Excitotoxic neuroprotection and vulnerability with CaMKII inhibition

Molecular and Cellular Neuroscience

Volumn 46, Issue 4, 2011, Pages 720-730

  Ashpole, Nicole M ^a   Hudmon, Andy ^a  

^a INDIANA UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Aberrant calcium signaling; CaMKII inactivation; Cortical neurons; Excitotoxicity; Ischemia; Kinase inhibitors

Indexed keywords

CALCIUM CALMODULIN DEPENDENT PROTEIN KINASE II; CYCLOHEXIMIDE; GLUTAMIC ACID; PROTEIN KINASE INHIBITOR;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME INHIBITION; EXCITOTOXICITY; IMMUNOCYTOCHEMISTRY; NERVE CELL CULTURE; NERVE CELL NECROSIS; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; RAT;

AMINO ACID SEQUENCE; ANIMALS; BENZYLAMINES; CALCIUM; CALCIUM SIGNALING; CALCIUM-CALMODULIN-DEPENDENT PROTEIN KINASE TYPE 2; CELLS, CULTURED; GLUTAMIC ACID; MOLECULAR SEQUENCE DATA; NEURONS; NEUROPROTECTIVE AGENTS; PEPTIDES; PROTEIN KINASE INHIBITORS; RATS; RATS, SPRAGUE-DAWLEY; SULFONAMIDES;

EID: 79953043596     PISSN: 10447431     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mcn.2011.02.003     Document Type: Article

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