The HINT1 tumor suppressor regulates both γ-H2AX and ATM in response to DNA damage

Journal of Cell Biology

Volumn 183, Issue 2, 2008, Pages 253-265

  Li, Haiyang ^a   Balajee, Adayabalam S ^b   Su, Tao ^a   Cen, Bo ^a   Hei, Tom K ^a,b,c   Weinstein, I Bernard ^a,c,d  

^a NewYork Presbyterian Hospital   (United States)

^b Center for Radiological Research   (United States)

^c Department of Environmental Health Science ^*  (United States)

^d Och Spine at New York Presbyterian Hospitals   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ATM PROTEIN; DNA; CELL CYCLE PROTEIN; DNA BINDING PROTEIN; GAMMA H2AX PROTEIN, MOUSE; GAMMA-H2AX PROTEIN, MOUSE; HINT1 PROTEIN, MOUSE; HISTONE; NERVE PROTEIN; PROTEIN P53; PROTEIN SERINE THREONINE KINASE; TUMOR SUPPRESSOR PROTEIN;

ACETYLATION; ANIMAL CELL; APOPTOSIS; ARTICLE; CHROMOSOME ABERRATION; CONTROLLED STUDY; DNA DAMAGE; GENE FUNCTION; GENE LOCUS; MOUSE; NONHUMAN; PRIORITY JOURNAL; TUMOR SUPPRESSOR GENE; ANIMAL; CYTOLOGY; FIBROBLAST; GAMMA RADIATION; GENE EXPRESSION REGULATION; GENETIC RECOMBINATION; GENOMIC INSTABILITY; HUMAN; METABOLISM; PROTEIN BINDING; RADIATION EXPOSURE;

ACETYLATION; ANIMALS; APOPTOSIS; CELL CYCLE PROTEINS; CHROMOSOME ABERRATIONS; DNA DAMAGE; DNA-BINDING PROTEINS; FIBROBLASTS; GAMMA RAYS; GENE EXPRESSION REGULATION; GENOMIC INSTABILITY; HISTONES; HUMANS; MICE; NERVE TISSUE PROTEINS; PROTEIN BINDING; PROTEIN-SERINE-THREONINE KINASES; RECOMBINATION, GENETIC; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

EID: 55949096154     PISSN: 00219525     EISSN: 00219525     Source Type: Journal    
DOI: 10.1083/jcb.200711150     Document Type: Article

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