Cardioprotection by GSK-3 inhibition: Role of enhanced glycogen synthesis and attenuation of calcium overload

Cardiovascular Research

Volumn 86, Issue 3, 2010, Pages 478-486

  Omar, Mohamed A ^a   Wang, Lianguo ^a   Clanachan, Alexander S ^a  

^a UNIVERSITY OF ALBERTA   (Canada)

Author keywords

Calcium; Glucose metabolism; GSK 3; Heart; Ischaemia reperfusion

Indexed keywords

2 (2,4 DICHLOROPHENYL) 3 (1 METHYL 3 INDOLYL)MALEIMIDE; CALCIUM; GLUCOSE; GLYCOGEN; GLYCOGEN SYNTHASE KINASE 3; INDO 1; PALMITIC ACID; PROTON;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CALCIUM CELL LEVEL; CELL DEATH; CONTROLLED STUDY; CONVALESCENCE; ENZYME INHIBITION; FLUORESCENCE IMAGING; GLUCOSE METABOLISM; GLUCOSE OXIDATION; GLYCOGEN SYNTHESIS; GLYCOLYSIS; HEART INFARCTION; HEART LEFT VENTRICLE; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE ISCHEMIA; HEART PROTECTION; ISOLATED HEART; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; REPERFUSION;

ANIMALS; CALCIUM; CARDIOTONIC AGENTS; DISEASE MODELS, ANIMAL; DOWN-REGULATION; GLUCOSE; GLYCOGEN; GLYCOGEN SYNTHASE KINASE 3; GLYCOLYSIS; HYDROGEN-ION CONCENTRATION; INDOLES; MALE; MALEIMIDES; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; PALMITIC ACID; PERFUSION; PROTEIN KINASE INHIBITORS; RATS; RATS, SPRAGUE-DAWLEY; RECOVERY OF FUNCTION; SODIUM-CALCIUM EXCHANGER; SODIUM-HYDROGEN ANTIPORTER; TIME FACTORS; UP-REGULATION; VENTRICULAR FUNCTION, LEFT;

EID: 77952345317     PISSN: 00086363     EISSN: 17553245     Source Type: Journal    
DOI: 10.1093/cvr/cvp421     Document Type: Article

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