Effects of compound heterozygosity at the Xpd locus on cancer and ageing in mouse models

DNA Repair

Volumn 11, Issue 11, 2012, Pages 874-883

  van de Ven, Marieke ^a,d   Andressoo, Jaan Olle ^b   van der Horst, Gijsbertus T J ^a   Hoeijmakers, Jan H J ^a   Mitchell, James R ^c  

^a ERASMUS MEDICAL CENTER   (Netherlands)

^b UNIVERSITY OF HELSINKI   (Finland)

^c HARVARD SCHOOL OF PUBLIC HEALTH   (United States)

^d NETHERLANDS CANCER INSTITUTE   (Netherlands)

Author keywords

Cancer; Cockayne syndrome; Compound heterozygosity; Nucleotide excision repair; Trichothiodystrophy; Xeroderma pigmentosum

Indexed keywords

HELICASE;

AGING; ALLELE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL PROLIFERATION; DNA DAMAGE; GENE LOCUS; GENOTYPE; HETEROZYGOSITY; HOMOZYGOSITY; MOUSE; MUTANT; NEOPLASM; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; SKIN CANCER; SUN EXPOSURE; SUNBURN; TRICHOTHIODYSTROPHY; ULTRAVIOLET IRRADIATION; XERODERMA PIGMENTOSUM;

AGING; ALLELES; ANIMALS; BODY WEIGHT; COCKAYNE SYNDROME; DISEASE MODELS, ANIMAL; DNA DAMAGE; GENES, DOMINANT; HETEROZYGOTE; INSULIN RESISTANCE; MICE; MICE, INBRED C57BL; PHENOTYPE; SKIN NEOPLASMS; TRICHOTHIODYSTROPHY SYNDROMES; ULTRAVIOLET RAYS; XERODERMA PIGMENTOSUM; XERODERMA PIGMENTOSUM GROUP D PROTEIN;

MUS;

EID: 84868206689     PISSN: 15687864     EISSN: 15687856     Source Type: Journal    
DOI: 10.1016/j.dnarep.2012.08.003     Document Type: Article

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