Epigenetic alterations may regulate temporary reversal of cd4+ T cell activation caused by trichloroethylene exposure

Toxicological Sciences

Volumn 127, Issue 1, 2012, Pages 169-178

  Gilbert, Kathleen M ^a   Nelson, Ashley R ^a   Cooney, Craig A ^b   Reisfeld, Brad ^c   Blossom, Sarah J ^a  

^a UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES   (United States)

^b CENTRAL ARKANSAS VETERANS HEALTHCARE SYSTEM   (United States)

^c Veterinary Dentistry and Oral Surgery   (United States)

Author keywords

Epigenetics; Immunotoxicity; Trichloroethylene

Indexed keywords

CYCLIN DEPENDENT KINASE INHIBITOR 1A; DNA METHYLTRANSFERASE 1; DRINKING WATER; GAMMA INTERFERON; HERMES ANTIGEN; INTERLEUKIN 2; L SELECTIN; TRICHLOROETHYLENE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CD4+ T LYMPHOCYTE; CELL FUNCTION; CONTROLLED STUDY; DNA METHYLATION; ENDOGENOUS RETROVIRUS; ENVIRONMENTAL EXPOSURE; EPIGENETICS; FEMALE; GENE EXPRESSION; GENE MUTATION; IMMUNE DEFICIENCY; INTERFERON PRODUCTION; MOUSE; NONHUMAN; ONCOGENE; PHENOTYPE; RECEPTOR DOWN REGULATION; REGULATORY T LYMPHOCYTE; RETROPOSON; T LYMPHOCYTE ACTIVATION; TH17 CELL; TH2 CELL;

EID: 84860170571     PISSN: 10966080     EISSN: 10960929     Source Type: Journal    
DOI: 10.1093/toxsci/kfs093     Document Type: Article

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