Clustered DNA lesion sites as a source of mutations during human colorectal tumourigenesis

Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis

Volumn 646, Issue 1-2, 2008, Pages 60-68

  Radford, Ian R ^a   Lobachevsky, Pavel N ^a  

^a NONE

Author keywords

Carcinogenesis; Clustered DNA lesion site; Mutation; Reactive oxygen species

Indexed keywords

APC; K RAS PROTEIN; PHOSPHATIDYLINOSITOL 3 KINASE; PHOSPHATIDYLINOSITOL 3 KINASE CATALYTIC ALPHA POLYPEPTIDE; POLYPEPTIDE; PROTEIN P53; UNCLASSIFIED DRUG;

ARTICLE; CARCINOGENESIS; COLORECTAL TUMOR; CONTROLLED STUDY; DNA DAMAGE; GENE MUTATION; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL;

1-PHOSPHATIDYLINOSITOL 3-KINASE; BASE SEQUENCE; CLUSTER ANALYSIS; COLORECTAL NEOPLASMS; DATA INTERPRETATION, STATISTICAL; DNA; DNA MUTATIONAL ANALYSIS; GENES, P53; GENES, RAS; HUMANS; MODELS, GENETIC; MOLECULAR SEQUENCE DATA; MUTATION; TUMOR SUPPRESSOR PROTEIN P53;

RATTUS;

EID: 54149098939     PISSN: 00275107     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mrfmmm.2008.08.017     Document Type: Article

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