-
1
-
-
24044481966
-
Copper, oxidative stress, and human health
-
DOI 10.1016/j.mam.2005.07.015, PII S0098299705000488
-
Uriu-Adams JY, Keen CL (2005) Copper, oxidative stress, and human health. Mol Aspects Med 26: 268-298. (Pubitemid 41225353)
-
(2005)
Molecular Aspects of Medicine
, vol.26
, Issue.4-5 SPEC. ISS
, pp. 268-298
-
-
Uriu-Adams, J.Y.1
Keen, C.L.2
-
3
-
-
38049158328
-
Cause of death in Wilson disease
-
Walshe JM (2007) Cause of death in Wilson disease. Mov Disord 22: 2216-2220.
-
(2007)
Mov Disord
, vol.22
, pp. 2216-2220
-
-
Walshe, J.M.1
-
4
-
-
0043196697
-
Cancer therapy with tetrathiomolybdate: Antiangiogenesis by lowering body copper
-
Brewer GJ, Merajver SD (2002) Cancer therapy with tetrathiomolybdate: antiangiogenesis by lowering body copper. Integr Cancer Ther 1: 327-337.
-
(2002)
Integr Cancer Ther
, vol.1
, pp. 327-337
-
-
Brewer, G.J.1
Merajver, S.D.2
-
5
-
-
33646134391
-
The use of tetrathiomolybdate in treating fibrotic, inflammatory, and autoimmune diseases, including the non-obese diabetic mouse model
-
Brewer GJ, Dick R, Zeng C, Hou G (2006) The use of tetrathiomolybdate in treating fibrotic, inflammatory, and autoimmune diseases, including the non-obese diabetic mouse model. J Inorg Biochem 100: 927-930.
-
(2006)
J Inorg Biochem
, vol.100
, pp. 927-930
-
-
Brewer, G.J.1
Dick, R.2
Zeng, C.3
Hou, G.4
-
6
-
-
33748340865
-
Copper binding by tetrathiomolybdate attenuates angiogenesis and tumor cell proliferation through the inhibition of superoxide dismutase 1
-
DOI 10.1158/1078-0432.CCR-06-0171
-
Juarez JC, Betancourt Jr O, Pirie-Shepherd SR, Guan X, Price ML, Shaw DE, Mazar AP, Donate F (2006) Copper binding by tetrathiomolybdate attenuates angiogenesis and tumor cell proliferation through the inhibition of superoxide dismutase 1. Clin Cancer Res 12: 4974-4981. (Pubitemid 44338586)
-
(2006)
Clinical Cancer Research
, vol.12
, Issue.16
, pp. 4974-4982
-
-
Juarez, J.C.1
Betancourt Jr., O.2
Pirie-Shepherd, S.R.3
Guan, X.4
Price, M.L.5
Shaw, D.E.6
Mazar, A.P.7
Donate, F.8
-
7
-
-
33846110887
-
Tetrathiomolybdate is effective in a mouse model of arthritis
-
McCubbin MD, Hou G, Abrams GD, Dick R, Zhang Z, Brewer GJ (2006) Tetrathiomolybdate is effective in a mouse model of arthritis. J Rheumatol 33: 2501-2506. (Pubitemid 46067832)
-
(2006)
Journal of Rheumatology
, vol.33
, Issue.12
, pp. 2501-2506
-
-
McCubbin, M.D.1
Hou, G.2
Abrams, G.D.3
Dick, R.4
Zhang, Z.5
Brewer, G.J.6
-
8
-
-
42449134964
-
Tetrathiomolybdate protects against bile duct ligation-induced cholestatic liver injury and fibrosis
-
DOI 10.1124/jpet.107.131227
-
Song M, Song Z, Barve S, Zhang J, Chen T, Liu M, Arteel GE, Brewer GJ, McClain CJ (2008) Tetrathiomolybdate protects against bile duct ligation- induced cholestatic liver injury and fibrosis. J Pharmacol Exp Ther 325: 409-416. (Pubitemid 351574799)
-
(2008)
Journal of Pharmacology and Experimental Therapeutics
, vol.325
, Issue.2
, pp. 409-416
-
-
Song, M.1
Song, Z.2
Barve, S.3
Zhang, J.4
Chen, T.5
Liu, M.6
Arteel, G.E.7
Brewer, G.J.8
McClain, C.J.9
-
9
-
-
56349135090
-
Efficacy of tetrathiomolybdate in a mouse model of multiple sclerosis
-
G. Hou, G.D. Abrams, R. Dick, G.J. Brewer, Efficacy of tetrathiomolybdate in a mouse model of multiple sclerosis. Transl. Res. 152 (2008) 239-244.
-
(2008)
Transl. Res.
, vol.152
, pp. 239-244
-
-
Hou, G.1
Abrams, G.D.2
Dick, R.3
Brewer, G.J.4
-
10
-
-
49249098236
-
A copper (II)-selective chelator ameliorates diabetes- Evoked renal fibrosis and albuminuria, and suppresses pathogenic TGF-beta activation in the kidneys of rats used as a model of diabetes
-
Gong D, Lu J, Chen X, Reddy S, Crossman CJ, Glyn-Jones S, Choong YS, Kennedy J, Barry B, Zhang S, Chan YK, Ruggiero K, Phillips AR, Cooper GJ (2008) A copper (II)-selective chelator ameliorates diabetes- evoked renal fibrosis and albuminuria, and suppresses pathogenic TGF-beta activation in the kidneys of rats used as a model of diabetes. Diabetologia 51: 1741-1751.
-
(2008)
Diabetologia
, vol.51
, pp. 1741-1751
-
-
Gong, D.1
Lu, J.2
Chen, X.3
Reddy, S.4
Crossman, C.J.5
Glyn-Jones, S.6
Choong, Y.S.7
Kennedy, J.8
Barry, B.9
Zhang, S.10
Chan, Y.K.11
Ruggiero, K.12
Phillips, A.R.13
Cooper, G.J.14
-
11
-
-
0025096104
-
Autoxidative glycosylation and possible involvement of peroxides and free radicals in LDL modification by glucose
-
Hunt JV, Smith CC, Wolff SP (1990) Autoxidative glycosylation and possible involvement of peroxides and free radicals in LDL modification by glucose. Diabetes 39: 1420-1424.
-
(1990)
Diabetes
, vol.39
, pp. 1420-1424
-
-
Hunt, J.V.1
Smith, C.C.2
Wolff, S.P.3
-
12
-
-
0029152174
-
Glycation accelerates the oxidation of low density lipoprotein by copper ions
-
Kobayashi K, Watanabe J, Umeda F, Nawata H (1995) Glycation accelerates the oxidation of low density lipoprotein by copper ions. Endocr J 42: 461-465.
-
(1995)
Endocr J
, vol.42
, pp. 461-465
-
-
Kobayashi, K.1
Watanabe, J.2
Umeda, F.3
Nawata, H.4
-
13
-
-
34447251553
-
Copper-mediated formation of hydrogen peroxide from the amylin peptide: A novel mechanism for degeneration of islet cells in type-2 diabetes mellitus?
-
DOI 10.1016/j.febslet.2007.06.061, PII S0014579307007168
-
Masad A, Hayes L, Tabner BJ, Turnbull S, Cooper LJ, Fullwood NJ, German MJ, Kametani F, El-Agnaf OM, Allsop D (2007) Copper-mediated formation of hydrogen peroxide from the amylin peptide: A novel mechanism for degeneration of islet cells in type-2 diabetes mellitus? FEBS Lett 581: 3489-3493. (Pubitemid 47048200)
-
(2007)
FEBS Letters
, vol.581
, Issue.18
, pp. 3489-3493
-
-
Masad, A.1
Hayes, L.2
Tabner, B.J.3
Turnbull, S.4
Cooper, L.J.5
Fullwood, N.J.6
German, M.J.7
Kametani, F.8
El-Agnaf, O.M.A.9
Allsop, D.10
-
14
-
-
0030039242
-
Oxidative damage to DNA in diabetes mellitus
-
DOI 10.1016/S0140-6736(96)90013-6
-
Dandona P, Thusu K, Cook S, Snyder B, Makowski J, Armstrong D, Nicotera T (1996) Oxidative damage to DNA in diabetes mellitus. Lancet 347: 444-445. (Pubitemid 26054071)
-
(1996)
Lancet
, vol.347
, Issue.8999
, pp. 444-445
-
-
Dandona, P.1
Thusu, K.2
Cook, S.3
Snyder, B.4
Makowski, J.5
Armstrong, D.6
Nicotera, T.7
-
15
-
-
0032998220
-
Hyperglycemia causes oxidative stress in pancreatic β-cells of GK rats, a model of type 2 diabetes
-
Ihara Y, Toyokuni S, Uchida K, Odaka H, Tanaka T, Ikeda H, Hiai H, Seino Y, Yamada Y (1999) Hyperglycemia causes oxidative stress in pancreatic β-cells of GK rats, a model of type 2 diabetes. Diabetes 48: 927-932. (Pubitemid 29153099)
-
(1999)
Diabetes
, vol.48
, Issue.4
, pp. 927-932
-
-
Ihara, Y.1
Toyokuni, S.2
Uchida, K.3
Odaka, H.4
Tanaka, T.5
Ikeda, H.6
Hiai, H.7
Seino, Y.8
Yamada, Y.9
-
16
-
-
46749153447
-
Hepatic failure and enhanced oxidative stress in mitochondrial diabetes
-
DOI 10.1507/endocrj.K07E-091
-
Takahashi Y, Iida K, Takeno R, Kitazawa R, Kitazawa S, Kitamura H, Fujioka Y, Yamada H, Kanda F, Ohta S, Nishimaki K, Fujimoto M, Kondo T, Iguchi G, Takahashi K, Kaji H, Okimura Y, Chihara K (2008) Hepatic failure and enhanced oxidative stress in mitochondrial diabetes. Endocr J 55: 509-514. (Pubitemid 351949745)
-
(2008)
Endocrine Journal
, vol.55
, Issue.3
, pp. 509-514
-
-
Takahashi, Y.1
Iida, K.2
Takeno, R.3
Kitazawa, R.4
Kitazawa, S.5
Kitamura, H.6
Fujioka, Y.7
Yamada, H.8
Kanda, F.9
Ohta, S.10
Nishimaki, K.11
Fujimoto, M.12
Kondo, T.13
Iguchi, G.14
Takahashi, K.15
Kaji, H.16
Okimura, Y.17
Chihara, K.18
-
17
-
-
0031020374
-
Glycation-dependent, reactive oxygen species-mediated suppression of the insulin gene promoter activity in HIT cells
-
Matsuoka T, Kajimoto Y, Watada H, Kaneto H, Kishimoto M, Umayahara Y, Fujitani Y, Kamada T, Kawamori R, Yamasaki Y (1997) Glycation-dependent, reactive oxygen species-mediated suppression of the insulin gene promoter activity in HIT cells. J Clin Invest 99 144-150. (Pubitemid 27058305)
-
(1997)
Journal of Clinical Investigation
, vol.99
, Issue.1
, pp. 144-150
-
-
Matsuoka, T.-A.1
Kajimoto, Y.2
Watada, H.3
Kaneto, H.4
Kishimoto, M.5
Umayahara, Y.6
Fujitani, Y.7
Kamada, T.8
Kawamori, R.9
Yamasaki, Y.10
-
18
-
-
0031683542
-
Prolonged oxidative stress impairs insulin-induced GLUT4 translocation in 3T3-L1 adipocytes
-
DOI 10.2337/diabetes.47.10.1562
-
Rudich A, Tirosh A, Potashnik R, Hemi R, Kanety H, Bashan N (1998) Prolonged oxidative stress impairs insulin-induced GLUT4 translocation in 3T3-L1 adipocytes. Diabetes 47: 1562-1569. (Pubitemid 28445343)
-
(1998)
Diabetes
, vol.47
, Issue.10
, pp. 1562-1569
-
-
Rudich, A.1
Tlrosh, A.2
Potashnik, R.3
Hemi, R.4
Kanety, H.5
Bashan, N.6
-
19
-
-
0032863183
-
Prevention of glucose toxicity in HIT-T15 cells and Zucker diabetic fatty rats by antioxidants
-
DOI 10.1073/pnas.96.19.10857
-
Tanaka Y, Gleason CE, Tran POT, Harmon JS, Robertson RP (1999) Prevention of glucose toxicity in HIT-T15 cells and Zucker diabetic fatty rats by antioxidants. Proc Natl Acad Sci USA 96: 10857-10862. (Pubitemid 29442234)
-
(1999)
Proceedings of the National Academy of Sciences of the United States of America
, vol.96
, Issue.19
, pp. 10857-10862
-
-
Tanaka, Y.1
Gleason, C.E.2
Tran, P.O.T.3
Harmon, J.S.4
Robertson, R.P.5
-
20
-
-
0033537830
-
Oxidative stress disrupts insulin-induced cellular redistribution of insulin receptor substrate-1 and phosphatidylinositol 3-kinase in 3T3-L1 adipocytes: A putative cellular mechanism for impaired protein kinase B activation and glut4 translocation
-
Tirosh A, Potashnik R, Bashan N, Rudich A (1999) Oxidative stress disrupts insulin-induced cellular redistribution of insulin receptor substrate-1 and phosphatidylinositol 3-kinase in 3T3-L1 adipocytes: a putative cellular mechanism for impaired protein kinase B activation and GLUT4 translocation. J Biol Chem 274: 10595-10602. (Pubitemid 129518360)
-
(1999)
Journal of Biological Chemistry
, vol.274
, Issue.15
, pp. 10595-10602
-
-
Tirosh, A.1
Potashnik, R.2
Bashan, N.3
Rudich, A.4
-
21
-
-
0035903192
-
Activation of the Hexosamine Pathway Leads to Deterioration of Pancreatic β-Cell Function through the Induction of Oxidative Stress
-
DOI 10.1074/jbc.M104115200
-
Kaneto H, Xu G, Song KH, Suzuma K, Bonner-Weir S, SA, Weir GC (2001) Activation of the hexosamine pathway leads to deterioration of pancreatic β-cell function by provoking oxidative stress. J Biol Chem 276: 31099-31104. (Pubitemid 37384975)
-
(2001)
Journal of Biological Chemistry
, vol.276
, Issue.33
, pp. 31099-31104
-
-
Kaneto, H.1
Xu, G.2
Song, K.-H.3
Suzuma, K.4
Bonner-Weir, S.5
Sharma, A.6
Weir, G.C.7
-
22
-
-
0037119417
-
Involvement of c-Jun n-terminal kinase in oxidative stress-mediated suppression of insulin gene expression
-
Kaneto H, Xu G, Fujii N, Kim S, Bonner-Weir S, Weir GC (2002) Involvement of c-Jun n-terminal kinase in oxidative stress-mediated suppression of insulin gene expression. J Biol Chem 277: 30010-30018.
-
(2002)
J Biol Chem
, vol.277
, pp. 30010-30018
-
-
Kaneto, H.1
Xu, G.2
Fujii, N.3
Kim, S.4
Bonner-Weir, S.5
Weir, G.C.6
-
23
-
-
0036796875
-
Oxidative stress and stress-activated signaling pathways: A unifying hypothesis of type 2 diabetes
-
Evans JL, Goldfine ID, Maddux BA, Grodsky GM (2002) Oxidative stress and stress-activated signaling pathways: a unifying hypothesis of type 2 diabetes. Endocrine Rev. 23: 599-622.
-
(2002)
Endocrine Rev
, vol.23
, pp. 599-622
-
-
Evans, J.L.1
Goldfine, I.D.2
Maddux, B.A.3
Grodsky, G.M.4
-
24
-
-
10744227975
-
2-terminal Kinase
-
DOI 10.2337/diabetes.52.12.2896
-
Kawamori D, Kajimoto Y, Kaneto H, Umayahara Y, Fujitani Y, Miyatsuka T, Watada H, Leibiger IB, Yamasaki Y, Hori M (2003) Oxidative stress induces nucleo-cytoplasmic translocation of pancreatic transcription factor PdX-1 through activation of c-Jun n-terminal kinase. Diabetes 52: 2896-2904. (Pubitemid 37466858)
-
(2003)
Diabetes
, vol.52
, Issue.12
, pp. 2896-2904
-
-
Kawamori, D.1
Kajimoto, Y.2
Kaneto, H.3
Umayahara, Y.4
Fujitani, Y.5
Miyatsuka, T.6
Watada, H.7
Leibiger, I.B.8
Yamasaki, Y.9
Hori, M.10
-
25
-
-
5644248079
-
Chronic oxidative stress as a central mechanism for glucose toxicity in pancreatic islet beta cells in diabetes
-
Robertson RP (2004) Chronic oxidative stress as a central mechanism for glucose toxicity in pancreatic islet beta cells in diabetes. J Biol Chem 271: 42351-42354.
-
(2004)
J Biol Chem
, vol.271
, pp. 42351-42354
-
-
Robertson, R.P.1
-
26
-
-
18244370762
-
Inflammation, stress, and diabetes
-
Wellen KE, Hotamisligil G (2005) Inflammation, stress, and diabetes. J Clin Invest 115: 1111-1119.
-
(2005)
J Clin Invest
, vol.115
, pp. 1111-1119
-
-
Wellen, K.E.1
Hotamisligil, G.2
-
27
-
-
43449091944
-
PDX-1 and MafA play a crucial role in pancreatic β-cell differentiation and maintenance of mature β-cell function
-
DOI 10.1507/endocrj.K07E-041
-
Kaneto H, Miyatsuka T, Kawamori D, Yamamoto K, Kato K, Shiraiwa T, Katakami N, Yamasaki Y, Matsuhisa M, Matsuoka TA (2008) PDX-1 and MafA play a crucial role in pancreatic β-cell differentiation and maintenance of mature β-cell function. Endocr J 55: 235-252. (Pubitemid 351670948)
-
(2008)
Endocrine Journal
, vol.55
, Issue.2
, pp. 235-252
-
-
Kaneto, H.1
Miyatsuka, T.2
Kawamori, D.3
Yamamoto, K.4
Kato, K.5
Shiraiwa, T.6
Katakami, N.7
Yamasaki, Y.8
Matsuhisa, M.9
Matsuoka, T.-A.10
-
28
-
-
40649115005
-
Roles and regulation of transcription factor MafA in islet β-cells
-
Aramata S, Han SI, Kataoka K (2007) Roles and regulation of transcription factor MafA in islet β-cells. Endocr J 54: 656-666.
-
(2007)
Endocr J
, vol.54
, pp. 656-666
-
-
Aramata, S.1
Han, S.I.2
Kataoka, K.3
-
29
-
-
0033499583
-
Beneficial effects of antioxidants in diabetes: Possible protection of pancreatic β-cells against glucose toxicity
-
Kaneto H, Kajimoto Y, Miyagawa J, Matsuoka T, Fujitani Y, Umayahara Y, Hanafusa T, Matsuzawa Y, Yamasaki Y, Hori M (1999) Beneficial effects of antioxidants for diabetes: possible protection of pancreatic β-cells against glucose toxicity. Diabetes 48: 2398-2406. (Pubitemid 30395432)
-
(1999)
Diabetes
, vol.48
, Issue.12
, pp. 2398-2406
-
-
Kaneto, H.1
Kajimoto, Y.2
Miyagawa, J.3
Matsuoka, T.4
Fujitani, Y.5
Umayahara, Y.6
Hanafusa, T.7
Matsuzawa, Y.8
Yamasaki, Y.9
Hori, M.10
-
30
-
-
0032863183
-
Prevention of glucose toxicity in HIT-T15 cells and Zucker diabetic fatty rats by antioxidants
-
DOI 10.1073/pnas.96.19.10857
-
Tanaka Y, Gleason CE, Tran POT, Harmon JS, Robertson RP (1999) Prevention of glucose toxicity in HIT-T15 cells and Zucker diabetic fatty rats by antioxidants. Proc Natl Acad Sci USA 96: 10857-10862. (Pubitemid 29442234)
-
(1999)
Proceedings of the National Academy of Sciences of the United States of America
, vol.96
, Issue.19
, pp. 10857-10862
-
-
Tanaka, Y.1
Gleason, C.E.2
Tran, P.O.T.3
Harmon, J.S.4
Robertson, R.P.5
-
31
-
-
0141727767
-
N-acetylcysteine and taurine prevent hyperglycemia-induced insulin resistance in vivo: Possible role of oxidative stress
-
Haber CA, Lam TK, Yu Z, Gupta N, Goh T, Bogdanovic E, Giacca A, Fantus IG (2003) N-acetylcysteine and taurine prevent hyperglycemia-induced insulin resis tance in vivo: possible role of oxidative stress. Am J Physiol 285: E744-753. (Pubitemid 37176425)
-
(2003)
American Journal of Physiology - Endocrinology and Metabolism
, vol.285
, Issue.4
-
-
Haber, C.A.1
Lam, T.K.T.2
Yu, Z.3
Gupta, N.4
Goh, T.5
Bogdanovic, E.6
Giacca, A.7
Fantus, I.G.8
-
32
-
-
36148988058
-
Transgenic expression of antioxidant protein thioredoxin in pancreatic beta cells prevents progression of type 2 diabetes mellitus
-
DOI 10.1089/ars.2007.1586
-
Yamamoto M, Yamato E, Toyoda SI, Tashiro F, Ikegami H, Yodoi J, Miyazaki JI (2008) Transgenic expression of antioxidant protein thioredoxin in pancreatic beta cells prevents progression of type 2 diabetes mellitus. Antioxid Redox Signal 10: 43-49. (Pubitemid 350115982)
-
(2008)
Antioxidants and Redox Signaling
, vol.10
, Issue.1
, pp. 43-49
-
-
Yamamoto, M.1
Yamato, E.2
Shu-Ichi, T.3
Tashiro, F.4
Ikegami, H.5
Yodoi, J.6
Miyazaki, J.-I.7
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