-
1
-
-
19944429270
-
Distinct gene expression profiles in norepinephrine- and epinephrine-producing hereditary and sporadic pheochromocytomas: Activation of hypoxia-driven angiogenic pathways in von Hippel-Lindau syndrome
-
DOI 10.1677/erc.1.00838
-
Eisenhofer, G., et al., Distinct gene expression profiles in norepinephrine- and epinephrine-producing hereditary and sporadic pheochromocytomas: activation of hypoxia-driven angiogenic pathways in von Hippel-Lindau syndrome. Endocr Relat Cancer, 2004. 11(4): p. 897-911. (Pubitemid 40065561)
-
(2004)
Endocrine-Related Cancer
, vol.11
, Issue.4
, pp. 897-911
-
-
Eisenhofer, G.1
Huynh, T.-T.2
Pacak, K.3
Brouwers, F.M.4
Walther, M.M.5
Linehan, W.M.6
Munson, P.J.7
Mannelli, M.8
Goldstein, D.S.9
Elkahloun, A.G.10
-
2
-
-
27544468721
-
Novel pheochromocytoma susceptibility loci identified by integrative genomics
-
DOI 10.1158/0008-5472.CAN-05-1427
-
Dahia, P.L., et al., Novel pheochromocytoma susceptibility loci identified by integrative genomics. Cancer Res, 2005. 65(21): p. 9651-8. (Pubitemid 41541440)
-
(2005)
Cancer Research
, vol.65
, Issue.21
, pp. 9651-9658
-
-
Dahia, P.L.M.1
Hao, K.2
Rogus, J.3
Colin, C.4
Pujana, M.A.G.5
Ross, K.6
Magoffin, D.7
Aronin, N.8
Cascon, A.9
Hayashida, C.Y.10
Li, C.11
Toledo, S.P.A.12
Stiles, C.D.13
-
3
-
-
33845488766
-
Transcriptional regulation of phenylethanolamine N-methyltransferase in pheochromocytomas from patients with von Hippel-Lindau syndrome and multiple endocrine neoplasia type 2
-
Huynh, T.T., et al., Transcriptional regulation of phenylethanolamine N-methyltransferase in pheochromocytomas from patients with von Hippel-Lindau syndrome and multiple endocrine neoplasia type 2. Ann N Y Acad Sci, 2006. 1073: p. 241-52.
-
(2006)
Ann N Y Acad Sci
, vol.1073
, pp. 241-252
-
-
Huynh, T.T.1
-
4
-
-
80053139912
-
Integrative genomic analysis reveals somatic mutations in pheochromocytoma and paraganglioma
-
Burnichon, N., et al., Integrative genomic analysis reveals somatic mutations in pheochromocytoma and paraganglioma. Hum Mol Genet, 2011. 20(20): p. 3974-85.
-
(2011)
Hum Mol Genet
, vol.20
, Issue.20
, pp. 3974-3985
-
-
Burnichon, N.1
-
5
-
-
78649404935
-
Research resource: Transcriptional profiling reveals different pseudohypoxic signatures in SDHB and VHL-related pheochromocytomas
-
Lopez-Jimenez, E., et al., Research resource: Transcriptional profiling reveals different pseudohypoxic signatures in SDHB and VHL-related pheochromocytomas. Mol Endocrinol, 2010. 24(12): p. 2382-91.
-
(2010)
Mol Endocrinol
, vol.24
, Issue.12
, pp. 2382-2391
-
-
Lopez-Jimenez, E.1
-
6
-
-
77956357888
-
Microarray analysis reveals differential expression of benign and malignant pheochromocytoma
-
Waldmann, J., et al., Microarray analysis reveals differential expression of benign and malignant pheochromocytoma. Endocr Relat Cancer, 2010. 17(3): p. 743-56.
-
(2010)
Endocr Relat Cancer
, vol.17
, Issue.3
, pp. 743-756
-
-
Waldmann, J.1
-
7
-
-
33845487372
-
Gene expression profiling of benign and malignant pheochromocytoma
-
DOI 10.1196/annals.1353.058, Pheochromocytoma: First International Symposium
-
Brouwers, F.M., et al., Gene expression profiling of benign and malignant pheochromocytoma. Ann N YAcad Sci, 2006. 1073: p. 541-56. (Pubitemid 44912049)
-
(2006)
Annals of the New York Academy of Sciences
, vol.1073
, pp. 541-556
-
-
Brouwers, F.M.1
Elkahloun, A.G.2
Munson, P.J.3
Eisenhofer, G.4
Barb, J.5
Linehan, W.M.6
Lenders, J.W.M.7
De Krijger, R.8
Mannelli, M.9
Udelsman, R.10
Ocal, I.T.11
Shulkin, B.L.12
Bornstein, S.R.13
Breza, J.14
Ksinantova, L.15
Pacak, K.16
-
8
-
-
36849030813
-
Identification of potential gene markers and insights into the pathophysiology of pheochromocytoma malignancy
-
DOI 10.1210/jc.2007-1253
-
Thouennon, E., et al., Identification of potential gene markers and insights into the pathophysiology of pheochromocytoma malignancy. J Clin Endocrinol Metab, 2007. 92(12): p. 4865-72. (Pubitemid 350223470)
-
(2007)
Journal of Clinical Endocrinology and Metabolism
, vol.92
, Issue.12
, pp. 4865-4872
-
-
Thouennon, E.1
Elkahloun, A.G.2
Guillemot, J.3
Gimenez-Roqueplo, A.-P.4
Bertherat, J.5
Pierre, A.6
Ghzili, H.7
Grumolato, L.8
Muresan, M.9
Klein, M.10
Lefebvre, H.11
Ouafik, L.12
Vaudry, H.13
Plouin, P.-F.14
Yon, L.15
Anouar, Y.16
-
9
-
-
77955594623
-
A HIF1alpha regulatory loop links hypoxia and mitochondrial signals in pheochromocytomas
-
Dahia, P.L., et al., A HIF1alpha regulatory loop links hypoxia and mitochondrial signals in pheochromocytomas. PLoS Genet, 2005. 1(1): p. 72-80.
-
(2005)
PLoS Genet
, vol.1
, Issue.1
, pp. 72-80
-
-
Dahia, P.L.1
-
10
-
-
33644551073
-
Gene expression programs in response to hypoxia: Cell type specificity and prognostic significance in human cancers
-
Chi, J.T., et al., Gene expression programs in response to hypoxia: cell type specificity and prognostic significance in human cancers. PLoS Med, 2006. 3(3): p. e47.
-
(2006)
PLoS Med
, vol.3
, Issue.3
-
-
Chi, J.T.1
-
11
-
-
0142166332
-
Targeting HIF-1 for cancer therapy
-
Semenza, G.L., Targeting HIF-1 for cancer therapy. Nat Rev Cancer, 2003. 3(10): p. 721-32. (Pubitemid 37328811)
-
(2003)
Nature Reviews Cancer
, vol.3
, Issue.10
, pp. 721-732
-
-
Semenza, G.L.1
-
12
-
-
0031020884
-
Endothelial PAS domain protein 1 (EPAS1), a transcription factor selectively expressed in endothelial cells
-
Tian, H., S.L. McKnight, and D.W. Russell, Endothelial PAS domain protein 1 (EPAS1), a transcription factor selectively expressed in endothelial cells. Genes Dev, 1997. 11(1): p. 72-82. (Pubitemid 27043825)
-
(1997)
Genes and Development
, vol.11
, Issue.1
, pp. 72-82
-
-
Tian, H.1
McKnight, S.L.2
Russell, D.W.3
-
13
-
-
0029051439
-
Hypoxia-inducible factor 1 is a basic-helixloop-helix-PAS heterodimer regulated by cellular O2 tension
-
Wang, G.L., et al., Hypoxia-inducible factor 1 is a basic-helixloop- helix-PAS heterodimer regulated by cellular O2 tension. Proc Natl Acad Sci U S A, 1995. 92(12): p. 5510-4.
-
(1995)
Proc Natl Acad Sci U S A
, vol.92
, Issue.12
, pp. 5510-5514
-
-
Wang, G.L.1
-
14
-
-
0035834409
-
A conserved family of prolyl-4-hydroxylases that modify HIF
-
DOI 10.1126/science.1066373
-
Bruick, R.K. and S.L. McKnight, A conserved family of prolyl-4-hydroxylases that modify HIF. Science, 2001. 294(5545): p. 1337-40. (Pubitemid 33063099)
-
(2001)
Science
, vol.294
, Issue.5545
, pp. 1337-1340
-
-
Bruick, R.K.1
McKnight, S.L.2
-
15
-
-
17944375360
-
C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation
-
DOI 10.1016/S0092-8674(01)00507-4
-
Epstein,A.C., et al., C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation. Cell, 2001. 107(1): p. 43-54 (Pubitemid 32972036)
-
(2001)
Cell
, vol.107
, Issue.1
, pp. 43-54
-
-
Epstein, A.C.R.1
Gleadle, J.M.2
McNeill, L.A.3
Hewitson, K.S.4
O'Rourke, J.5
Mole, D.R.6
Mukherji, M.7
Metzen, E.8
Wilson, M.I.9
Dhanda, A.10
Tian, Y.-M.11
Masson, N.12
Hamilton, D.L.13
Jaakkola, P.14
Barstead, R.15
Hodgkin, J.16
Maxwell, P.H.17
Pugh, C.W.18
Schofield, C.J.19
Ratcliffe, P.J.20
more..
-
16
-
-
0038205886
-
The role of iron and 2-oxoglutarate oxygenases in signalling
-
DOI 10.1042/BST0310510
-
Hewitson, K.S., et al., The role of iron and 2-oxoglutarate oxygenases in signalling. Biochem Soc Trans, 2003. 31(Pt 3): p. 510-5. (Pubitemid 36752676)
-
(2003)
Biochemical Society Transactions
, vol.31
, Issue.3
, pp. 510-515
-
-
Hewitson, K.S.1
McNeill, L.A.2
Elkins, J.M.3
Schofield, C.J.4
-
17
-
-
70349472696
-
The Warburg effect is genetically determined in inherited pheochromocytomas
-
Favier, J., et al., The Warburg effect is genetically determined in inherited pheochromocytomas. PLoS One, 2009. 4(9): p. e7094.
-
(2009)
PLoS One
, vol.4
, Issue.9
-
-
Favier, J.1
-
18
-
-
28544446058
-
Mitochondrial tumour suppressors: A genetic and biochemical update
-
DOI 10.1038/nrc1737
-
Gottlieb, E. and I.P. Tomlinson, Mitochondrial tumour suppressors: a genetic and biochemical update. Nat Rev Cancer, 2005. 5 (11): p. 857-66. (Pubitemid 41746030)
-
(2005)
Nature Reviews Cancer
, vol.5
, Issue.11
, pp. 857-866
-
-
Gottlieb, E.1
Tomlinson, I.P.M.2
-
19
-
-
0034964421
-
Gene mutations in the succinate dehydrogenase subunit SDHB cause susceptibility to familial pheochromocytoma and to familial paraganglioma
-
DOI 10.1086/321282
-
Astuti, D., et al., Gene mutations in the succinate dehydrogenase subunit SDHB cause susceptibility to familial pheochromocytoma and to familial paraganglioma. Am J Hum Genet, 2001. 69(1): p. 49-54. (Pubitemid 32614017)
-
(2001)
American Journal of Human Genetics
, vol.69
, Issue.1
, pp. 49-54
-
-
Astuti, D.1
Latif, F.2
Dallol, A.3
Dahia, P.L.M.4
Douglas, F.5
George, E.6
Skoldberg, F.7
Husebye, E.S.8
Eng, C.9
Maher, E.R.10
-
20
-
-
77950342008
-
SDHAF2 mutations in familial and sporadic paraganglioma and phaeochromocytoma
-
Bayley, J.P., et al., SDHAF2 mutations in familial and sporadic paraganglioma and phaeochromocytoma. Lancet Oncol, 2010. 11 (4): p. 366-72.
-
(2010)
Lancet Oncol
, vol.11
, Issue.4
, pp. 366-372
-
-
Bayley, J.P.1
-
21
-
-
0034602950
-
Mutations in SDHD, a mitochondrial complex II gene, in hereditary paraganglioma
-
DOI 10.1126/science.287.5454.848
-
Baysal, B.E., et al., Mutations in SDHD, a mitochondrial complex II gene, in hereditary paraganglioma. Science, 2000. 287(5454): p. 848-51. (Pubitemid 30084326)
-
(2000)
Science
, vol.287
, Issue.5454
, pp. 848-851
-
-
Baysal, B.E.1
Ferrell, R.E.2
Willett-Brozick, J.E.3
Lawrence, E.C.4
Myssiorek, D.5
Bosch, A.6
Van Der, M.A.7
Taschner, P.E.M.8
Rubinstein, W.S.9
Myers, E.N.10
Richard III, C.W.11
Cornelisse, C.J.12
Devilee, P.13
Devlin, B.14
-
22
-
-
77958164441
-
SDHA is a tumor suppressor gene causing paraganglioma
-
Burnichon, N., et al., SDHA is a tumor suppressor gene causing paraganglioma. Hum Mol Genet, 2010. 19(15): p. 3011-20.
-
(2010)
Hum Mol Genet
, vol.19
, Issue.15
, pp. 3011-3020
-
-
Burnichon, N.1
-
23
-
-
69549088424
-
SDH5, a gene required for flavination of succinate dehydrogenase, is mutated in paraganglioma
-
Hao, H.X., et al., SDH5, a gene required for flavination of succinate dehydrogenase, is mutated in paraganglioma. Science, 2009. 325(5944): p. 1139-42.
-
(2009)
Science
, vol.325
, Issue.5944
, pp. 1139-1142
-
-
Hao, H.X.1
-
24
-
-
0033767445
-
Mutations in SDHC cause autosomal dominant paraganglioma, type 3
-
Niemann, S. and U. Muller, Mutations in SDHC cause autosomal dominant paraganglioma, type 3. Nat Genet, 2000. 26(3): p. 268-70.
-
(2000)
Nat Genet
, vol.26
, Issue.3
, pp. 268-270
-
-
Niemann, S.1
Muller, U.2
-
25
-
-
19944433653
-
Succinate links TCA cycle dysfunction to oncogenesis by inhibiting HIF-alpha prolyl hydroxylase
-
DOI 10.1016/j.ccr.2004.11.022, PII S153561080400368X
-
Selak, M.A., et al., Succinate links TCA cycle dysfunction to oncogenesis by inhibiting HIF-alpha prolyl hydroxylase. Cancer Cell, 2005. 7(1): p. 77-85. (Pubitemid 40126384)
-
(2005)
Cancer Cell
, vol.7
, Issue.1
, pp. 77-85
-
-
Selak, M.A.1
Armour, S.M.2
MacKenzie, E.D.3
Boulahbel, H.4
Watson, D.G.5
Mansfield, K.D.6
Pan, Y.7
Simon, M.C.8
Thompson, C.B.9
Gottlieb, E.10
-
26
-
-
37849022071
-
Loss of the SdhB, but Not the SdhA, subunit of complex II triggers reactive oxygen species-dependent hypoxiainducible factor activation and tumorigenesis
-
Guzy, R.D., et al., Loss of the SdhB, but Not the SdhA, subunit of complex II triggers reactive oxygen species-dependent hypoxiainducible factor activation and tumorigenesis. Mol Cell Biol, 2008. 28(2): p. 718-31.
-
(2008)
Mol Cell Biol
, vol.28
, Issue.2
, pp. 718-731
-
-
Guzy, R.D.1
-
27
-
-
0037097861
-
FIH-1 is an asparaginyl hydroxylase enzyme that regulates the transcriptional activity of hypoxia-inducible factor
-
DOI 10.1101/gad.991402
-
Lando, D., et al., FIH-1 is an asparaginyl hydroxylase enzyme that regulates the transcriptional activity of hypoxia-inducible factor. Genes Dev, 2002. 16(12): p. 1466-71. (Pubitemid 34686328)
-
(2002)
Genes and Development
, vol.16
, Issue.12
, pp. 1466-1471
-
-
Lando, D.1
Peet, D.J.2
Gorman, J.J.3
Whelan, D.A.4
Whitelaw, M.L.5
Bruick, R.K.6
-
28
-
-
0035887011
-
FIH-1: A novel protein that interacts with HIF-1alpha and VHL to mediate repression of HIF-1 transcriptional activity
-
DOI 10.1101/gad.924501
-
Mahon, P.C., K. Hirota, and G.L. Semenza, FIH-1: a novel protein that interacts with HIF-1alpha and VHL to mediate repression of HIF-1 transcriptional activity. Genes Dev, 2001. 15(20): p. 2675-86. (Pubitemid 32988878)
-
(2001)
Genes and Development
, vol.15
, Issue.20
, pp. 2675-2686
-
-
Mahon, P.C.1
Hirota, K.2
Semenza, G.L.3
-
29
-
-
40949132841
-
Turn me on: Regulating HIF transcriptional activity
-
DOI 10.1038/sj.cdd.4402315, PII 4402315, The biology of Hypoxia-inducible factors
-
Lisy, K. and D.J. Peet, Turn me on: regulating HIF transcriptional activity. Cell Death Differ, 2008. 15(4): p. 642-9. (Pubitemid 351405067)
-
(2008)
Cell Death and Differentiation
, vol.15
, Issue.4
, pp. 642-649
-
-
Lisy, K.1
Peet, D.J.2
-
30
-
-
70450239624
-
Inhibition of succinate dehydrogenase dysregulates histone modification in mammalian cells
-
Cervera, A.M., et al., Inhibition of succinate dehydrogenase dysregulates histone modification in mammalian cells. Mol Cancer, 2009. 8: p. 89.
-
(2009)
Mol Cancer
, vol.8
, pp. 89
-
-
Cervera, A.M.1
-
31
-
-
44149105533
-
The emerging functions of histone demethylases
-
DOI 10.1016/j.gde.2007.12.003, PII S0959437X07002158
-
Agger, K., et al., The emerging functions of histone demethylases. Curr Opin Genet Dev, 2008. 18(2): p. 159-68. (Pubitemid 351749815)
-
(2008)
Current Opinion in Genetics and Development
, vol.18
, Issue.2
, pp. 159-168
-
-
Agger, K.1
Christensen, J.2
Cloos, P.A.3
Helin, K.4
-
32
-
-
84863718281
-
Loss of the candidate tumor suppressor BTG3 triggers acute cellular senescence via the ERK-JMJD3-p16 (INK4a) signaling axis
-
Lin, T.Y., et al., Loss of the candidate tumor suppressor BTG3 triggers acute cellular senescence via the ERK-JMJD3-p16 (INK4a) signaling axis. Oncogene, 2011.
-
(2011)
Oncogene
-
-
Lin, T.Y.1
-
33
-
-
79953666919
-
p53 interaction with JMJD3 results in its nuclear distribution during mouse neural stem cell differentiation
-
Sola, S., et al., p53 interaction with JMJD3 results in its nuclear distribution during mouse neural stem cell differentiation. PLoS One, 2011. 6(3): p. e18421.
-
(2011)
PLoS One
, vol.6
, Issue.3
-
-
Sola, S.1
-
34
-
-
77649175595
-
Germline mutations in TMEM127 confer susceptibility to pheochromocytoma
-
Qin, Y., et al., Germline mutations in TMEM127 confer susceptibility to pheochromocytoma. Nat Genet, 2010. 42(3): p. 229-33.
-
(2010)
Nat Genet
, vol.42
, Issue.3
, pp. 229-233
-
-
Qin, Y.1
-
35
-
-
0034603172
-
Transforming ability of MEN2A-RET requires activation of the phosphatidylinositol 3-kinase/AKT signaling pathway
-
DOI 10.1074/jbc.275.5.3568
-
Segouffin-Cariou, C. and M. Billaud, Transforming ability of MEN2A-RET requires activation of the phosphatidylinositol 3-kinase/AKT signaling pathway. J Biol Chem, 2000. 275(5): p. 3568-76. (Pubitemid 30083066)
-
(2000)
Journal of Biological Chemistry
, vol.275
, Issue.5
, pp. 3568-3576
-
-
Segouffin-Cariou, C.1
Billaud, M.2
-
36
-
-
20844435467
-
The NF1 tumor suppressor critically regulates TSC2 and mTOR
-
DOI 10.1073/pnas.0503224102
-
Johannessen, C.M., et al., The NF1 tumor suppressor critically regulates TSC2 and mTOR. Proc Natl Acad Sci U S A, 2005. 102 (24): p. 8573-8. (Pubitemid 40862777)
-
(2005)
Proceedings of the National Academy of Sciences of the United States of America
, vol.102
, Issue.24
, pp. 8573-8578
-
-
Johannessen, C.M.1
Reczek, E.E.2
James, M.F.3
Brems, H.4
Legius, E.5
Cichowski, K.6
-
37
-
-
78650200503
-
Spectrum and prevalence of FP/TMEM127 gene mutations in pheochromocytomas and paragangliomas
-
Yao, L., et al., Spectrum and prevalence of FP/TMEM127 gene mutations in pheochromocytomas and paragangliomas. JAMA, 2010. 304(23): p. 2611-9.
-
(2010)
JAMA
, vol.304
, Issue.23
, pp. 2611-2619
-
-
Yao, L.1
-
38
-
-
44349134920
-
Activation of PI3K/Akt and MAPK pathways regulates Myc-mediated transcription by phosphorylating and promoting the degradation of Mad1
-
DOI 10.1073/pnas.0802785105
-
Zhu, J., J. Blenis, and J. Yuan, Activation of PI3K/Akt and MAPK pathways regulates Myc-mediated transcription by phosphorylating and promoting the degradation of Mad1. Proc Natl Acad Sci U S A, 2008. 105(18): p. 6584-9. (Pubitemid 351754550)
-
(2008)
Proceedings of the National Academy of Sciences of the United States of America
, vol.105
, Issue.18
, pp. 6584-6589
-
-
Zhu, J.1
Blenis, J.2
Yuan, J.3
-
39
-
-
77949347378
-
Regulation of gene expression in hepatic cells by the mammalian Target of Rapamycin (mTOR)
-
Jimenez, R.H., et al., Regulation of gene expression in hepatic cells by the mammalian Target of Rapamycin (mTOR). PLoS One, 2010. 5(2): p. e9084.
-
(2010)
PLoS One
, vol.5
, Issue.2
-
-
Jimenez, R.H.1
-
40
-
-
0033693018
-
Duplication of the mutant RET allele in trisomy 10 or loss of the wild-type allele in multiple endocrine neoplasia type 2-associated pheochromocytomas
-
Huang, S.C., et al., Duplication of the mutant RET allele in trisomy 10 or loss of the wild-type allele in multiple endocrine neoplasia type 2-associated pheochromocytomas. Cancer Res, 2000. 60(22): p. 6223-6.
-
(2000)
Cancer Res
, vol.60
, Issue.22
, pp. 6223-6226
-
-
Huang, S.C.1
-
41
-
-
0037242089
-
Amplification and overexpression of mutant RET in multiple endocrine neoplasia type 2-associated medullary thyroid carcinoma
-
DOI 10.1210/jc.2002-021254
-
Huang, S.C., et al., Amplification and overexpression of mutant RET in multiple endocrine neoplasia type 2-associated medullary thyroid carcinoma. J Clin Endocrinol Metab, 2003. 88(1): p. 459-63. (Pubitemid 36115202)
-
(2003)
Journal of Clinical Endocrinology and Metabolism
, vol.88
, Issue.1
, pp. 459-463
-
-
Huang, S.C.1
Torres-Cruz, J.2
Pack, S.D.3
Koch, C.A.4
Vortmeyer, A.O.5
Mannan, P.6
Lubensky, I.A.7
Gagel, R.F.8
Zhuang, Z.9
-
42
-
-
79959752614
-
Exome sequencing identifies MAX mutations as a cause of hereditary pheochromocytoma
-
Comino-Mendez, I., et al., Exome sequencing identifies MAX mutations as a cause of hereditary pheochromocytoma. Nat Genet, 2011. 43(7): p. 663-7.
-
(2011)
Nat Genet
, vol.43
, Issue.7
, pp. 663-667
-
-
Comino-Mendez, I.1
-
43
-
-
77950193818
-
Maternally expressed gene 3, an imprinted noncoding RNA gene, is associated with meningioma pathogenesis and progression
-
Zhang, X., et al., Maternally expressed gene 3, an imprinted noncoding RNA gene, is associated with meningioma pathogenesis and progression. Cancer Res, 2010. 70(6): p. 2350-8.
-
(2010)
Cancer Res
, vol.70
, Issue.6
, pp. 2350-2358
-
-
Zhang, X.1
-
44
-
-
18944384938
-
Epigenetic alteration at the DLK1-GTL2 imprinted domain in human neoplasia: Analysis of neuroblastoma, phaeochromocytoma and Wilms' tumour
-
DOI 10.1038/sj.bjc.6602478
-
Astuti, D., et al., Epigenetic alteration at the DLK1-GTL2 imprinted domain in human neoplasia: analysis of neuroblastoma, phaeochromocytoma and Wilms' tumour. Br J Cancer, 2005. 92(8): p. 1574-80. (Pubitemid 40705027)
-
(2005)
British Journal of Cancer
, vol.92
, Issue.8
, pp. 1574-1580
-
-
Astuti, D.1
Latif, F.2
Wagner, K.3
Gentle, D.4
Cooper, W.N.5
Catchpoole, D.6
Grundy, R.7
Ferguson-Smith, A.C.8
Maher, E.R.9
-
45
-
-
11244259404
-
Inactivation of imprinted genes induced by cellular stress and tumorigenesis
-
Pantoja, C., et al., Inactivation of imprinted genes induced by cellular stress and tumorigenesis. Cancer Res, 2005. 65(1): p. 26-33. (Pubitemid 40070792)
-
(2005)
Cancer Research
, vol.65
, Issue.1
, pp. 26-33
-
-
Pantoja, C.1
De Los, R.L.2
Matheu, A.3
Antequera, F.4
Serrano, M.5
-
46
-
-
0345172377
-
A Pituitary-Derived MEG3 Isoform Functions as a Growth Suppressor in Tumor Cells
-
DOI 10.1210/jc.2003-030222
-
Zhang, X., et al., A pituitary-derived MEG3 isoform functions as a growth suppressor in tumor cells. J Clin Endocrinol Metab, 2003. 88(11): p. 5119-26. (Pubitemid 37452705)
-
(2003)
Journal of Clinical Endocrinology and Metabolism
, vol.88
, Issue.11
, pp. 5119-5126
-
-
Zhang, X.1
Zhou, Y.2
Mehta, K.R.3
Danila, D.C.4
Scolavino, S.5
Johnson, S.R.6
Klibanski, A.7
-
47
-
-
17844397712
-
Hypermethylation of the promoter region is associated with the loss of MEG3 gene expression in human pituitary tumors
-
DOI 10.1210/jc.2004-1848
-
Zhao, J., et al., Hypermethylation of the promoter region is associated with the loss of MEG3 gene expression in human pituitary tumors. J Clin Endocrinol Metab, 2005. 90(4): p. 2179-86. (Pubitemid 40586259)
-
(2005)
Journal of Clinical Endocrinology and Metabolism
, vol.90
, Issue.4
, pp. 2179-2186
-
-
Zhao, J.1
Dahle, D.2
Zhou, Y.3
Zhang, X.4
Klibanski, A.5
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