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Volumn 103, Issue 1, 2001, Pages 203-218
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Oligodendroglial apoptosis occurs along degenerating axons and is associated with FAS and p75 expression following spinal cord injury in the rat
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Author keywords
Caspases; Death receptors; Glia; Neurotrauma; Programmed cell death; amyloid precursor
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Indexed keywords
CASPASE 3;
CASPASE 8;
CASPASE INHIBITOR;
DNA;
DNA NUCLEOTIDYLEXOTRANSFERASE;
FAS ANTIGEN;
PROTEIN P75;
ANALYTIC METHOD;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
APOPTOSIS;
ARTICLE;
ASTROCYTE;
CELL SPECIFICITY;
CELLULAR DISTRIBUTION;
CONTROLLED STUDY;
ENZYME ACTIVATION;
FEMALE;
GLIA CELL;
GRAY MATTER;
IMMUNOHISTOCHEMISTRY;
MICROGLIA;
NERVE FIBER DEGENERATION;
NICK END LABELING;
NONHUMAN;
OLIGODENDROGLIA;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
RAT;
SPINAL CORD INJURY;
WHITE MATTER;
ANIMALS;
ANTIGENS, CD95;
APOPTOSIS;
AXONS;
BLOTTING, WESTERN;
ELECTROPHORESIS, AGAR GEL;
FEMALE;
IMMUNOHISTOCHEMISTRY;
IN SITU NICK-END LABELING;
MICROSCOPY, ELECTRON;
NERVE DEGENERATION;
OLIGODENDROGLIA;
RATS;
RATS, WISTAR;
RECEPTOR, NERVE GROWTH FACTOR;
RECEPTORS, NERVE GROWTH FACTOR;
SPINAL CORD;
SPINAL CORD INJURIES;
TIME FACTORS;
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EID: 0035961212
PISSN: 03064522
EISSN: None
Source Type: Journal
DOI: 10.1016/S0306-4522(00)00538-8 Document Type: Article |
Times cited : (349)
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References (82)
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