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Volumn 285, Issue 1-2, 2009, Pages 159-171

Increases in nuclear p65 activation in dystrophic skeletal muscle are secondary to increases in the cellular expression of p65 and are not solely produced by increases in IκB-α kinase activity

Author keywords

Akt; Becker muscular dystrophy; Cell signaling; Diaphragm; Duchenne muscular dystrophy; Limb musculature; mdx mouse; NF B; Respiratory musculature

Indexed keywords

I KAPPA B ALPHA; I KAPPA B BETA; I KAPPA B KINASE; I KAPPA B KINASE ALPHA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; PROTEIN KINASE B; PROTEIN P100; PROTEIN P50; PROTEIN P52; SUMO 1 PROTEIN; SYNAPTOTAGMIN I; TRANSCRIPTION FACTOR RELB; TUMOR NECROSIS FACTOR RECEPTOR ASSOCIATED FACTOR 3;

EID: 69549127944     PISSN: 0022510X     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.jns.2009.06.030     Document Type: Article
Times cited : (17)

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