AKT1 represses gene conversion induced by different genotoxic stresses and induces supernumerary centrosomes and aneuploidy in hamster ovary cells

Oncogene

Volumn 28, Issue 22, 2009, Pages 2231-2237

  Plo, I ^a   Lopez, B ^a  

^a INSTITUT DE RADIOPROTECTION ET DE SÛRETÉ NUCLÉAIRE   (France)

Author keywords

AKT1; Aneuploidy; BRCA1; Breast and ovary cancer; Centrosome; Homologousre combination

Indexed keywords

BRCA1 ASSOCIATED RING DOMAIN PROTEIN 1; BRCA1 PROTEIN; PROTEIN AKT1; PROTEIN KINASE B; RAD51 PROTEIN; UNCLASSIFIED DRUG;

ANEUPLOIDY; ANIMAL CELL; APOPTOSIS; ARTICLE; CARCINOGENICITY; CELL LINE; CELL PROLIFERATION; CENTROSOME; CONTROLLED STUDY; GENE CONVERSION; GENE OVEREXPRESSION; GENOMIC INSTABILITY; GENOTOXICITY; HAMSTER; HOMOLOGOUS RECOMBINATION; HUMAN; HUMAN CELL; IONIZING RADIATION; NONHUMAN; ONCOGENE; OVARY CELL; PRIORITY JOURNAL; PROTEIN FUNCTION; SUPERNUMERARY CHROMOSOME; ULTRAVIOLET RADIATION;

ANEUPLOIDY; ANIMALS; BRCA1 PROTEIN; CENTROSOME; CHO CELLS; CRICETINAE; CRICETULUS; DNA BREAKS, DOUBLE-STRANDED; DNA DAMAGE; GENE CONVERSION; GENE EXPRESSION REGULATION; MITOSIS; PROTO-ONCOGENE PROTEINS C-AKT; RAD51 RECOMBINASE; ULTRAVIOLET RAYS;

CRICETINAE;

EID: 67349281546     PISSN: 09509232     EISSN: 14765594     Source Type: Journal    
DOI: 10.1038/onc.2009.85     Document Type: Article

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