Pathophysiological role of omega pore current in channelopathies

Frontiers in Pharmacology

Volumn 3 JUN, Issue , 2012, Pages

  Jurkat Rott, Karin ^a   Groome, James ^b   Lehmann Horn, Frank ^a  

^a UNIVERSITY OF ULM   (Germany)

^b Nucl Eng Idaho State University   (United States)

Author keywords

Cytotoxic edema; Degeneration; Epilepsy and neuromyotonia; Familial hemiplegic migraine; Hyperkalemic and hypokalemic periodic paralysis; Long QT syndrome; Myotonia and paramyotonia; Sodium overload

Indexed keywords

ACETAZOLAMIDE; CALCIUM CHANNEL CAV1.1; CALCIUM CHANNEL CAV2.1; POTASSIUM CHANNEL KCNQ1; POTASSIUM CHANNEL KCNQ2; SODIUM CHANNEL NAV1.1; SODIUM CHANNEL NAV1.4; SODIUM CHANNEL NAV1.5; UNCLASSIFIED DRUG; VOLTAGE GATED CALCIUM CHANNEL;

ARTICLE; BRAIN EDEMA; CACNA1A GENE; CACNA1S GENE; CALCIUM CHANNELOPATHY; CELL MEMBRANE DEPOLARIZATION; CELL MEMBRANE POTENTIAL; CHANNELOPATHY; FAMILIAL HEMIPLEGIC MIGRAINE; FEBRILE CONVULSION; GENE; GENE MUTATION; GENERALIZED EPILEPSY; HUMAN; HYPERNATREMIA; HYPERPOLARIZATION; HYPOKALEMIC PERIODIC PARALYSIS; ION CURRENT; KCNH2 GENE; KCNQ1 GENE; KCNQ2 GENE; MYOKYMIA; NEUROTOXICITY; NONHUMAN; OMEGA CURRENT; PATHOPHYSIOLOGY; POTASSIUM CHANNELOPATHY; QT PROLONGATION; REPOLARIZATION; SCN1A GENE; SCN4A GENE; SCN5A GENE; SEVERE MYOCLONIC EPILEPSY IN INFANCY; SODIUM CHANNELOPATHY; SODIUM CURRENT; THOMSEN DISEASE; TISSUE DEGENERATION;

EID: 84866179225     PISSN: None     EISSN: 16639812     Source Type: Journal    
DOI: 10.3389/fphar.2012.00112     Document Type: Article

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