-
1
-
-
0035115801
-
The somatomedin hypothesis: 2001
-
DOI 10.1210/er.22.1.53
-
Le Roith D, Bondy C, Yakar S, Liu JL, Butler A. The somatomedin hypothesis: 2001. Endocr Rev. 2001;22:53-74. (Pubitemid 32187803)
-
(2001)
Endocrine Reviews
, vol.22
, pp. 53-74
-
-
Le Roith, D.1
Bondy, C.2
Yakar, S.3
Liu, J.-L.4
Butler, A.5
-
2
-
-
34548307174
-
Does IGF-I stimulate pancreatic islet cell growth?
-
Liu JL. Does IGF-I stimulate pancreatic islet cell growth? Cell Biochem Biophys. 2007;48:115-125.
-
(2007)
Cell Biochem Biophys
, vol.48
, pp. 115-125
-
-
Liu, J.L.1
-
3
-
-
0028879735
-
Insulin-like growth factor-1 (IGF-1) protectsNODmice from insulitis and diabetes
-
Bergerot I, Fabien N, Maguer V, Thivolet C. Insulin-like growth factor-1 (IGF-1) protectsNODmice from insulitis and diabetes. Clin Exp Immunol. 1995;102:335-340.
-
(1995)
Clin Exp Immunol
, vol.102
, pp. 335-340
-
-
Bergerot, I.1
Fabien, N.2
Maguer, V.3
Thivolet, C.4
-
4
-
-
0031055903
-
Enhanced growth of small bowel in transgenic mice expressing human insulin-like growth factor i
-
DOI 10.1053/gast.1997.v112.pm9024298
-
Ohneda K, UlshenMH,Fuller CR, D'Ercole AJ, Lund PK. Enhanced growth of small bowel in transgenic mice expressing human insulinlike growth factor I. Gastroenterology. 1997;112:444-454. (Pubitemid 27078831)
-
(1997)
Gastroenterology
, vol.112
, pp. 444-454
-
-
Ohneda, K.1
Ulshen, M.H.2
Fuller, C.R.3
D'Ercole, A.J.4
Lund, P.K.5
-
5
-
-
45549108397
-
Ageneral and islet cell-enriched overexpression of IGF-I results in normal islet cell growth, hypoglycemia, and significant resistance to experimental diabetes
-
Robertson K, Lu Y, De Jesus K, et al.Ageneral and islet cell-enriched overexpression of IGF-I results in normal islet cell growth, hypoglycemia, and significant resistance to experimental diabetes. Am J Physiol Endocrinol Metab. 2008;294:E928-E938.
-
(2008)
Am J Physiol Endocrinol Metab.
, vol.294
-
-
Robertson, K.1
Lu, Y.2
De Jesus, K.3
-
6
-
-
33847712503
-
Insulin-like growth factor-1 (IGF-1) induces WISP-2/CCN5 via multiple molecular cross-talks and is essential for mitogenic switch by IGF-1 axis in estrogen receptor-positive breast tumor cells
-
DOI 10.1158/0008-5472.CAN-06-3753
-
Dhar K, Banerjee S, Dhar G, Sengupta K, Banerjee SK. Insulin-like growth factor-1 (IGF-1) induces WISP-2/CCN5 via multiple molecular cross-talks and is essential for mitogenic switch by IGF-1 axis in estrogen receptor-positive breast tumor cells. Cancer Res. 2007; 67:1520-1526. (Pubitemid 46383375)
-
(2007)
Cancer Research
, vol.67
, pp. 1520-1526
-
-
Dhar, K.1
Banerjee, S.2
Dhar, G.3
Sengupta, K.4
Banerjee, S.K.5
-
7
-
-
0034744034
-
COP1, a member of the CCN family, is a heparin-induced growth arrest specific gene in vascular smooth muscle cells
-
DOI 10.1002/jcp.1100
-
Delmolino LM, Stearns NA, Castellot JJ. COP-1, a member of the CCN family, is a heparin-induced growth arrest specific gene in vascular smooth muscle cells. J Cell Physiol. 2001;188:45-55. (Pubitemid 32476495)
-
(2001)
Journal of Cellular Physiology
, vol.188
, pp. 45-55
-
-
Delmolino, L.M.1
Stearns, N.A.2
Castellot Jr., J.J.3
-
8
-
-
38549139109
-
Role of WISP-2/CCN5 in the maintenance of a differentiated and noninvasive phenotype in human breast cancer cells
-
DOI 10.1128/MCB.01335-07
-
Fritah A, Saucier C, De Wever O, et al. Role of WISP-2/CCN5 in the maintenance of a differentiated and noninvasive phenotype in human breast cancer cells. Mol Cell Biol. 2008;28:1114-1123. (Pubitemid 351160067)
-
(2008)
Molecular and Cellular Biology
, vol.28
, pp. 1114-1123
-
-
Fritah, A.1
Saucier, C.2
De Wever, O.3
Bracke, M.4
Bieche, I.5
Lidereau, R.6
Gespach, C.7
Drouot, S.8
Redeuilh, G.9
Sabbah, M.10
-
9
-
-
79959351079
-
First structural glimpse of CCN3andCCN5multifunctional signaling regulators elucidated by small angle X-ray scattering
-
Holbourn KP, Malfois M, Acharya KR. First structural glimpse of CCN3andCCN5multifunctional signaling regulators elucidated by small angle X-ray scattering. J Biol Chem. 2011;286:22243-22249.
-
(2011)
J Biol Chem
, vol.286
, pp. 22243-22249
-
-
Holbourn, K.P.1
Malfois, M.2
Acharya, K.R.3
-
10
-
-
0037392317
-
Proposal for a unified CCN nomenclature
-
DOI 10.1136/mp.56.2.127
-
Brigstock DR, Goldschmeding R, Katsube KI, et al. Proposal for a unified CCN nomenclature. Mol Pathol. 2003;56:127-128. (Pubitemid 36461474)
-
(2003)
Journal of Clinical Pathology - Molecular Pathology
, vol.56
, pp. 127-128
-
-
Brigstock, D.R.1
Goldschmeding, R.2
Katsube, K.-I.3
Lam, S.C.-T.4
Lau, L.F.5
Lyons, K.6
Naus, C.7
Perbal, B.8
Riser, B.9
Takigawa, M.10
Yeger, H.11
-
11
-
-
53149092430
-
The CCN family of proteins: Structure-function relationships
-
Holbourn KP, Acharya KR, Perbal B. The CCN family of proteins: structure-function relationships. Trends Biochem Sci. 2008;33: 461-473.
-
(2008)
Trends Biochem Sci.
, vol.33
, pp. 461-473
-
-
Holbourn, K.P.1
Acharya, K.R.2
Perbal, B.3
-
12
-
-
84874256781
-
Regulation of pancreatic function by connective tissue growth factor (CTGF, CCN2)
-
Charrier A, Brigstock DR. Regulation of pancreatic function by connective tissue growth factor (CTGF, CCN2). Cytokine Growth Factor Rev. 2013;24:59-68.
-
(2013)
Cytokine Growth Factor Rev
, vol.24
, pp. 59-68
-
-
Charrier, A.1
Brigstock, D.R.2
-
13
-
-
84866403676
-
EconomidesAN,GannonM.Inactivation of the dual Bmp/Wnt inhibitor Sostdc1 enhances pancreatic islet function
-
HenleyKD,Gooding KA, EconomidesAN,GannonM.Inactivation of the dual Bmp/Wnt inhibitor Sostdc1 enhances pancreatic islet function. Am J Physiol Endocrinol Metab. 2012;303:E752-E761.
-
(2012)
Am J Physiol Endocrinol Metab.
, vol.303
-
-
Henley, K.D.1
Gooding, K.A.2
-
14
-
-
80053059611
-
Connective tissue growth factor acts within both endothelial cells and βcells to promote proliferation of developing βcells
-
GuneyMA,Petersen CP, Boustani A, et al. Connective tissue growth factor acts within both endothelial cells and βcells to promote proliferation of developing βcells. Proc Natl Acad Sci USA. 2011;108: 15242-15247.
-
(2011)
Proc Natl Acad Sci USA
, vol.108
, pp. 15242-15247
-
-
Guney, M.A.1
Petersen, C.P.2
Boustani, A.3
-
15
-
-
61449178818
-
Connective tissue growth factor (CTGF) inactivation leads to defects in islet cell lineage allocation and βcells proliferation during embryogenesis
-
Crawford LA, Guney MA, Oh YA, et al. Connective tissue growth factor (CTGF) inactivation leads to defects in islet cell lineage allocation and βcells proliferation during embryogenesis. Mol Endocrinol. 2009;23:324-336.
-
(2009)
Mol Endocrinol
, vol.23
, pp. 324-336
-
-
Crawford, L.A.1
Guney, M.A.2
Oh, Y.A.3
-
16
-
-
79955439452
-
Pancreatic islet-specific overexpression of Reg3β protein induced the expression of pro-islet genes and protected mice against streptozotocin-induced diabetes mellitus
-
Xiong X, Wang X, Li B, et al. Pancreatic islet-specific overexpression of Reg3 β protein induced the expression of pro-islet genes and protected mice against streptozotocin-induced diabetes mellitus. Am J Physiol Endocrinol Metab. 2011;300:E669-E680.
-
(2011)
Am J Physiol Endocrinol Metab.
, vol.300
-
-
Xiong, X.1
Wang, X.2
Li, B.3
-
17
-
-
70349515011
-
The CB1 antagonist rimonabant decreases insulin hypersecretion in rat pancreatic islets
-
Getty-Kaushik L, Richard AM, Deeney JT, Krawczyk S, Shirihai O, Corkey BE. The CB1 antagonist rimonabant decreases insulin hypersecretion in rat pancreatic islets. Obesity (Silver Spring). 2009; 17:1856-1860.
-
(2009)
Obesity (Silver Spring)
, vol.17
, pp. 1856-1860
-
-
Getty-Kaushik, L.1
Richard, A.M.2
Deeney, J.T.3
Krawczyk, S.4
Shirihai, O.5
Corkey, B.E.6
-
18
-
-
9444293983
-
Pancreatic-specific inactivation of IGF-I gene causes enlarged pancreatic islets and significant resistance to diabetes
-
DOI 10.2337/diabetes.53.12.3131
-
Lu Y, Herrera PL, Guo Y, et al. Pancreatic-specific inactivation of IGF-I gene causes enlarged pancreatic islets and significant resistance to diabetes. Diabetes. 2004;53:3131-3141. (Pubitemid 39564488)
-
(2004)
Diabetes
, vol.53
, pp. 3131-3141
-
-
Lu, Y.1
Herrera, P.L.2
Guo, Y.3
Sun, D.4
Tang, Z.5
Leroith, D.6
Liu, J.-L.7
-
19
-
-
33745851489
-
Activation of the Reg family genes by pancreatic-specific IGF-I gene deficiency and after streptozotocin-induced diabetes in mouse pancreas
-
DOI 10.1152/ajpendo.00596.2005
-
Lu Y, Ponton A, Okamoto H, Takasawa S, Herrera PL, Liu JL. Activation of the Reg family genes by pancreatic-specific IGF-I gene deficiency and after streptozotocin-induced diabetes in mouse pancreas. Am J Physiol Endocrinol Metab. 2006;291:E50-E58. (Pubitemid 44035309)
-
(2006)
American Journal of Physiology - Endocrinology and Metabolism
, vol.291
-
-
Lu, Y.1
Ponton, A.2
Okamoto, H.3
Takasawa, S.4
Herrera, P.L.5
Liu, J.-L.6
-
20
-
-
77957787939
-
Reg2 protects mouse insulinoma cells from streptozotocin-induced mitochondrial disruption and apoptosis
-
Liu L, Liu JL, Srikant CB. Reg2 protects mouse insulinoma cells from streptozotocin-induced mitochondrial disruption and apoptosis. Growth Factors. 2010;28:370-378.
-
(2010)
Growth Factors
, vol.28
, pp. 370-378
-
-
Liu, L.1
Liu, J.L.2
Srikant, C.B.3
-
21
-
-
0029048139
-
Apoptosis during HL-60 cell differentiation is closely related to aG0/G1 cell cycle arrest
-
doi: 10.1210/en.2013-1735
-
Terui Y, Furukawa Y, Kikuchi J, Saito M. Apoptosis during HL-60 cell differentiation is closely related to aG0/G1 cell cycle arrest. J Cell Physiol. 1995;164:74-84. doi: 10.1210/en.2013-1735
-
(1995)
J Cell Physiol
, vol.164
, pp. 74-84
-
-
Terui, Y.1
Furukawa, Y.2
Kikuchi, J.3
Saito, M.4
-
23
-
-
34447297824
-
Loss of WISP-2/CCN5 signaling in human pancreatic cancer: A potential mechanism for epithelial-mesenchymal-transition
-
DOI 10.1016/j.canlet.2007.02.012, PII S0304383507000675
-
Dhar G, Mehta S, Banerjee S, et al. Loss of WISP-2/CCN5 signaling in human pancreatic cancer: a potential mechanism for epithelialmesenchymal- transition. Cancer Lett. 2007;254:63-70. (Pubitemid 47058806)
-
(2007)
Cancer Letters
, vol.254
, pp. 63-70
-
-
Dhar, G.1
Mehta, S.2
Banerjee, S.3
Gardner, A.4
McCarty, B.M.5
Mathur, S.C.6
Campbell, D.R.7
Kambhampati, S.8
Banerjee, S.K.9
-
24
-
-
0347990598
-
Induction of β-Cell proliferation and retinoblastoma protein phosphorylation in rat and human islets using adenovirus-mediated transfer of cyclin-dependent kinase-4 and cyclin D1
-
DOI 10.2337/diabetes.53.1.149
-
Cozar-Castellano I, Takane KK, Bottino R, Balamurugan AN, Stewart AF. Induction of βcells proliferation and retinoblastoma protein phosphorylation in rat and human islets using adenovirusmediated transfer of cyclin-dependent kinase-4 and cyclin D1. Diabetes. 2004;53:149-159. (Pubitemid 38044709)
-
(2004)
Diabetes
, vol.53
, pp. 149-159
-
-
Cozar-Castellano, I.1
Takane, K.K.2
Bottino, R.3
Balamurugan, A.N.4
Stewart, A.F.5
-
25
-
-
33644754585
-
Akt induces β-cell proliferation by regulating cyclin D1, cyclin D2, and p21 levels and cyclin-dependent kinase-4 activity
-
DOI 10.2337/diabetes.55.02.06.db05-0757
-
Fatrai S, Elghazi L, Balcazar N, et al. Akt inducesβcells proliferation by regulating cyclin D1, cyclin D2, and p21 levels and cyclin-dependent kinase-4 activity. Diabetes. 2006;55:318-325. (Pubitemid 43343259)
-
(2006)
Diabetes
, vol.55
, pp. 318-325
-
-
Fatrai, S.1
Elghazi, L.2
Balcazar, N.3
Cras-Meneur, C.4
Krits, I.5
Kiyokawa, H.6
Bernal-Mizrachi, E.7
-
26
-
-
84873712772
-
WISP2 regulates preadipocyte commitment and PPARγ activation by BMP4
-
Hammarstedt A, Hedjazifar S, Jenndahl L, et al. WISP2 regulates preadipocyte commitment and PPARγ activation by BMP4. Proc Natl Acad Sci USA. 2013;110:2563-2568.
-
(2013)
Proc Natl Acad Sci USA
, vol.110
, pp. 2563-2568
-
-
Hammarstedt, A.1
Hedjazifar, S.2
Jenndahl, L.3
-
27
-
-
79953780809
-
CCN5, a novel transcriptional repressor of the transforming growth factor β signaling pathway
-
Sabbah M, Prunier C, Ferrand N, et al. CCN5, a novel transcriptional repressor of the transforming growth factor β signaling pathway. Mol Cell Biol. 2011;31:1459-1469.
-
(2011)
Mol Cell Biol
, vol.31
, pp. 1459-1469
-
-
Sabbah, M.1
Prunier, C.2
Ferrand, N.3
-
28
-
-
77954675497
-
CCN5, a secreted protein, localizes to the nucleus
-
Wiesman KC, Wei L, Baughman C, Russo J, Gray MR, Castellot JJ. CCN5, a secreted protein, localizes to the nucleus. J Cell Commun Signal. 2010;4:91-98.
-
(2010)
J Cell Commun Signal
, vol.4
, pp. 91-98
-
-
Wiesman, K.C.1
Wei, L.2
Baughman, C.3
Russo, J.4
Gray, M.R.5
Castellot, J.J.6
-
29
-
-
3543049948
-
Identification of rCop-1, a new member of the CCN protein family, as a negative regulator for cell transformation
-
Zhang R, Averboukh L, Zhu W, et al. Identification of rCop-1, a new member of the CCN protein family, as a negative regulator for cell transformation. Mol Cell Biol. 1998;18:6131-6141. (Pubitemid 28450564)
-
(1998)
Molecular and Cellular Biology
, vol.18
, pp. 6131-6141
-
-
Zhang, K.1
Averboukh, L.2
Zhu, W.3
Zhang, H.4
Jo, H.5
Dempsey, P.J.6
Coffey, R.J.7
Pardee, A.B.8
Liang, P.9
-
30
-
-
79961126182
-
Wisp2/CCN5 up-regulated in the central nervous system of GM3-only mice facilitates neurite formation in Neuro2a cells via integrin-Akt signaling
-
Ohkawa Y, Ohmi Y, Tajima O, Yamauchi Y, Furukawa K, Furukawa K. Wisp2/CCN5 up-regulated in the central nervous system of GM3-only mice facilitates neurite formation in Neuro2a cells via integrin-Akt signaling. Biochem Biophys Res Commun. 2011;411: 483-489.
-
(2011)
Biochem Biophys Res Commun
, vol.411
, pp. 483-489
-
-
Ohkawa, Y.1
Ohmi, Y.2
Tajima, O.3
Yamauchi, Y.4
Furukawa, K.5
Furukawa, K.6
-
32
-
-
62849114529
-
Regulation of phosphoinositide 3-kinase expression in health and disease
-
Kok K, Geering B, Vanhaesebroeck B. Regulation of phosphoinositide 3-kinase expression in health and disease. Trends Biochem Sci. 2009;34:115-127.
-
(2009)
Trends Biochem Sci
, vol.34
, pp. 115-127
-
-
Kok, K.1
Geering, B.2
Vanhaesebroeck, B.3
-
33
-
-
0032571317
-
A novel class II phosphoinositide 3-kinase predominantly expressed in the liver and its enhanced expression during liver regeneration
-
DOI 10.1074/jbc.273.13.7731
-
Ono F, Nakagawa T, Saito S, et al.Anovel class II phosphoinositide 3-kinase predominantly expressed in the liver and its enhanced expression during liver regeneration. J Biol Chem. 1998;273:7731- 7736. (Pubitemid 28152804)
-
(1998)
Journal of Biological Chemistry
, vol.273
, pp. 7731-7736
-
-
Ono, F.1
Nakagawa, T.2
Saito, S.3
Owada, Y.4
Sakagami, H.5
Goto, K.6
Suzuki, M.7
Matsuno, S.8
Kondo, H.9
-
34
-
-
0034455322
-
CCN proteins are distinct from and should not be considered members of the insulin-like growth factor-binding protein superfamily
-
DOI 10.1210/en.141.6.2254
-
Grotendorst GR, Lau LF, Perbal B. CCN proteins are distinct from and should not be considered members of the insulin-like growth factor-binding protein superfamily. Endocrinology. 2000;141: 2254-2256. (Pubitemid 32274380)
-
(2000)
Endocrinology
, vol.141
, pp. 2254-2256
-
-
Grotendorst, G.R.1
Lau, L.F.2
Perbal, B.3
-
35
-
-
20444504753
-
Structural and functional properties of CCN proteins
-
DOI 10.1016/S0083-6729(05)70003-0, PII S0083672905700030
-
Rachfal AW, Brigstock DR. Structural and functional properties of CCN proteins. Vitam Horm. 2005;70:69-103. (Pubitemid 43589023)
-
(2005)
Vitamins and Hormones
, vol.70
, pp. 69-103
-
-
Rachfal, A.W.1
Brigstock, D.R.2
-
36
-
-
0036931171
-
Identification of differentially expressed mRNA during pancreas regeneration of rat by mRNA differential display
-
DOI 10.1016/S0006-291X(02)02741-9, PII S0006291X02027419
-
Lim HW, Lee JE, Shin SJ, et al. Identification of differentially expressed mRNA during pancreas regeneration of rat by mRNA differential display. Biochem Biophys Res Commun. 2002;299:806- 812. (Pubitemid 36034558)
-
(2002)
Biochemical and Biophysical Research Communications
, vol.299
, pp. 806-812
-
-
Lim, H.W.1
Lee, J.E.2
Shin, S.J.3
Lee, Y.E.4
Oh, S.H.5
Park, J.Y.6
Seong, J.K.7
Park, J.-S.8
-
37
-
-
84866386719
-
Glucocorticoids induce CCN5/WISP-2 expression and attenuate invasion in oestrogen receptor-negative human breast cancer cells
-
Ferrand N, Stragier E, Redeuilh G, Sabbah M. Glucocorticoids induce CCN5/WISP-2 expression and attenuate invasion in oestrogen receptor-negative human breast cancer cells. Biochem J. 2012;447: 71-79.
-
(2012)
Biochem J
, vol.447
, pp. 71-79
-
-
Ferrand, N.1
Stragier, E.2
Redeuilh, G.3
Sabbah, M.4
-
38
-
-
0037251255
-
WISP-2 gene in human breast cancer: Estrogen and progesterone inducible expression and regulation of tumor cell proliferation
-
Banerjee S, Saxena N, Sengupta K, Tawfik O, Mayo MS, Banerjee SK. WISP-2 gene in human breast cancer: estrogen and progesterone inducible expression and regulation of tumor cell proliferation. Neoplasia. 2003;5:63-73. (Pubitemid 36151059)
-
(2003)
Neoplasia
, vol.5
, pp. 63-73
-
-
Banerjee, S.1
Saxena, N.2
Sengupta, K.3
Tawfik, O.4
Mayo, M.S.5
Banerjee, S.K.6
-
39
-
-
33846820239
-
Molecular cloning and characterization of the human WISP-2/CCN5 gene promoter reveal its upregulation by oestrogens
-
DOI 10.1677/joe.1.07009
-
Fritah A, Redeuilh G, Sabbah M. Molecular cloning and characterization of the human WISP-2/CCN5 gene promoter reveal its upregulation by oestrogens. J Endocrinol. 2006;191:613-624. (Pubitemid 46210287)
-
(2006)
Journal of Endocrinology
, vol.191
, pp. 613-624
-
-
Fritah, A.1
Redeuilh, G.2
Sabbah, M.3
-
40
-
-
17144365869
-
Epidermal growth factor induces WISP-2/CCN5 expression in estrogen receptor-γ-positive breast tumor cells through multiple molecular cross-talks
-
DOI 10.1158/1541-7786.MCR-04-0130
-
Banerjee S, Sengupta K, Saxena NK, Dhar K, Banerjee SK. Epidermal growth factor induces WISP-2/CCN5 expression in estrogen receptor-γ-positive breast tumor cells through multiple molecular cross-talks. Mol Cancer Res. 2005;3:151-162. (Pubitemid 40524898)
-
(2005)
Molecular Cancer Research
, vol.3
, pp. 151-162
-
-
Banerjee, S.1
Sengupta, K.2
Saxena, N.K.3
Dhar, K.4
Banerjee, S.K.5
-
41
-
-
33748281220
-
WISP-2/CCN5 is involved as a novel signaling intermediate in phorbol ester-protein kinase Cα-mediated breast tumor cell proliferation
-
DOI 10.1021/bi060888p
-
Sengupta K, Banerjee S, Dhar K, et al. WISP-2/CCN5 is involved as a novel signaling intermediate in phorbol ester-protein kinase Cα mediated breast tumor cell proliferation. Biochemistry. 2006;45: 10698-10709. (Pubitemid 44320485)
-
(2006)
Biochemistry
, vol.45
, pp. 10698-10709
-
-
Sengupta, K.1
Banerjee, S.2
Dhar, K.3
Saxena, N.K.4
Mehta, S.5
Campbell, D.R.6
Banerjee, S.K.7
-
42
-
-
13044295427
-
WISP genes are members of the connective tissue growth factor family that are up-regulated in Wnt-1-transformed cells and aberrantly expressed in human colon tumors
-
DOI 10.1073/pnas.95.25.14717
-
Pennica D, Swanson TA, Welsh JW, et al. WISP genes are members of the connective tissue growth factor family that are up-regulated in Wnt-1-transformed cells and aberrantly expressed in human colon tumors. Proc Natl Acad Sci. 1998;95:14717-14722. (Pubitemid 29003696)
-
(1998)
Proceedings of the National Academy of Sciences of the United States of America
, vol.95
, pp. 14717-14722
-
-
Pennica, D.1
Swanson, T.A.2
Welsh, J.W.3
Roy, M.A.4
Lawrence, D.A.5
Lee, J.6
Brush, J.7
Taneyhill, L.A.8
Deuel, B.9
Lew, M.10
Watanabe, C.11
Cohen, R.L.12
Melhem, M.F.13
Finley, G.G.14
Quirke, P.15
Goddard, A.D.16
Hillan, K.J.17
Gurney, A.L.18
Botstein, D.19
Levine, A.J.20
more..
-
43
-
-
49549108896
-
Wnt and beyond Wnt: Multiple mechanisms control the transcriptional property of beta-catenin
-
Jin T, George Fantus I, Sun J. Wnt and beyond Wnt: multiple mechanisms control the transcriptional property of beta-catenin. Cell Signal. 2008;20:1697-1704.
-
(2008)
Cell Signal
, vol.20
, pp. 1697-1704
-
-
Jin, T.1
George Fantus, I.2
Sun, J.3
-
44
-
-
84867191630
-
The involvement of the wnt signaling pathway and TCF7L2 in diabetes mellitus: The current understanding, dispute, and perspective
-
Ip W, Chiang YT, Jin T. The involvement of the wnt signaling pathway and TCF7L2 in diabetes mellitus: the current understanding, dispute, and perspective. Cell Biosci. 2012;2:28.
-
(2012)
Cell Biosci.
, vol.2
, pp. 28
-
-
Ip, W.1
Chiang, Y.T.2
Jin, T.3
-
45
-
-
0037221550
-
CCN5 is a growth arrest-specific gene that regulates smooth muscle cell proliferation and motility
-
Lake AC, Bialik A, Walsh K, Castellot JJ Jr. CCN5 is a growth arrest-specific gene that regulates smooth muscle cell proliferation and motility. Am J Pathol. 2003;162:219-231. (Pubitemid 36042004)
-
(2003)
American Journal of Pathology
, vol.162
, pp. 219-231
-
-
Lake, A.C.1
Bialik, A.2
Walsh, K.3
Castellot Jr., J.J.4
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