Phosphorylation of HtrA2 by cyclin-dependent kinase-5 is important for mitochondrial function

Cell Death and Differentiation

Volumn 19, Issue 2, 2012, Pages 257-266

  Fitzgerald, J C ^a   Camprubi, M D ^a   Dunn, L ^a   Wu, H C ^a   Ip, N Y ^b   Kruger, R ^c   Martins, L M ^d   Wood, N W ^a   Plun Favreau, H ^a  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

^b HONG KONG UNIVERSITY OF SCIENCE AND TECHNOLOGY   (Hong Kong)

^c GERMAN CENTER FOR NEURODEGENERATIVE DISEASES DZNE   (Germany)

^d MRC TOXICOLOGY UNIT   (United Kingdom)

Author keywords

Cdk5; HtrA2; mitochondria; Parkinson's disease; phosphorylation

Indexed keywords

CYCLIN DEPENDENT KINASE 5; SERINE PROTEINASE OMI;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; BRAIN; CELL PROTECTION; CELLULAR STRESS RESPONSE; CONTROLLED STUDY; ENZYME PHOSPHORYLATION; HUMAN; HUMAN CELL; HUMAN TISSUE; INTRACELLULAR SIGNALING; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRION; MOUSE; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION;

ANIMALS; CYCLIN-DEPENDENT KINASE 5; CYTOSOL; HUMANS; MICE; MITOCHONDRIA; MITOCHONDRIAL PROTEINS; PHOSPHORYLATION; PHOSPHOSERINE; PROTEIN BINDING; PROTEIN TRANSPORT; SERINE ENDOPEPTIDASES;

MUS;

EID: 84855664067     PISSN: 13509047     EISSN: 14765403     Source Type: Journal    
DOI: 10.1038/cdd.2011.90     Document Type: Article

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