Homocysteine induces caspase activation by endoplasmic reticulum stress in platelets from type 2 diabetics and healthy donors

Thrombosis and Haemostasis

Volumn 103, Issue 5, 2010, Pages 1022-1032

  Zbidi, Hanene ^a   Redondo, Pedro C ^b   López, Jose J ^b   Bartegi, Aghleb ^a   Salido, Gines M ^b   Rosado, Juan A ^b  

^a UNIVERSITY OF MONASTIR   (Tunisia)

^b UNIVERSITY OF EXTREMADURA   (Spain)

Author keywords

Apoptosis; Caspases; Diabetes mellitus; ER stress; Homocysteine

Indexed keywords

CASPASE; CASPASE 3; CASPASE 9; HOMOCYSTEINE; INITIATION FACTOR 2ALPHA; CINNAMIC ACID DERIVATIVE; PROTEIN KINASE R; SALUBRINAL; THIOUREA;

ARTICLE; CELL VIABILITY; CONTROLLED STUDY; ENDOPLASMIC RETICULUM; ENDOPLASMIC RETICULUM STRESS; ENZYME ACTIVATION; ENZYME PHOSPHORYLATION; HUMAN; HYPERHOMOCYSTEINEMIA; MICROANGIOPATHY; MORTALITY; NON INSULIN DEPENDENT DIABETES MELLITUS; PRIORITY JOURNAL; RISK FACTOR; THROMBOCYTE; VASCULAR DISEASE; ANALOGS AND DERIVATIVES; APOPTOSIS; BLOOD DONOR; CALCIUM SIGNALING; CELL CULTURE; COMPLICATION; DIABETIC ANGIOPATHY; DRUG EFFECTS; GENETICS; METABOLISM; PATHOLOGY; PHYSIOLOGICAL STRESS;

APOPTOSIS; BLOOD DONORS; BLOOD PLATELETS; CALCIUM SIGNALING; CASPASE 3; CASPASE 9; CELLS, CULTURED; CINNAMATES; DIABETES MELLITUS, TYPE 2; DIABETIC ANGIOPATHIES; EIF-2 KINASE; ENDOPLASMIC RETICULUM; ENZYME ACTIVATION; HOMOCYSTEINE; HUMANS; HYPERHOMOCYSTEINEMIA; STRESS, PHYSIOLOGICAL; THIOUREA;

EID: 77952039180     PISSN: 03406245     EISSN: None     Source Type: Journal    
DOI: 10.1160/TH09-08-0552     Document Type: Article

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