Cognitive impairment and increased Aβ levels induced by paraquat exposure are attenuated by enhanced removal of mitochondrial H2O2

Neurobiology of Aging

Volumn 33, Issue 2, 2012, Pages 432.e15-432.e26

  Chen, Liuji ^a,b   Yoo, Si Eun ^b   Na, Ren ^b   Liu, Yuhong ^a,b   Ran, Qitao ^a,b,c  

^a Mays Cancer Center at UT Health San Antonio   (United States)

^b UNIVERSITY OF TEXAS HEALTH SCIENCE CENTER AT SAN ANTONIO   (United States)

^c SOUTH TEXAS VETERANS HEALTH CARE SYSTEM   (United States)

Author keywords

Aging; Alzheimer's disease; Beta amyloid; Mitochondria; Oxidative stress; Peroxiredoxin 3; Pesticide exposure

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; HYDROGEN PEROXIDE; PARAQUAT; PEROXIREDOXIN 3;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; BRAIN LEVEL; BRAIN MITOCHONDRION; CELL DAMAGE; COGNITIVE DEFECT; CONTROLLED STUDY; DISEASE ACTIVITY; DISEASE ASSOCIATION; DISEASE COURSE; DISEASE EXACERBATION; DISORDERS OF MITOCHONDRIAL FUNCTIONS; FEMALE; LEARNING DISORDER; MALE; MEMORY DISORDER; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; STATE DEPENDENT LEARNING; TRANSGENIC MOUSE; WILD TYPE;

EID: 82755161811     PISSN: 01974580     EISSN: 15581497     Source Type: Journal    
DOI: 10.1016/j.neurobiolaging.2011.01.008     Document Type: Article

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