H2O2-induced left ventricular dysfunction in isolated working rat hearts is independent of calcium accumulation

Journal of Molecular and Cellular Cardiology

Volumn 45, Issue 6, 2008, Pages 787-795

  Wang, Lianguo ^a   Lopaschuk, Gary D ^a   Clanachan, Alexander S ^a  

^a UNIVERSITY OF ALBERTA   (Canada)

Author keywords

Ca2+ overload; H2O2; Late INa; LV dysfunction; Oxidative stress

Indexed keywords

CALCIUM ION; HYDROGEN PEROXIDE; LIDOCAINE; N [1 [4 (4 FLUOROPHENOXY)BUTYL] 4 PIPERIDINYL] N METHYL 2 BENZOTHIAZOLAMINE; OUABAIN; REACTIVE OXYGEN METABOLITE; SEA ANEMONE TOXIN;

ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; DIASTOLE; DRUG EFFECT; HEART LEFT VENTRICLE; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE CONTRACTILITY; HEART MUSCLE ISCHEMIA; ISOLATED HEART; MALE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; REPERFUSION INJURY; SODIUM CURRENT; SYSTOLE;

ANIMALS; ANTI-ARRHYTHMIA AGENTS; BENZOTHIAZOLES; CALCIUM; CALCIUM CHANNEL BLOCKERS; CARDIOTONIC AGENTS; CNIDARIAN VENOMS; HYDROGEN PEROXIDE; ION TRANSPORT; LIDOCAINE; MALE; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; ORGAN CULTURE TECHNIQUES; OUABAIN; OXIDANTS; PIPERIDINES; RATS; RATS, SPRAGUE-DAWLEY; VENTRICULAR DYSFUNCTION, LEFT;

ACTINIARIA; RATTUS; STROPHANTHUS GRATUS;

EID: 55649104113     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2008.08.010     Document Type: Article

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