Widespread genomic breaks generated by activation-induced cytidine deaminase are prevented by homologous recombination

Nature Immunology

Volumn 11, Issue 9, 2010, Pages 820-826

  Hasham, Muneer G ^a   Donghia, Nina M ^a   Coffey, Eliot ^a   Maynard, Jane ^a   Snow, Kathy J ^a   Ames, Jacquelyn ^a   Wilpan, Robert Y ^a   He, Yishu ^a   King, Benjamin L ^a   Mills, Kevin D ^a  

^a JACKSON LABORATORY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ACTIVATION INDUCED CYTIDINE DEAMINASE; NUCLEIC ACID BINDING PROTEIN; UNCLASSIFIED DRUG; XRCC2 PROTEIN;

ARTICLE; B LYMPHOCYTE ACTIVATION; CELL CYCLE S PHASE; CELL SURVIVAL; CHROMOSOMAL INSTABILITY; CHROMOSOME 7Q; DOUBLE STRANDED DNA BREAK; ENZYME ACTIVITY; FLUORESCENCE IN SITU HYBRIDIZATION; GENE REARRANGEMENT; GENOMIC INSTABILITY; HOMOLOGOUS RECOMBINATION; LYMPHOCYTOTOXICITY; NEOPLASM; PRIORITY JOURNAL; SOMATIC MUTATION;

B-LYMPHOCYTES; CELL CYCLE; CELL SURVIVAL; CELLS, CULTURED; CYTIDINE DEAMINASE; DNA BREAKS; DNA-BINDING PROTEINS; FLOW CYTOMETRY; GENOMIC INSTABILITY; HUMANS; RECOMBINATION, GENETIC; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;

EID: 77955921194     PISSN: 15292908     EISSN: 15292916     Source Type: Journal    
DOI: 10.1038/ni.1909     Document Type: Article

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