Dopamine prevents lipid peroxidation-induced accumulation of toxic α-synuclein oligomers by preserving autophagy-lysosomal function

Frontiers in Cellular Neuroscience

Volumn , Issue MAY, 2013, Pages

  Jiang, Peizhou ^a   Gan, Ming ^a   Yen, Shu Hui C ^a  

^a MAYO CLINIC   (United States)

Author keywords

synuclein; Aggregation; Autophagy; Dopamine; Parkinson's disease

Indexed keywords

ALPHA SYNUCLEIN; ARACHIDONIC ACID; CASPASE 3; DOPAMINE; DOPAMINE TRANSPORTER; POLYUNSATURATED FATTY ACID; REACTIVE OXYGEN METABOLITE; RETINOIC ACID; TETRACYCLINE;

ARTICLE; AUTOOXIDATION; AUTOPHAGY; BIOCHEMISTRY; CELL LOSS; CELL PROTECTION; CELL SURVIVAL; CELL VIABILITY; CONCENTRATION RESPONSE; CONTROLLED STUDY; CORPUS STRIATUM; CYTOTOXICITY; DOPAMINERGIC NERVE CELL; ENZYME ACTIVATION; GENE OVEREXPRESSION; HISTOPATHOLOGY; HUMAN; HUMAN CELL; LEWY BODY; LIPID PEROXIDATION; LYSOSOME; NERVE CELL CULTURE; NERVE CELL DIFFERENTIATION; PARKINSON DISEASE; PATHOGENESIS; PROTEIN ASSEMBLY; PROTEIN EXPRESSION; PROTEIN INTERACTION; SUBSTANTIA NIGRA; WILD TYPE;

EID: 84878738135     PISSN: 16625102     EISSN: None     Source Type: Journal    
DOI: 10.3389/fncel.2013.00081     Document Type: Article

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