Sevoflurane preconditioning improves mitochondrial function and long-term neurologic sequelae after transient cerebral ischemia: Role of mitochondrial permeability transition

Critical Care Medicine

Volumn 40, Issue 9, 2012, Pages 2685-2693

  Ye, Ruidong ^a,c   Yang, Qianzi ^a   Kong, Xiangwei ^b   Li, Nanlin ^a   Zhang, Yunxia ^a   Han, Junliang ^a   Xiong, Lize ^a   Liu, Xinfeng ^c   Zhao, Gang ^a  

^a FOURTH MILITARY MEDICAL UNIVERSITY   (China)

^b FOURTH MILITARY MEDICAL UNIVERSITY   (China)

^c JINLING HOSPITAL   (China)

Author keywords

ischemia; middle cerebral artery occlusion; mitocho ndria; mitochondrial permeability transition pore; neuroprotection; sevoflurane preconditioning

Indexed keywords

ATRACTYLOSIDE; CALCIUM; CYCLOSPORIN A; HYDROGEN PEROXIDE; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; SEVOFLURANE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BRAIN FUNCTION; BRAIN INFARCTION SIZE; CONTROLLED STUDY; DISEASE ASSOCIATION; DISEASE SEVERITY; IN VITRO STUDY; ISCHEMIC PRECONDITIONING; LABORATORY TEST; MALE; MIDDLE CEREBRAL ARTERY OCCLUSION; MITOCHONDRIAL PERMEABILITY; MITOCHONDRION; NEUROLOGIC DISEASE; NONHUMAN; PRIORITY JOURNAL; RAT; SPRAGUE DAWLEY RAT; TRANSIENT ISCHEMIC ATTACK;

ANIMALS; BRAIN; DISEASE MODELS, ANIMAL; ISCHEMIC ATTACK, TRANSIENT; ISCHEMIC PRECONDITIONING; METHYL ETHERS; MITOCHONDRIA; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; NEUROPROTECTIVE AGENTS; RANDOM ALLOCATION; RATS; RATS, SPRAGUE-DAWLEY; REFERENCE VALUES; REPERFUSION INJURY; SENSITIVITY AND SPECIFICITY; STATISTICS, NONPARAMETRIC;

EID: 84865526121     PISSN: 00903493     EISSN: 15300293     Source Type: Journal    
DOI: 10.1097/CCM.0b013e318258fb90     Document Type: Article

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