Nemo-like kinase (NLK) negatively regulates NF-kappa B activity through disrupting the interaction of TAK1 with IKKβ

Biochimica et Biophysica Acta - Molecular Cell Research

Volumn 1843, Issue 7, 2014, Pages 1365-1372

  Li, Shang Ze ^a   Zhang, Hui Hui ^a   Liang, Jun Bo ^b   Song, Yang ^a   Jin, Bing Xue ^a   Xing, Na Na ^a   Fan, Guo Chang ^c   Du, Run Lei ^a   Zhang, Xiao Dong ^a  

^a WUHAN UNIVERSITY   (China)

^b TSINGHUA UNIVERSITY   (China)

^c UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

IKK ; NF B; NLK; TAK1; TNF

Indexed keywords

I KAPPA B ALPHA; I KAPPA B KINASE BETA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; NEMO LIKE KINASE; PROTEIN SERINE THREONINE KINASE; SYNAPTOTAGMIN I; TRANSCRIPTION FACTOR RELA; TRANSFORMING GROWTH FACTOR BETA ACTIVATED KINASE 1; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG; I KAPPA B KINASE; IKBKB PROTEIN, HUMAN; MAP KINASE KINASE KINASE 7; MITOGEN ACTIVATED PROTEIN KINASE KINASE KINASE; NLK PROTEIN, HUMAN; PROTEIN BINDING; SIGNAL PEPTIDE; SMALL INTERFERING RNA;

ARTICLE; CELL STRAIN HCT116; CELL STRAIN HEK293; CONTROLLED STUDY; EMBRYO; ENZYME DEGRADATION; ENZYME INHIBITION; ENZYME PHOSPHORYLATION; FEMALE; GENE INACTIVATION; GENE OVEREXPRESSION; HELA CELL; HUMAN; HUMAN CELL; NUCLEAR LOCALIZATION SIGNAL; PRIORITY JOURNAL; PROTEIN ASSEMBLY; PROTEIN PROTEIN INTERACTION; REGULATORY MECHANISM; SIGNAL TRANSDUCTION; TRANSCRIPTION INITIATION; ANTAGONISTS AND INHIBITORS; CELL NUCLEUS; DRUG EFFECTS; GENE EXPRESSION REGULATION; GENETICS; HCT116 CELL LINE; HEK293 CELL LINE; METABOLISM; PHOSPHORYLATION; PROTEIN DEGRADATION;

CELL NUCLEUS; GENE EXPRESSION REGULATION; HCT116 CELLS; HEK293 CELLS; HUMANS; I-KAPPA B KINASE; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MAP KINASE KINASE KINASES; NF-KAPPA B; PHOSPHORYLATION; PROTEIN BINDING; PROTEIN-SERINE-THREONINE KINASES; PROTEOLYSIS; RNA, SMALL INTERFERING; SIGNAL TRANSDUCTION; TUMOR NECROSIS FACTOR-ALPHA;

EID: 84899083046     PISSN: 01674889     EISSN: 18792596     Source Type: Journal    
DOI: 10.1016/j.bbamcr.2014.03.028     Document Type: Article

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