HDLs protect the MIN6 insulinoma cell line against tunicamycin-induced apoptosis without inhibiting ER stress and without restoring ER functionality

Molecular and Cellular Endocrinology

Volumn 381, Issue 1-2, 2013, Pages 291-301

  Puyal, Julien ^a   Pétremand, Jannick ^a   Dubuis, Gilles ^a   Rummel, Coralie ^a   Widmann, Christian ^a  

^a UNIVERSITY OF LAUSANNE   (Switzerland)

Author keywords

Apoptosis; Endoplasmic reticulum; HDL; Pancreatic beta cell; Tunicamycin

Indexed keywords

HIGH DENSITY LIPOPROTEIN; PROTEIN KINASE B; STRESS ACTIVATED PROTEIN KINASE; TUNICAMYCIN;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL PROTECTION; CONTROLLED STUDY; ENDOPLASMIC RETICULUM STRESS; MOUSE; NONHUMAN; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; PROTEIN FOLDING; PROTEIN TRANSPORT;

APOPTOSIS; ENDOPLASMIC RETICULUM; HDL; PANCREATIC BETA CELL; TUNICAMYCIN;

ANIMALS; APOPTOSIS; CELL LINE, TUMOR; CYTOPROTECTION; DNA-BINDING PROTEINS; ENDOPLASMIC RETICULUM; ENDOPLASMIC RETICULUM STRESS; EXTRACELLULAR SIGNAL-REGULATED MAP KINASES; HEAT-SHOCK PROTEINS; HEK293 CELLS; HUMANS; INSULIN-SECRETING CELLS; JNK MITOGEN-ACTIVATED PROTEIN KINASES; LIPOPROTEINS, HDL; MAP KINASE SIGNALING SYSTEM; MICE; PHOSPHORYLATION; PROTEIN PROCESSING, POST-TRANSLATIONAL; PROTO-ONCOGENE PROTEINS C-AKT; THAPSIGARGIN; TRANSCRIPTION FACTOR CHOP; TRANSCRIPTION FACTORS; TUNICAMYCIN;

EID: 84884249306     PISSN: 03037207     EISSN: 18728057     Source Type: Journal    
DOI: 10.1016/j.mce.2013.08.016     Document Type: Article

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