Genetic ablation of receptor for advanced glycation end products promotes functional recovery in mouse model of spinal cord injury

Molecular and Cellular Biochemistry

Volumn 390, Issue 1-2, 2014, Pages 215-223

  Guo, Ji Dong ^a   Li, Li ^a   Shi, Ya Min ^a   Wang, Hua Dong ^a   Yuan, Yan Li ^a   Shi, Xiu Xiu ^a   Hou, Shu Xun ^a  

^a CHINESE PLA GENERAL HOSPITAL   (China)

Author keywords

Apoptosis; Inflammation; Nitrosative stress; Oxidative stress; Receptor for advanced glycation end products; Spinal cord injury

Indexed keywords

ADVANCED GLYCOSYLATION END-PRODUCT RECEPTOR; CASPASE 3; IMMUNOGLOBULIN RECEPTOR; INDUCIBLE NITRIC OXIDE SYNTHASE; INTERLEUKIN 6; NOS2 PROTEIN, MOUSE; REACTIVE OXYGEN METABOLITE;

ANIMAL; BIOSYNTHESIS; CONVALESCENCE; DISEASE MODEL; GENE EXPRESSION REGULATION; GENETICS; HUMAN; INFLAMMATION; METABOLISM; MOUSE; OXIDATIVE STRESS; PATHOLOGY; SPINAL CORD INJURY;

ANIMALS; CASPASE 3; DISEASE MODELS, ANIMAL; GENE EXPRESSION REGULATION; HUMANS; INFLAMMATION; INTERLEUKIN-6; MICE; NITRIC OXIDE SYNTHASE TYPE II; OXIDATIVE STRESS; REACTIVE OXYGEN SPECIES; RECEPTORS, IMMUNOLOGIC; RECOVERY OF FUNCTION; SPINAL CORD INJURIES;

EID: 84898904071     PISSN: 03008177     EISSN: 15734919     Source Type: Journal    
DOI: 10.1007/s11010-014-1972-z     Document Type: Article

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