-
1
-
-
0037283601
-
The relative contributions of insulin resistance and beta-cell dysfunction to the pathophysiology of type 2 diabetes
-
10.1007/s00125-003-1190-9 1:CAS:528:DC%2BD3sXhtVKkt74%3D 12637977
-
SE Kahn 2003 The relative contributions of insulin resistance and beta-cell dysfunction to the pathophysiology of type 2 diabetes Diabetologia 46 3 19 10.1007/s00125-003-1190-9 1:CAS:528:DC%2BD3sXhtVKkt74%3D 12637977
-
(2003)
Diabetologia
, vol.46
, pp. 3-19
-
-
Kahn, S.E.1
-
2
-
-
67349170464
-
Insulin resistance and exercise capacity in male children and adolescents with non-alcholic fatty liver disease
-
10.1007/s00592-008-0063-6 1:CAS:528:DC%2BD1MXlsFKksrs%3D 18839055
-
M Manco U Giordano A Turchetta, et al. 2009 Insulin resistance and exercise capacity in male children and adolescents with non-alcholic fatty liver disease Acta Diabetol 46 2 97 104 10.1007/s00592-008-0063-6 1:CAS:528:DC%2BD1MXlsFKksrs%3D 18839055
-
(2009)
Acta Diabetol
, vol.46
, Issue.2
, pp. 97-104
-
-
Manco, M.1
Giordano, U.2
Turchetta, A.3
-
3
-
-
0037219411
-
β-cell deficit and increased β-cell apoptosis in humans with type 2 diabetes
-
DOI 10.2337/diabetes.52.1.102
-
AE Butler J Janson S Bonner-Weir, et al. 2003 β-cell deficit and increased β-cell apoptosis in humans with type 2 diabetes Diabetes 52 102 110 10.2337/diabetes.52.1.102 1:CAS:528:DC%2BD3sXisVaisA%3D%3D 12502499 (Pubitemid 36042113)
-
(2003)
Diabetes
, vol.52
, Issue.1
, pp. 102-110
-
-
Butler, A.E.1
Janson, J.2
Bonner-Weir, S.3
Ritzel, R.4
Rizza, R.A.5
Butler, P.C.6
-
4
-
-
69849107894
-
Association between insulin secretion, insulin sensitivity and type 2 diabetes susceptibility variants identified in genome-wide association studies
-
10.1007/s00592-008-0080-5 1:CAS:528:DC%2BD1MXptVGmsrk%3D 19082521
-
SM Ruchat CE Elks RJ Loos, et al. 2009 Association between insulin secretion, insulin sensitivity and type 2 diabetes susceptibility variants identified in genome-wide association studies Acta Diabetol 46 3 217 226 10.1007/s00592-008-0080-5 1:CAS:528:DC%2BD1MXptVGmsrk%3D 19082521
-
(2009)
Acta Diabetol
, vol.46
, Issue.3
, pp. 217-226
-
-
Ruchat, S.M.1
Elks, C.E.2
Loos, R.J.3
-
5
-
-
12344323484
-
Type 2 diabetes - A matter of β-cell life and death?
-
DOI 10.1126/science.1104345
-
CJ Rhodes 2005 Type 2 diabetes: a matter of beta-cell life and death? Science 307 380 384 10.1126/science.1104345 1:CAS:528:DC%2BD2MXksFyhtg%3D%3D 15662003 (Pubitemid 40139973)
-
(2005)
Science
, vol.307
, Issue.5708
, pp. 380-384
-
-
Rhodes, C.J.1
-
6
-
-
59449083573
-
Genetic variations in the pancreatic ATP-sensitive potassium channel, beta-cell dysfunction, and susceptibility to type 2 diabetes
-
10.1007/s00592-008-0056-5 1:CAS:528:DC%2BD1MXht1Orsrc%3D 18758683
-
DA Chistiakov VA Potapov DC Khodirev, et al. 2009 Genetic variations in the pancreatic ATP-sensitive potassium channel, beta-cell dysfunction, and susceptibility to type 2 diabetes Acta Diabetol 46 1 43 49 10.1007/s00592-008- 0056-5 1:CAS:528:DC%2BD1MXht1Orsrc%3D 18758683
-
(2009)
Acta Diabetol
, vol.46
, Issue.1
, pp. 43-49
-
-
Chistiakov, D.A.1
Potapov, V.A.2
Khodirev, D.C.3
-
7
-
-
0032969276
-
Islet amyloid: A long-recognized but underappreciated pathological feature of type 2 diabetes
-
DOI 10.2337/diabetes.48.2.241
-
SE Kahn S Andrikopoulos CB Verchere 1999 Islet amyloid: a long-recognized but underappreciated pathological feature of type 2 diabetes Diabetes 48 241 253 10.2337/diabetes.48.2.241 1:CAS:528:DyaK1MXpsFKguw%3D%3D 10334297 (Pubitemid 29061094)
-
(1999)
Diabetes
, vol.48
, Issue.2
, pp. 241-253
-
-
Kahn, S.E.1
Andrikopoulos, S.2
Verchere, C.B.3
-
8
-
-
1542375103
-
Islet amyloid: A complication of islet dysfunction or an aetiological factor in Type 2 diabetes?
-
DOI 10.1007/s00125-003-1304-4
-
A Clark MR Nilsson 2004 Islet amyloid: a complication of islet dysfunction or an aetiological factor in type 2 diabetes? Diabetologia 47 157 169 10.1007/s00125-003-1304-4 1:CAS:528:DC%2BD2cXjtFCktLY%3D 14722650 (Pubitemid 38315352)
-
(2004)
Diabetologia
, vol.47
, Issue.2
, pp. 157-169
-
-
Clark, A.1
Nilsson, M.R.2
-
9
-
-
0034982387
-
High glucose causes apoptosis in cultured human pancreatic islets of Langerhans: A potential role for regulation of specific Bcl family genes toward an apoptotic cell death program
-
10.2337/diabetes.50.6.1290 1:CAS:528:DC%2BD3MXkvFSgsLk%3D 11375329
-
M Federici M Hribal L Perego, et al. 2001 High glucose causes apoptosis in cultured human pancreatic islets of Langerhans: a potential role for regulation of specific Bcl family genes toward an apoptotic cell death program Diabetes 50 6 1290 1301 10.2337/diabetes.50.6.1290 1:CAS:528: DC%2BD3MXkvFSgsLk%3D 11375329
-
(2001)
Diabetes
, vol.50
, Issue.6
, pp. 1290-1301
-
-
Federici, M.1
Hribal, M.2
Perego, L.3
-
10
-
-
59449102999
-
Differential lipid profile and hormonal response in type 2 diabetes by exogenous insulin aspart versus the insulin secretagogue repaglinide, at the same glycemic control
-
10.1007/s00592-008-0055-6 1:CAS:528:DC%2BD1MXht1Ors74%3D 18777156
-
SI Chisalita T Lindström P Eson Jennersjö, et al. 2009 Differential lipid profile and hormonal response in type 2 diabetes by exogenous insulin aspart versus the insulin secretagogue repaglinide, at the same glycemic control Acta Diabetol 46 1 35 42 10.1007/s00592-008-0055-6 1:CAS:528:DC%2BD1MXht1Ors74%3D 18777156
-
(2009)
Acta Diabetol
, vol.46
, Issue.1
, pp. 35-42
-
-
Chisalita, S.I.1
Lindström, T.2
Eson Jennersjö, P.3
-
11
-
-
0025957988
-
Basal and stimulated plasma levels of pancreatic amylin indicate its co-secretion with insulin in humans
-
10.1007/BF00404025 1:CAS:528:DyaK3MXltleiu7Y%3D 2055340
-
E Hartter T Svoboda B Ludvik, et al. 1991 Basal and stimulated plasma levels of pancreatic amylin indicate its co-secretion with insulin in humans Diabetologia 34 52 54 10.1007/BF00404025 1:CAS:528:DyaK3MXltleiu7Y%3D 2055340
-
(1991)
Diabetologia
, vol.34
, pp. 52-54
-
-
Hartter, E.1
Svoboda, T.2
Ludvik, B.3
-
12
-
-
0025302287
-
Evidence of cosecretion of islet amyloid polypeptide and insulin by beta cells
-
10.2337/diabetes.39.5.634 1:CAS:528:DyaK3cXktVSrsb0%3D 2185112
-
SE Kahn DA D'Alessio MW Schwartz, et al. 1990 Evidence of cosecretion of islet amyloid polypeptide and insulin by beta cells Diabetes 39 634 638 10.2337/diabetes.39.5.634 1:CAS:528:DyaK3cXktVSrsb0%3D 2185112
-
(1990)
Diabetes
, vol.39
, pp. 634-638
-
-
Kahn, S.E.1
D'Alessio, D.A.2
Schwartz, M.W.3
-
13
-
-
0029972089
-
Islet amyloid polypeptide: Actions and role in the pathogenesis of diabetes
-
1:CAS:528:DyaK28XjtlKrtbk%3D 8736810
-
A Clark SB Charge MK Badman, et al. 1996 Islet amyloid polypeptide: actions and role in the pathogenesis of diabetes Biochem Soc Trans 24 594 599 1:CAS:528:DyaK28XjtlKrtbk%3D 8736810
-
(1996)
Biochem Soc Trans
, vol.24
, pp. 594-599
-
-
Clark, A.1
Charge, S.B.2
Badman, M.K.3
-
14
-
-
69549126592
-
Pancreatic islet amyloidosis, beta-cell apoptosis, and alpha-cell proliferation are determinants of islet remodeling in type-2 diabetic baboons
-
Guardado-Mendoza R, Davalli AM, Chavez AO, et al (2009) Pancreatic islet amyloidosis, beta-cell apoptosis, and alpha-cell proliferation are determinants of islet remodeling in type-2 diabetic baboons. Proc Natl Acad Sci 18; 106(33):13992-13997
-
(2009)
Proc Natl Acad Sci
, vol.18-106
, Issue.33
, pp. 13992-13997
-
-
Guardado-Mendoza, R.1
Davalli, A.M.2
Chavez, A.O.3
-
15
-
-
34547638958
-
High expression rates of human islet amyloid polypeptide induce endoplasmic reticulum stress-mediated β-cell apoptosis, a characteristic of humans with type 2 but not type 1 diabetes
-
DOI 10.2337/db07-0197
-
CJ Huang CY Lin L Haataja, et al. 2007 High expression rates of human islet amyloid polypeptide induce endoplasmic reticulum stress mediated beta-cell apoptosis, a characteristic of humans with type 2 but not type 1 diabetes Diabetes 56 8 2016 2027 10.2337/db07-0197 1:CAS:528:DC%2BD2sXos12qs7w%3D 17475933 (Pubitemid 47201819)
-
(2007)
Diabetes
, vol.56
, Issue.8
, pp. 2016-2027
-
-
Huang, C.-J.1
Lin, C.-Y.2
Haataja, L.3
Gurlo, T.4
Butler, A.E.5
Rizza, R.A.6
Butler, P.C.7
-
16
-
-
0042822112
-
Increased β-cell apoptosis prevents adaptive increase in β-cell mass in mouse model of type 2 diabetes: Evidence for role of islet amyloid formation rather than direct action of amyloid
-
DOI 10.2337/diabetes.52.9.2304
-
AE Butler J Janson WC Soeller, et al. 2003 Increased beta cell apoptosis prevents adaptive increase in beta cell mass in mouse model of type 2 diabetes: evidence for role of islet amyloid formation rather than direct action of amyloid Diabetes 52 2304 2314 10.2337/diabetes.52.9.2304 1:CAS:528: DC%2BD3sXntFeqtbY%3D 12941770 (Pubitemid 37059501)
-
(2003)
Diabetes
, vol.52
, Issue.9
, pp. 2304-2314
-
-
Butler, A.E.1
Janson, J.2
Soeller, W.C.3
Butler, P.C.4
-
17
-
-
2542581025
-
Diabetes due to a progressive defect in β-cell mass in rats transgenic for human islet amyloid polypeptide (HIP rat): A new model for type 2 diabetes
-
DOI 10.2337/diabetes.53.6.1509
-
AE Butler J Jang T Gurlo, et al. 2004 Diabetes due to a progressive defect in beta cell mass in rats transgenic for human islet amyloid polypeptide (HIP rat): a new model for type 2 diabetes Diabetes 53 1509 1516 10.2337/diabetes.53.6.1509 1:CAS:528:DC%2BD2cXkslCgs7o%3D 15161755 (Pubitemid 38697663)
-
(2004)
Diabetes
, vol.53
, Issue.6
, pp. 1509-1516
-
-
Butler, A.E.1
Jang, J.2
Gurlo, T.3
Carty, M.D.4
Soeller, W.C.5
Butler, P.C.6
-
18
-
-
61449123871
-
Oxidative stress is induced by islet amyloid formation and time-dependently mediates amyloid-induced beta cell apoptosis
-
10.1007/s00125-008-1255-x 1:CAS:528:DC%2BD1MXisFSisrs%3D 19148619
-
S Zraika RL Hull J Udayasankar, et al. 2009 Oxidative stress is induced by islet amyloid formation and time-dependently mediates amyloid-induced beta cell apoptosis Diabetologia 52 4 626 635 10.1007/s00125-008-1255-x 1:CAS:528:DC%2BD1MXisFSisrs%3D 19148619
-
(2009)
Diabetologia
, vol.52
, Issue.4
, pp. 626-635
-
-
Zraika, S.1
Hull, R.L.2
Udayasankar, J.3
-
19
-
-
64649101777
-
Successful versus failed adaptation to high-fat diet-induced insulin resistance: The role of IAPP-induced beta-cell endoplasmic reticulum stress
-
10.2337/db08-1464 1:CAS:528:DC%2BD1MXktleiurk%3D 19151199
-
AV Matveyenko T Gurlo M Daval, et al. 2009 Successful versus failed adaptation to high-fat diet-induced insulin resistance: the role of IAPP-induced beta-cell endoplasmic reticulum stress Diabetes 58 4 906 916 10.2337/db08-1464 1:CAS:528:DC%2BD1MXktleiurk%3D 19151199
-
(2009)
Diabetes
, vol.58
, Issue.4
, pp. 906-916
-
-
Matveyenko, A.V.1
Gurlo, T.2
Daval, M.3
-
20
-
-
0024463227
-
Hyperproinsulinemia and amyloid in NIDDM: Clues to etiology of islet β-cell dysfunction?
-
10.2337/diabetes.38.11.1333 1:CAS:528:DyaK3cXlsVeh 2695369
-
D Porte Jr SE Kahn 1989 Hyperproinsulinemia and amyloid in NIDDM: clues to etiology of islet β-cell dysfunction? Diabetes 38 1333 1336 10.2337/diabetes.38.11.1333 1:CAS:528:DyaK3cXlsVeh 2695369
-
(1989)
Diabetes
, vol.38
, pp. 1333-1336
-
-
Porte Jr., D.1
Kahn, S.E.2
-
21
-
-
0024425540
-
Islet amyloid polypeptide (IAPP) and pro-IAPP immunoreactivity in human islets of Langerhans
-
DOI 10.1016/0168-8227(89)90008-9
-
P Westermark U Engstrom GT Westermark, et al. 1989 Islet amyloid polypeptide (IAPP) and pro-IAPP immunoreactivity in human islets of Langerhans Diabetes Res Clin Pract 7 219 226 10.1016/0168-8227(89)90008-9 1:CAS:528:DyaL1MXmtV2nt78%3D 2691219 (Pubitemid 19231484)
-
(1989)
Diabetes Research and Clinical Practice
, vol.7
, Issue.3
, pp. 219-226
-
-
Westermark, P.1
Engstrom, U.2
Westermark, G.T.3
Johnson, K.H.4
Permerth, J.5
Betsholtz, C.6
-
22
-
-
0032896436
-
Prolonged exposure of pancreatic beta cells to raised glucose concentrations results in increased cellular content of islet amyloid polypeptide precursors
-
DOI 10.1007/s001250051138
-
X Hou Z Ling E Quartier, et al. 1999 Prolonged exposure of pancreatic beta cells to raised glucose concentrations results in increased cellular content of islet amyloid polypeptide precursors Diabetologia 42 188 194 10.1007/s001250051138 1:CAS:528:DyaK1MXhtFajtr0%3D 10064099 (Pubitemid 29064241)
-
(1999)
Diabetologia
, vol.42
, Issue.2
, pp. 188-194
-
-
Hou, X.1
Ling, Z.2
Quartier, E.3
Foriers, A.4
Schuit, F.5
Pipeleers, D.6
Van Schravendijk, C.F.H.7
-
23
-
-
33749318236
-
2-terminal processing of human proislet amyloid polypeptide by the prohormone convertase PC2 leads to amyloid formation and cell death
-
DOI 10.2337/db05-1566
-
L Marzban CJ Rhodes DF Steiner, et al. 2006 Impaired NH2-terminal processing of human proislet amyloid polypeptide by the prohormone convertase PC2 leads to amyloid formation and cell death Diabetes 55 8 2192 2201 10.2337/db05-1566 1:CAS:528:DC%2BD28Xot1Ogtb0%3D 16873681 (Pubitemid 44743615)
-
(2006)
Diabetes
, vol.55
, Issue.8
, pp. 2192-2201
-
-
Marzban, L.1
Rhodes, C.J.2
Steiner, D.F.3
Haataja, L.4
Halban, P.A.5
Verchere, C.B.6
-
24
-
-
0035123349
-
The prohormone convertase enzyme 2 (PC2) is essential for processing pro-islet amyloid polypeptide at the NH2-terminal cleavage site
-
10.2337/diabetes.50.3.534 1:CAS:528:DC%2BD3MXhslGhu70%3D 11246872
-
J Wang J Xu J Finnerty, et al. 2001 The prohormone convertase enzyme 2 (PC2) is essential for processing pro-islet amyloid polypeptide at the NH2-terminal cleavage site Diabetes 50 3 534 539 10.2337/diabetes.50.3.534 1:CAS:528:DC%2BD3MXhslGhu70%3D 11246872
-
(2001)
Diabetes
, vol.50
, Issue.3
, pp. 534-539
-
-
Wang, J.1
Xu, J.2
Finnerty, J.3
-
25
-
-
0037379714
-
Islet amyloid polypeptide and type 2 diabetes
-
DOI 10.1016/S0531-5565(03)00004-4
-
L Marzban K Park CB Verchere 2003 Islet amyloid polypeptide and type 2 diabetes Exp Gerontol 38 347 351 10.1016/S0531-5565(03)00004-4 1:CAS:528:DC%2BD3sXisV2lu7s%3D 12670620 (Pubitemid 36379069)
-
(2003)
Experimental Gerontology
, vol.38
, Issue.4
, pp. 347-351
-
-
Marzban, L.1
Park, K.2
Verchere, C.B.3
-
26
-
-
0033758248
-
Amyloidogenicity of recombinant human proislet amyloid polypeptide (ProIAPP)
-
10.1016/S1074-5521(00)00034-X 1:CAS:528:DC%2BD3cXosVWitL8%3D 11094339
-
M Krampert J Bernhagen J Schmucker, et al. 2000 Amyloidogenicity of recombinant human proislet amyloid polypeptide (ProIAPP) Chem Biol 7 855 871 10.1016/S1074-5521(00)00034-X 1:CAS:528:DC%2BD3cXosVWitL8%3D 11094339
-
(2000)
Chem Biol
, vol.7
, pp. 855-871
-
-
Krampert, M.1
Bernhagen, J.2
Schmucker, J.3
-
27
-
-
33646509867
-
Intracellular amyloid-like deposits contain unprocessed pro-islet amyloid polypeptide (proIAPP) in beta cells of transgenic mice overexpressing the gene for human IAPP and transplanted human islets
-
10.1007/s00125-006-0206-7 1:CAS:528:DC%2BD28Xktlaltbw%3D 16570161
-
JF Paulsson A Andersson P Westermark, et al. 2006 Intracellular amyloid-like deposits contain unprocessed pro-islet amyloid polypeptide (proIAPP) in beta cells of transgenic mice overexpressing the gene for human IAPP and transplanted human islets Diabetologia 49 1237 1246 10.1007/s00125-006-0206-7 1:CAS:528:DC%2BD28Xktlaltbw%3D 16570161
-
(2006)
Diabetologia
, vol.49
, pp. 1237-1246
-
-
Paulsson, J.F.1
Andersson, A.2
Westermark, P.3
-
28
-
-
0031851293
-
Definition, diagnosis and classification of diabetes mellitus and its complications. Part 1: Diagnosis and classification of diabetes mellitus. Provisional report of a WHO consultation
-
DOI 10.1002/(SICI)1096-9136(199807)15:7<539::AID-DIA668>3.0.CO;2-S
-
KG Alberti PZ Zimmet 1998 Definition, diagnosis and classification of diabetes mellitus and its complications. 1. Diagnosis and classification of diabetes mellitus, provisional report of a WHO consultation Diabetes Med 15 539 553 10.1002/(SICI)1096-9136(199807)15:7<539::AID-DIA668>3.0.CO;2-S 1:STN:280:DyaK1czkvFCrsA%3D%3D (Pubitemid 28319935)
-
(1998)
Diabetic Medicine
, vol.15
, Issue.7
, pp. 539-553
-
-
Alberti, K.G.M.M.1
Zimmet, P.Z.2
-
29
-
-
67349227494
-
Amyloidogenic propensities and conformational properties of ProIAPP and IAPP in the presence of lipid bilayer membranes
-
Jha S, Sellin D, Seidel R, et al (2009) Amyloidogenic propensities and conformational properties of ProIAPP and IAPP in the presence of lipid bilayer membranes. J Mol Biol 26; 389(5):907-920
-
(2009)
J Mol Biol
, vol.26-389
, Issue.5
, pp. 907-920
-
-
Jha, S.1
Sellin, D.2
Seidel, R.3
-
31
-
-
0028912883
-
Increased secretory demand rather than a defect in the proinsulin conversion mechanism causes hyperproinsulinemia in a glucose-infusion rat model of non-insulindependent diabetes mellitus
-
10.1172/JCI117748 1:CAS:528:DyaK2MXktlKhtL0%3D 7883951
-
C Alarcon JL Leahy GT Schuppin, et al. 1995 Increased secretory demand rather than a defect in the proinsulin conversion mechanism causes hyperproinsulinemia in a glucose-infusion rat model of non-insulindependent diabetes mellitus J Clin Invest 95 1032 1039 10.1172/JCI117748 1:CAS:528:DyaK2MXktlKhtL0%3D 7883951
-
(1995)
J Clin Invest
, vol.95
, pp. 1032-1039
-
-
Alarcon, C.1
Leahy, J.L.2
Schuppin, G.T.3
-
32
-
-
0033048453
-
The mechanism of islet amyloid polypeptide toxicity is membrane disruption by intermediatesized toxic amyloid particles
-
10.2337/diabetes.48.3.491 1:CAS:528:DyaK1MXhsFymsbk%3D 10078548
-
J Janson RH Ashley D Harrison, et al. 1999 The mechanism of islet amyloid polypeptide toxicity is membrane disruption by intermediatesized toxic amyloid particles Diabetes 48 491 498 10.2337/diabetes.48.3.491 1:CAS:528: DyaK1MXhsFymsbk%3D 10078548
-
(1999)
Diabetes
, vol.48
, pp. 491-498
-
-
Janson, J.1
Ashley, R.H.2
Harrison, D.3
-
33
-
-
0042822112
-
Increased β-cell apoptosis prevents adaptive increase in β-cell mass in mouse model of type 2 diabetes: Evidence for role of islet amyloid formation rather than direct action of amyloid
-
DOI 10.2337/diabetes.52.9.2304
-
AE Butler J Janson WC Soeller, et al. 2003 Increased β-cell apoptosis prevents adaptive increase in β-cell mass in mouse model of type 2 diabetes: evidence for role of islet amyloid formation rather than direct action of amyloid Diabetes 52 2304 2314 10.2337/diabetes.52.9.2304 1:CAS:528:DC%2BD3sXntFeqtbY%3D 12941770 (Pubitemid 37059501)
-
(2003)
Diabetes
, vol.52
, Issue.9
, pp. 2304-2314
-
-
Butler, A.E.1
Janson, J.2
Soeller, W.C.3
Butler, P.C.4
-
34
-
-
20444496481
-
Calcium dysregulation and membrane disruption as a ubiquitous neurotoxic mechanism of soluble amyloid oligomers
-
DOI 10.1074/jbc.M500997200
-
A Demuro E Mina R Kayed, et al. 2005 Calcium dysregulation and membrane disruption as a ubiquitous neurotoxic mechanism of soluble amyloid oligomers J Biol Chem 280 17294 17300 10.1074/jbc.M500997200 1:CAS:528:DC%2BD2MXjsFOguro%3D 15722360 (Pubitemid 41389198)
-
(2005)
Journal of Biological Chemistry
, vol.280
, Issue.17
, pp. 17294-17300
-
-
Demuro, A.1
Mina, E.2
Kayed, R.3
Milton, S.C.4
Parker, I.5
Glabe, C.G.6
-
35
-
-
33645728523
-
Neurodegenerative diseases: New concepts of pathogenesis and their therapeutic implications
-
10.1146/annurev.pathol.1.110304.100113 1:CAS:528:DC%2BD28XktVOitbo%3D
-
DM Skovronsky VM-Y Lee JQ Trojanowski 2006 Neurodegenerative diseases: new concepts of pathogenesis and their therapeutic implications Annu Rev Pathol Mech Dis 1 151 170 10.1146/annurev.pathol.1.110304.100113 1:CAS:528: DC%2BD28XktVOitbo%3D
-
(2006)
Annu Rev Pathol Mech Dis
, vol.1
, pp. 151-170
-
-
Skovronsky, D.M.1
Vm-Y, L.2
Trojanowski, J.Q.3
-
36
-
-
21344470854
-
Aberrant processing of human proislet amyloid polypeptide results in increased amyloid formation
-
DOI 10.2337/diabetes.54.7.2117
-
JF Paulsson GT Westermark 2005 Aberrant processing of human proislet amyloid polypeptide results in increased amyloid formation Diabetes 54 7 2117 2125 10.2337/diabetes.54.7.2117 1:CAS:528:DC%2BD2MXmtVeltr8%3D 15983213 (Pubitemid 40911275)
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(2005)
Diabetes
, vol.54
, Issue.7
, pp. 2117-2125
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Paulsson, J.F.1
Westermark, G.T.2
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