Energy metabolism and ageing regulation: Metabolically driven deamidation of triosephosphate isomerase may contribute to proteostatic dysfunction

Ageing Research Reviews

Volumn 10, Issue 4, 2011, Pages 498-502

  Hipkiss, Alan R ^a  

^a ASTON UNIVERSITY   (United Kingdom)

Author keywords

Ageing; Altered proteins; Deamidation; Dihydroxyacetone; Glycation; Glycolysis; Methylglyoxal; Proteotoxicity; Triosephosphate isomerase

Indexed keywords

TRIOSEPHOSPHATE ISOMERASE;

AEROBIC EXERCISE; AGING; DEAMINATION; DIET RESTRICTION; ENERGY METABOLISM; GLYCATION; GLYCOLYSIS; HUMAN; LIFESPAN; NONHUMAN; PHENOTYPE; PROTEIN FUNCTION; PROTEOSTATIC DYSFUNCTION; REVIEW;

ANIMALS; CELL AGING; ENERGY METABOLISM; GLYCOLYSIS; HUMANS; PHENOTYPE; PROTEOSTASIS DEFICIENCIES; TISSUE DISTRIBUTION; TRIOSE-PHOSPHATE ISOMERASE;

EID: 80052324056     PISSN: 15681637     EISSN: 18729649     Source Type: Journal    
DOI: 10.1016/j.arr.2011.05.003     Document Type: Review

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