Role of Akt isoforms in IGF-I-mediated signaling and survival in myoblasts

Biochemical and Biophysical Research Communications

Volumn 389, Issue 1, 2009, Pages 117-121

  Matheny Jr , Ronald W ^a,b   Adamo, Martin L ^a,b  

^a Mays Cancer Center at UT Health San Antonio   (United States)

^b UNIVERSITY OF TEXAS HEALTH SCIENCE CENTER AT SAN ANTONIO   (United States)

Author keywords

Akt PKB; Apoptosis; Insulin like growth factor I; Myoblast; Oxidative stress

Indexed keywords

HYDROGEN PEROXIDE; PROTEIN BAD; PROTEIN KINASE AKT1; PROTEIN KINASE AKT3; PROTEIN KINASE B; PROTEIN SERINE THREONINE KINASE; PROTEIN SUBUNIT; SOMATOMEDIN C; TRANSCRIPTION FACTOR FKHR; TRANSCRIPTION FACTOR FKHRL1; UNCLASSIFIED DRUG;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL DEATH; CELL SURVIVAL; MYOBLAST; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; SIGNAL TRANSDUCTION;

ANIMALS; APOPTOSIS; BCL-ASSOCIATED DEATH PROTEIN; CELL LINE; CELL SURVIVAL; FORKHEAD TRANSCRIPTION FACTORS; HYDROGEN PEROXIDE; INSULIN-LIKE GROWTH FACTOR I; ISOENZYMES; MICE; MYOBLASTS; OXIDATIVE STRESS; PROTO-ONCOGENE PROTEINS C-AKT; RNA, SMALL INTERFERING; SIGNAL TRANSDUCTION;

MAMMALIA;

EID: 70349148975     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2009.08.101     Document Type: Article

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