Mitochondrial superoxide plays a crucial role in the development of mitochondrial dysfunction during high glucose exposure in rat renal proximal tubular cells

Free Radical Biology and Medicine

Volumn 46, Issue 8, 2009, Pages 1149-1157

  Munusamy, Shankar ^a   MacMillan Crow, Lee Ann ^a  

^a UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES   (United States)

Author keywords

Diabetes; Free radicals; Hyperglycemia; Manganese superoxide dismutase; MitoSox red; Nephropathy; Superoxide

Indexed keywords

GLUCOSE; MANGANESE SUPEROXIDE DISMUTASE; STREPTOZOCIN; SUPEROXIDE;

ANIMAL CELL; ANIMAL MODEL; ARTICLE; CELL VIABILITY; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME INACTIVATION; HYPERGLYCEMIA; HYPERPOLARIZATION; KIDNEY INJURY; KIDNEY PROXIMAL TUBULE; MALE; MITOCHONDRIAL MEMBRANE POTENTIAL; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; STREPTOZOCIN DIABETES;

ADENOSINE TRIPHOSPHATE; ANIMALS; CELL LINE; CELL SURVIVAL; DIABETES MELLITUS, EXPERIMENTAL; DIABETIC NEPHROPATHIES; ELECTRON TRANSPORT COMPLEX III; HYPERGLYCEMIA; KIDNEY TUBULES, PROXIMAL; MALE; MEMBRANE POTENTIAL, MITOCHONDRIAL; MITOCHONDRIA; RATS; RATS, SPRAGUE-DAWLEY; SUPEROXIDE DISMUTASE; SUPEROXIDES; TRANSFECTION; TRANSGENES;

RATTUS;

EID: 63049127020     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.freeradbiomed.2009.01.022     Document Type: Article

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