NF-Y inactivation causes atypical neurodegeneration characterized by ubiquitin and p62 accumulation and endoplasmic reticulum disorganization

Nature Communications

Volumn 5, Issue , 2014, Pages

  Yamanaka, Tomoyuki ^a,b,c   Tosaki, Asako ^b,c   Kurosawa, Masaru ^a,b,c   Matsumoto, Gen ^a,b,c   Koike, Masato ^a   Uchiyama, Yasuo ^a   Maity, Sankar N ^d   Shimogori, Tomomi ^b   Hattori, Nobutaka ^a   Nukina, Nobuyuki ^a,b,c  

^a JUNTENDO UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

^b RIKEN BRAIN SCIENCE INSTITUTE   (Japan)

^c JAPAN SCIENCE AND TECHNOLOGY AGENCY   (Japan)

^d UNIVERSITY OF TEXAS MD ANDERSON CANCER CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GLUCOSE REGULATED PROTEIN 94; NUCLEAR FACTOR Y; PROTEIN P62; UBIQUITIN; CCAAT BINDING FACTOR; GLUCOSE-REGULATED PROTEINS; HEAT SHOCK PROTEIN; HEAT SHOCK PROTEIN 70; MEMBRANE PROTEIN; SIGNAL TRANSDUCING ADAPTOR PROTEIN; SQSTM1 PROTEIN, MOUSE;

CYTOLOGY; IMMUNOASSAY; NEUROLOGY; PHYSIOLOGICAL RESPONSE; PROTEIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN NERVE CELL; CHROMATIN IMMUNOPRECIPITATION; CONTROLLED STUDY; ENDOPLASMIC RETICULUM; ENDOPLASMIC RETICULUM STRESS; FEMALE; GENE DELETION; MALE; MITOSIS; MOUSE; NERVE DEGENERATION; NEUROPATHOLOGY; NONHUMAN; PROTEIN AGGREGATION; PROTEIN TARGETING; REGULATORY MECHANISM; ANIMAL; C57BL MOUSE; DEGENERATIVE DISEASE; GENETICS; METABOLISM; PHYSIOLOGY; TUMOR CELL LINE;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; ANIMALS; CCAAT-BINDING FACTOR; CELL LINE, TUMOR; ENDOPLASMIC RETICULUM; ENDOPLASMIC RETICULUM STRESS; FEMALE; HEAT-SHOCK PROTEINS; HSP70 HEAT-SHOCK PROTEINS; MALE; MEMBRANE PROTEINS; MICE; MICE, INBRED C57BL; NEURODEGENERATIVE DISEASES; UBIQUITIN;

EID: 84896842696     PISSN: None     EISSN: 20411723     Source Type: Journal    
DOI: 10.1038/ncomms4354     Document Type: Article

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