The delayed post-injury administration of soluble fas receptor attenuates post-traumatic neural degeneration and enhances functional recovery after traumatic cervical spinal cord injury

Journal of Neurotrauma

Volumn 29, Issue 8, 2012, Pages 1586-1599

  Robins Steele, Sherri ^a,b   Nguyen, Dung Hoang ^c,d   Fehlings, Michael G ^e  

^a UNIVERSITY OF TORONTO   (Canada)

^b SHERIDAN COLLEGE   (Canada)

^c UNIVERSITY HEALTH NETWORK   (Canada)

^d UNIVERSITY OF MANITOBA   (Canada)

^e TORONTO WESTERN HOSPITAL   (Canada)

Author keywords

apoptosis; Fas; neuroprotection; soluble Fas receptor; spinal cord injury

Indexed keywords

FAS ANTIGEN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL SURVIVAL; CERVICAL SPINAL CORD INJURY; COGNITION; COMPARATIVE STUDY; CONTROLLED STUDY; CONTUSION; FEMALE; LONG TERM CARE; NERVE CELL; NERVE DEGENERATION; NEUROPATHIC PAIN; NONHUMAN; OLIGODENDROGLIA; RAT; TREATMENT OUTCOME;

ANIMALS; ANTIGENS, CD95; APOPTOSIS; BEHAVIOR, ANIMAL; FEMALE; NERVE DEGENERATION; NEURALGIA; OLIGODENDROGLIA; PAIN MEASUREMENT; RATS; RATS, WISTAR; RECOVERY OF FUNCTION; SPINAL CORD INJURIES; TIME FACTORS;

EID: 84861991418     PISSN: 08977151     EISSN: 15579042     Source Type: Journal    
DOI: 10.1089/neu.2011.2005     Document Type: Article

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