Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis

European Journal of Immunology

Volumn 42, Issue 10, 2012, Pages 2574-2583

  Tu, Eric ^a   Ang, Desmond K Y ^a   Bellingham, Shayne A ^a   Hogan, Thea V ^a,d   Teng, Michele W L ^b,c   Smyth, Mark J ^b,c   Hill, Andrew F ^a   van Driel, Ian R ^a  

^a UNIVERSITY OF MELBOURNE   (Australia)

^b PETER MACCALLUM CANCER CENTRE   (Australia)

^c UNIVERSITY OF MELBOURNE   (Australia)

^d NATIONAL INSTITUTE FOR MEDICAL RESEARCH   (United Kingdom)

Author keywords

Autoimmune disease; Cytokines; IFN ; IL 17; Immunopathology; T cells

Indexed keywords

GAMMA INTERFERON; INTERLEUKIN 10; INTERLEUKIN 17; MESSENGER RNA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; AUTOIMMUNE GASTRITIS; CD4+ T LYMPHOCYTE; CD8+ T LYMPHOCYTE; CELL EXPANSION; CONTROLLED STUDY; CYTOKINE PRODUCTION; DISEASE COURSE; DISEASE SEVERITY; GASTRITIS; LYMPH NODE; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; WILD TYPE;

ADOPTIVE TRANSFER; ANIMALS; AUTOIMMUNE DISEASES; CD4-POSITIVE T-LYMPHOCYTES; CD8-POSITIVE T-LYMPHOCYTES; CELLS, CULTURED; DISEASE PROGRESSION; GASTRITIS; INTERFERON-GAMMA; INTERLEUKIN-17; MICE; MICE, INBRED BALB C; MICE, KNOCKOUT; TH17 CELLS;

EID: 84867210895     PISSN: 00142980     EISSN: 15214141     Source Type: Journal    
DOI: 10.1002/eji.201142341     Document Type: Article

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