Ranolazine decreases diastolic calcium accumulation caused by ATX-II or ischemia in rat hearts

Journal of Molecular and Cellular Cardiology

Volumn 41, Issue 6, 2006, Pages 1031-1038

  Fraser, Heather ^a   Belardinelli, Luiz ^a   Wang, Lianguo ^b   Light, Peter E ^b   McVeigh, Jeffrey J ^a   Clanachan, Alexander S ^b  

^a CV THERAPEUTICS INC   (United States)

^b UNIVERSITY OF ALBERTA   (Canada)

Author keywords

Ca2+ overload; Ischemia; LV function; Ranolazine; Voltage gated sodium channels

Indexed keywords

ANGIOTENSIN II; CALCIUM ION; RANOLAZINE;

ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; CALCIUM CURRENT; CALCIUM TRANSPORT; CONTROLLED STUDY; DRUG EFFECT; HEART LEFT VENTRICLE; HEART MUSCLE ISCHEMIA; HEART VENTRICLE FUNCTION; ISOLATED HEART; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; REPERFUSION; STATISTICAL SIGNIFICANCE;

ANIMALS; CALCIUM; CNIDARIAN VENOMS; DIASTOLE; HEART; MALE; MYOCARDIAL ISCHEMIA; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; PERFUSION; PIPERAZINES; RATS; RATS, SPRAGUE-DAWLEY; SODIUM CHANNELS; VENTRICULAR FUNCTION, LEFT;

ACTINIARIA;

EID: 33751214195     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2006.08.012     Document Type: Article

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