TRANSGENIC MOUSE;

ANIMALS; CEREBRAL CORTEX; FEMALE; GABAERGIC NEURONS; GENE-ENVIRONMENT INTERACTION; GLUTAMATE DECARBOXYLASE; INTERNEURONS; MICE; MICE, TRANSGENIC; NEUROGENESIS; PARVALBUMINS; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; RECEPTORS, GLUCOCORTICOID; RISK FACTORS; STRESS, PSYCHOLOGICAL;

EID: 84901273131     PISSN: None     EISSN: 21583188     Source Type: Journal    
DOI: 10.1038/tp.2014.13     Document Type: Article

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